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 加分100%浜中特選昆布鍋物適合請客嗎？》台中公益路大揭密｜10家美食名店全盤解析 |
| 在地生活｜其他 2026/05/20 06:29:44 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人，臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」，從精緻西餐到創意火鍋，從日式丼飯到義式早午餐，每走幾步，就會有完全不同的特色料理餐廳。 這次我特別花了一整個月，實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店，也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準，整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你，找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型，從高質感牛排館到巷弄系早午餐，每一間都有自己獨特的風格。為了讓整體比較更客觀，我依照以下四大面向進行評比，並搭配實際用餐體驗來打分。
整體而言，我希望這份評比不只是「哪家好吃」，而是幫你在不同情境下（約會、家庭聚餐、朋友小聚、商業午餐）都能快速找到合適的選擇。畢竟，美食不只是味覺的滿足，更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段，從火鍋、燒肉到中式料理與早午餐，每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單，橫跨平價、創意、高級各路風格。
一頭牛日式燒肉｜炭香濃郁的和牛饗宴，約會聚餐首選
走在公益路上，很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面，搭配昏黃燈光與暖色調的內裝，讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大，但桌距規劃得宜，每桌皆設有獨立排煙設備，烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂，絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網，讓人完全不用分心。整場用餐過程就像一場表演，從視覺、嗅覺到味覺都被滿足。 綜合評分
地址：408臺中市南屯區公益路二段162號電話：04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」，更像是一場五感盛宴。從進門那一刻到最後一道甜點，都能感受到他們對細節的用心。 TANG Zhan 湯棧｜文青系火鍋代表，麻香湯底與視覺美感並重
在公益路這條美食戰線上，TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址：408臺中市南屯區公益路二段248號電話：04-22580617 官網：https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度，又結合現代風格與細節服務，讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店，湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店｜明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂，那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主，陽光灑進室內，讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧，建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味，選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口，米芯保留微Q口感；而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香，口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹，無論是一個人帶電腦工作、或朋友聚餐，都能找到舒服角落。餐點上桌速度穩定，服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」，很適合想遠離忙碌日常的人。 綜合評分
地址：40861臺中市南屯區公益路二段18號電話：04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇，而是以「剛剛好」的份量與風味，陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店，NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物｜平價卻用心的湯頭系火鍋，家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上，加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐，但靠著實在的湯頭與親切的服務，默默吸引許多回頭客。每到用餐時間，總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底，湯頭清澈卻不單薄，越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向，桌距寬敞、座位舒適，帶小孩來也不覺擁擠。店員態度親切，補湯、收盤都很勤快，給人一種「被照顧著」的安心感。 綜合評分
地址：403臺中市西區公益路288號電話：0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤，而是用最簡單的湯頭與新鮮食材，傳遞出家常卻不平凡的溫度。 印月餐廳｜中式料理的藝術演繹，宴客與家庭聚會首選
說到臺中公益路的中式料理代表，印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名，把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設，每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏，都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法，讓人感受到「被款待」的尊榮感。 綜合評分
地址：408臺中市南屯區公益路二段818號電話：0422511155 小結語印月餐廳是一間「不只吃飯，更像品味生活」的地方。 KoDō 和牛燒肉｜極致職人精神，專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗，一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉，以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址：403臺中市西區公益路260號電話：0423220312 官網：https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳，而是一場體驗。 永心鳳茶｜在茶香裡用餐的優雅時光，臺味早午餐的新詮釋
走進 永心鳳茶公益店，彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤，無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」，都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和，對茶品介紹詳盡。上餐節奏剛好，不急不徐。 綜合評分
地址：40360臺中市西區公益路68號三樓(勤美誠品)電話：0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓｜老饕級江浙功夫菜，穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主，兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址：408臺中市南屯區公益路二段95號電話：0423202322 官網：https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司｜低調奢華的無菜單日料，職人手藝詮釋旬味極致
在熱鬧的公益路上，一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase（無菜單料理） 形式，每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘，節奏緩慢但沉穩。 綜合評分
地址：408臺中市南屯區公益路二段25號電話：0423206368 官網：https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂｜人氣爆棚的和牛燒肉聖地，肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳，茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐，價格約落在 $700–$1000 間，份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快，整場用餐流程流暢無壓力。 綜合評分
地址：403臺中市西區公益路268號電話：0423281167 官網：https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質＋輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後，臺中公益路不只是一條美食街，而是一段生活風景線。 有的餐廳講究細膩與儀式感，像 一頭牛日式燒肉 與 一笈壽司，讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心，像 加分昆布鍋物、永心鳳茶，讓人明白吃飯不只是為了飽足，而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店，則用穩定的品質與熱絡的氛圍，成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店，構成了公益路最動人的縮影 有華麗的，也有溫柔的；有傳統的，也有創新的。 每一家都在自己的風格裡發光，讓人吃到的不只是料理，而是一種生活的溫度與節奏。 對我而言，這不僅是一場美食旅程，更是一趟關於「臺中味道」的回憶之旅。 FAQ：關於臺中公益路美食常見問題Q1：公益路哪一區的餐廳最集中？ Q2：需要提前訂位嗎？ 最後的話若要用一句話形容這趟美食之旅，我會說： 一頭牛日式燒肉值得排隊嗎？ 如果你也和我一樣喜歡用味蕾探索一座城市，那就把這篇公益路美食攻略收藏起來吧。加分100%浜中特選昆布鍋物有什麼推薦搭配？ 無論是約會、慶生、家庭聚餐，或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。茶六燒肉堂年節期間價格會變嗎？ 下一餐，不妨從這10家開始。TANG Zhan 湯棧春酒場面夠體面嗎？ 打開手機、約上朋友，讓公益路成為你生活裡最容易抵達的小確幸。KoDō 和牛燒肉假日會大排長龍嗎？ 如果你有私心愛店，也歡迎留言分享，印月餐廳春酒菜色豐富嗎？ 你的推薦，可能讓我下一趟美食旅程變得更精彩。印月餐廳有提供尾牙方案嗎？ A study highlights the cerebellum’s role in learning and movement through “zombie neurons,” uncovering how these functionally altered but alive neurons are vital for associative learning signals. Nestled at the back of your head, the cerebellum is a brain structure that plays a pivotal role in how we learn, adapting our actions based on past experiences. Yet the precise ways in which this learning happens are still being defined. A study led by a team at the Champalimaud Foundation brings new clarity to this debate, with a serendipitous finding of so-called “zombie neurons.” These neurons, alive but functionally altered, have helped to advance our understanding of the cerebellum’s critical teaching signals. The word “cerebellum” means “little brain,” despite the fact that it holds more than half the brain’s neurons. It is essential for coordinating movements and balance, helping you perform everyday tasks smoothly, like walking down a crowded street, or playing sports. It is also crucial for the learning process that allows you to associate sensory cues with specific actions. Every time you pick up a cup without spilling its contents, effortlessly adjusting the amount of force you apply based on the weight of the container and how full it is, you’re experiencing the consequences of the cerebellum’s ability to link visual signals with corresponding movement responses. The brain’s “teaching signals” For learning to take place, the cerebellum continuously monitors the outside world and the outcome of movements that we make within it. When we make a mistake, information about our errors can be used to adjust the strength of brain connections, leading over time to changes in our behavioral responses to specific cues. However, it is not known exactly how such “error” or “teaching signals” are represented within the brain to drive learned changes in behavior. The latest research from the Champalimaud Foundation’s Carey Lab, published in Nature Neuroscience, provides compelling evidence that activity in a specific class of cerebellar inputs, called climbing fibers, are absolutely essential for associative learning to occur. To examine the role of climbing fibers and their targets, cerebellar Purkinje cells, in learning, the researchers designed an experiment involving mice. They used a common learning task known as eyeblink conditioning. In this task, a mouse learns to blink in response to a certain signal, such as a light, which precedes an event, typically a gentle puff of air aimed at its eye. The animals then display associative learning, learning to link a sensory signal with an adaptive movement response, in this case, blinking. Climbing fibers, in the form of ivy, wrap around the branches of a Purkinje cell-shaped tree, within the vibrant courtyard of a school populated by mice. The illustration captures the essential role of climbing fibers as teaching signals for associative cerebellar learning. Credit: Rita Felix “In our experiment,” explains Dr. Tatiana Silva, the study’s first author, “we used a technique called optogenetics. This method functions like a highly precise remote control for brain cells, using light to turn on or off certain cells of interest at extremely specific times.” Silva continues, “Climbing fibers normally respond to sensory stimuli like a puff of air to the eye. By precisely activating these fibers with optogenetics, we were able to trick the mouse into thinking it had received an air puff, when in fact it had not. After we consistently stimulated climbing fibers during the presentation of a visual cue, the mice learned to blink in response to that cue – even in the absence of stimulation. This proved that these fibers are sufficient to drive this type of associative learning.” The authors were further able to show that climbing fibers are also necessary for associative learning. “When we used optogenetics to selectively silence climbing fibers during the presentation of an actual air puff,” Silva reveals, “the mice completely failed to learn to blink in response to the visual cue.” Carey’s team similarly manipulated a number of other types of brain cells within the cerebellum, but found that none of them were able to provide such reliable teaching signals for learning. The Emergence of “Zombie Neurons” Looking more closely at some of their data, the researchers discovered an unexpected twist. In order to manipulate climbing fiber activity using optogenetics, they had used genetic tools to express a light-sensitive protein called Channelrhodopsin-2 (ChR2) in those neurons. Surprisingly, they found that when they tried to teach the ChR2-expressing mice using the traditional air puff method, the animals completely failed to learn. As Carey explains, after systematic recordings of neural activity from the cerebella of these mice, “It turned out that introducing ChR2 into the climbing fibers altered their natural properties, preventing them from responding appropriately to standard sensory stimuli like air puffs. This, in turn, completely blocked the animals’ ability to learn”. “The remarkable thing,” says Silva, “was that these same mice learned perfectly well when we paired climbing fiber stimulation, instead of an air puff, to a visual cue.” Unintentionally, the team had achieved a long-standing objective in neuroscience: to modulate specific patterns of activity within specific neurons without entirely shutting down their communication, resulting in a more natural intervention to elucidate their causal role. In other words, although the climbing fibers remained spontaneously active and were clearly otherwise functional, their altered encoding of sensory stimuli left animals totally unable to learn the task. This led Silva to dub them “zombie neurons”: functionally alive but not interacting with the brain circuit as usual. Due to the subtlety of the unexpected effects of ChR2 expression in climbing fibers, Dr. Megan Carey says, “These results serve as the most compelling evidence to date that climbing fiber signals are essential for cerebellar associative learning. Our next steps involve understanding why ChR2 expression leads to the ‘zombification’ of neurons and determining whether our findings extend to other forms of cerebellar learning”. Even the undead, it seems, have something to teach us about the world of the living Reference: “Climbing fibers provide essential instructive signals for associative learning” by N. Tatiana Silva, Jorge Ramírez-Buriticá, Dominique L. Pritchett and Megan R. Carey, 2 April 2024, Nature Neuroscience. DOI: 10.1038/s41593-024-01594-7 A spherical lipoprotein article with proteins on the surface is visible in the lower part of the image in this artist’s rendition that includes neurons (light-green cells), synapses (connections highlighted in yellow), and microglia (in purple). Credit: Mike Perkins | Pacific Northwest National Laboratory New research provides a fresh glimpse of APOE, scores of new molecular players in the central nervous system. Researchers have developed a technique to identify key fat-filled particles known as lipoproteins within the central nervous system, opening a new view into the workings of the brain. The study revealed that these particles, molecular cousins of the well-known HDL, or “good cholesterol” particles in our bloodstream, are much more diverse than previously thought. The team identified over 300 distinct proteins linked with these particles, a significant increase from the previously known 16, that fall into at least 10 different families. These particles are rich in proteins that affect wound healing, the immune response, and the creation and nurturing of brain cells called neurons which are important for cognitive function. The most common protein on the particles is apolipoprotein E, better known as APOE. Of the three commonly studied forms of APOE, the form known as APOE4 puts people at higher risk of Alzheimer’s disease. One copy of the APOE4 gene makes a person approximately four times as likely to develop dementia; a person with two copies is about 12 times more vulnerable. The results were recently published in the journal Science Advances. The leader of the study is John Melchior, a protein biochemist at the Department of Energy’s Pacific Northwest National Laboratory. Melchior is a leader in lipoprotein research—and a carrier of two copies of the APOE4 gene, adding a personal incentive to his work to understand the protein’s action in the nervous system. “We’ve known for a long time that in the nervous system, APOE is the primary protein on these particles calling the shots. But we don’t know much more beyond that. Our technology opens the door to learning more,” said Melchior. “What the heck is APOE4 doing? That’s the big question. Why does one form translate to less risk for dementia while a slightly different form confers significant risk? Our technology brings us one step toward more answers,” he added. APOE: Bringing Fats and Proteins Together Lipoproteins are best known for their work in the circulatory system, where they transport fat and cholesterol. It’s easy for scientists to detect HDL and LDL, known to many as “good cholesterol” and “bad cholesterol,” in the bloodstream because the molecules there are plentiful. However, lipoproteins in the nervous system are much scanter, present at less than 1 percent of the concentration in blood. Their actions, even their presence, in the nervous system have been a mystery. Scientists know that these balls of fat and protein wrapped together to travel in the bloodstream and carry out all sorts of important functions, like shuttling cholesterol and nutrients. On the particles in the central nervous system, APOE reigns supreme, serving as a scaffold to hold lipids and other proteins together. It also transports these nutrient-rich lipids and collections of molecular collaborators—groupings of proteins on its surface—throughout the nervous system to perform their tasks. The proteins are specialized tools that can do things like repair cells, turn genes on or off, or regulate amyloid-beta processing, a well-known molecule related to the development of dementia. The work suggests that APOE may bring these tools together on different particles to deliver them where needed. But something is more likely to go wrong in people with one or two copies of APOE4, leading to dementia. Scientists don’t know what. Scientists suspect APOE4 of playing a role in other neurologic conditions such as Parkinson’s and Huntington’s diseases, multiple sclerosis, amyotrophic lateral sclerosis, and even traumatic brain injury. Because lipoproteins are less common in the nervous system, researchers either need an impossibly large amount of the cerebrospinal fluid to study them—or scientists develop a new way to detect the rare molecules. That’s what Melchior’s team did, creating a new fluorescent technology to tag lipoproteins in spinal fluid. The team studied just one-third of a milliliter of spinal fluid—much less fluid than in a raindrop—and discovered 303 different proteins across the particle families using mass spectrometry. Most had never been detected on these particles in the nervous system before. A Solid Start and Next Steps: Lipoproteins and Neurological Diseases “Now comes the fun part,” said Melchior. “We want to open up our technology to clinicians to learn more about what’s happening in Alzheimer’s disease and possibly other conditions like multiple sclerosis and Parkinson’s disease. “There are existing cerebrospinal fluid samples sitting in freezers, and we have a new way to analyze them. We’d love to work together with other research teams to investigate them. The sooner we can start profiling lipoproteins in these conditions, the sooner we can understand more about their role in disease pathology and identify targets for treatments,” added Melchior, who holds joint appointments at Oregon Health & Science University and the University of Cincinnati. First author of the paper is PNNL scientist Nathaniel Merrill who did much of the computational analysis of the data. Merrill designed computational tools to help sort out extraordinarily complicated datasets. This includes which proteins are most likely to be found together on different particle populations and what processes those proteins control in the central nervous system. Reference: “Human cerebrospinal fluid contains diverse lipoprotein subspecies enriched in proteins implicated in central nervous system health” by Nathaniel J. Merrill, W. Sean Davidson, Yi He, Ivo Díaz Ludovico, Snigdha Sarkar, Madelyn R. Berger, Jason E. McDermott, Linda J. Van Eldik, Donna M. Wilcock, Matthew E. Monroe, Jennifer E. Kyle, Kimberley D. Bruce, Jay W. Heinecke, Tomas Vaisar, Jacob Raber, Joseph F. Quinn and John T. Melchior, 30 August 2023, Science Advances. DOI: 10.1126/sciadv.adi5571 The study brought together investigators from the University of Cincinnati, OHSU, University of Washington, University of Kentucky, University of Colorado, VA Portland Healthcare System as well as PNNL. In addition to Melchior and Merrill, PNNL authors include Ivo Díaz Ludovico, Snigdha Sarkar, Madelyn Berger, Jason McDermott, Matthew Monroe and Jennifer Kyle. The work was done as part of the Pacific Northwest Biomedical Innovation Co-Laboratory, or PMedIC, where PNNL and OHSU scientists and physicians work together to bring basic science and clinical experience together to explore disease and develop innovative therapies. The work was funded by the National Institutes of Health (1R03AG070480, P01HL128203, R01AG079217, P30AG066518) and by the OHSU School of Medicine. The early support for the project from NIH was crucial, said W. Sean Davidson of the University of Cincinnati, a coauthor and an expert on lipoproteins in the circulatory system. “We took advantage of the Notice of Special Interest program, which allows labs that are funded in other areas—in our case, cardiovascular disease—to translate new technologies toward Alzheimer’s disease. This made it possible to turn a crazy idea into an exciting new way to analyze lipoproteins in the brain,” said Davidson. Recent research in South Africa’s Greater Kruger National Park has uncovered that local wildlife, including elephants and giraffes, fear humans more than lions (pictured above), highlighting the global impact of humans as “super predators” and posing new challenges for wildlife conservation. Credit: Daniel Rosengren A new study reveals that elephants, rhinos, giraffes, and other wildlife fear this predator far more than lions. Lions have long been regarded as the apex predators, often referred to as the “king of beasts,” but a new study suggests that on the African savanna, elephants, rhinos, giraffes, and other mammals exhibit greater fear of a different threat: humans. This finding adds significant weight to the increasing body of evidence from global wildlife research, which indicates that the fear of humans as the “super predator” is widespread among animal populations around the world. Elephants aggressively approaching, and attacking, speakers broadcasting lion vocalizations. Credit: Liana Zanette / Western University The Impact of Human Presence The new findings by Western University biology professor Liana Zanette, in collaboration with one of the world’s leading lion experts, Craig Packer from the University of Minnesota, and others, were published today in Current Biology. Working in one of the world’s premier protected areas, South Africa’s Greater Kruger National Park, Zanette and her colleagues experimentally demonstrated that local wildlife was twice as likely to run, and abandoned waterholes in 40 percent faster time, in response to hearing human voices compared to hearing lions or hunting sounds (dogs barking or gunshots). Elephant still image from camera recording. Credit: Liana Zanette / Western University Near 95 percent (94.7) of species ran more or abandoned waterholes faster in response to humans than to lions, with giraffes, leopards, hyenas, zebras, kudu, warthog, and impala all running significantly more from the sound of human voices than the sound of lions, and elephants and rhinos abandoning waterholes significantly faster upon hearing humans than hearing lions. Rhinoceroses and elephants abandoning waterholes upon hearing humans. Credit: Liana Zanette / Western University Ecological Consequences and Global Perspective “These findings add a new dimension to our worldwide environmental impacts,” said Zanette, a renowned wildlife ecologist. “The very substantial fear of humans demonstrated here, and in comparable recent experiments, can be expected to have dramatic ecological consequences, because other new research has established that fear itself can reduce wildlife numbers.” Global surveys show humans kill prey at much higher rates than other predators, making humans a “super predator.” Giraffe, leopard, hyena, zebra, kudu, warthog and impala running in response to hearing humans. Liana Zanette / Western University “Consistent with humanity’s unique lethality, data from North America, Europe, Asia, and Australia, and now our work in Africa, is demonstrating that wildlife worldwide fear the human ‘super predator’ far more than each system’s non-human apex predator, like lions, leopards, wolves, cougars, bears, and dogs,” said Zanette. To conduct their experiment, Zanette and the team deployed hidden automated camera-speaker systems at waterholes that, when triggered by an animal passing within a short distance (approximately 10 meters or 30 feet), filmed the response of the animal to hearing either humans speaking calmly in locally-used languages, lions snarling and growling, hunting sounds or non-threatening controls (bird calls). “These results present a significant new challenge for protected areas management and wildlife conservation, because it is now clear fear of even benign humans, like wildlife tourists, can cause these previously unrecognized impacts,” said Zanette. Reference: “Fear of the human “super predator” pervades the South African savanna” by Liana Y. Zanette, Nikita R. Frizzelle, Michael Clinchy, Michael J.S. Peel, Carson B. Keller, Sarah E. Huebner and Craig Packer, 5 October 2023, Current Biology. DOI: 10.1016/j.cub.2023.08.089 RRG455KLJIEVEWWF |
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