cholesterol’s function. Our livers produce cholesterol and will reduce its production when we consume foods with cholesterol. Thus, in normal condition our body should have a balance of cholesterol to properly function as follow:
1). Low Density Cholesterol (LDL) to bind fate and carry them in our blood steam and transport the fate to all part of our body. We have bad name for fate but it’s required for our survival. We also have a bad name for LDL- “Bad Cholesterol”. But imagine we don’t have LDL then we would all die. 2). High Density Cholesterol (HDL)- This is the cholesterol picking up those fates drop in our blood steam by LDLs when them transport those fate for our body. They theory is that we don’t want too much fate dropping in our blood steam to clog blood vessels. LDL fate binding is not perfect thus we have HDL acting as cleaner.
Thus there is no so called good or bad cholesterol. And what is properly level of HDL and LDL also become an issue debatable. Once this ratio, namely, (LDL+HDL +some part of Trigylcerides)/ HDL was widely use to gauge whether we going to have problem or not. But I don’t see this number showing up on blood test Lipitor panel report anymore. Maybe this is also a part of new thinking.
By the way you mention taken Lipitor from Merck. I believe Lipitor (atorvastatin) is made by Pfizer. Side effect like cram is quite often when take so call “statin” medicine.
Victoria Zigmont and team examined the health records of 4,683 men and women who were diabetes-free at the start of the study but were at risk of heart disease. Of the total number of participants, 16% — or 755 people — were taking prescription statins at the start of the study, in 2011. The study ended in 2014. The analysis revealed that people who took statins were more than twice as likely to receive a diabetes diagnosis than those who did not take the medication. Additionally, people who took statins for longer than 2 years were more than three times as likely to develop diabetes.
