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TANG Zhan 湯棧價格合理嗎? 》公益路10大美食推薦|從燒肉到火鍋全攻略 |
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身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人,臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」,從精緻西餐到創意火鍋,從日式丼飯到義式早午餐,每走幾步,就會有完全不同的特色料理餐廳。 這次我特別花了一整個月,實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店,也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準,整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你,找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型,從高質感牛排館到巷弄系早午餐,每一間都有自己獨特的風格。為了讓整體比較更客觀,我依照以下四大面向進行評比,並搭配實際用餐體驗來打分。
整體而言,我希望這份評比不只是「哪家好吃」,而是幫你在不同情境下(約會、家庭聚餐、朋友小聚、商業午餐)都能快速找到合適的選擇。畢竟,美食不只是味覺的滿足,更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段,從火鍋、燒肉到中式料理與早午餐,每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單,橫跨平價、創意、高級各路風格。
一頭牛日式燒肉|炭香濃郁的和牛饗宴,約會聚餐首選
走在公益路上,很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面,搭配昏黃燈光與暖色調的內裝,讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大,但桌距規劃得宜,每桌皆設有獨立排煙設備,烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂,絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網,讓人完全不用分心。整場用餐過程就像一場表演,從視覺、嗅覺到味覺都被滿足。 綜合評分
地址:408臺中市南屯區公益路二段162號電話:04-23206800 官網:http://www.marihuana.com.tw/yakiniku/index.html 小結語一頭牛日式燒肉不僅是「吃肉的地方」,更像是一場五感盛宴。從進門那一刻到最後一道甜點,都能感受到他們對細節的用心。 TANG Zhan 湯棧|文青系火鍋代表,麻香湯底與視覺美感並重
在公益路這條美食戰線上,TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址:408臺中市南屯區公益路二段248號電話:04-22580617 官網:https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度,又結合現代風格與細節服務,讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店,湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店|明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂,那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主,陽光灑進室內,讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧,建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味,選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口,米芯保留微Q口感;而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香,口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹,無論是一個人帶電腦工作、或朋友聚餐,都能找到舒服角落。餐點上桌速度穩定,服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」,很適合想遠離忙碌日常的人。 綜合評分
地址:40861臺中市南屯區公益路二段18號電話:04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇,而是以「剛剛好」的份量與風味,陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店,NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物|平價卻用心的湯頭系火鍋,家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上,加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐,但靠著實在的湯頭與親切的服務,默默吸引許多回頭客。每到用餐時間,總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底,湯頭清澈卻不單薄,越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向,桌距寬敞、座位舒適,帶小孩來也不覺擁擠。店員態度親切,補湯、收盤都很勤快,給人一種「被照顧著」的安心感。 綜合評分
地址:403臺中市西區公益路288號電話:0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤,而是用最簡單的湯頭與新鮮食材,傳遞出家常卻不平凡的溫度。 印月餐廳|中式料理的藝術演繹,宴客與家庭聚會首選
說到臺中公益路的中式料理代表,印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名,把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設,每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏,都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法,讓人感受到「被款待」的尊榮感。 綜合評分
地址:408臺中市南屯區公益路二段818號電話:0422511155 小結語印月餐廳是一間「不只吃飯,更像品味生活」的地方。 KoDō 和牛燒肉|極致職人精神,專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗,一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉,以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址:403臺中市西區公益路260號電話:0423220312 官網:https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳,而是一場體驗。 永心鳳茶|在茶香裡用餐的優雅時光,臺味早午餐的新詮釋
走進 永心鳳茶公益店,彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤,無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」,都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和,對茶品介紹詳盡。上餐節奏剛好,不急不徐。 綜合評分
地址:40360臺中市西區公益路68號三樓(勤美誠品)電話:0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓|老饕級江浙功夫菜,穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主,兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址:408臺中市南屯區公益路二段95號電話:0423202322 官網:https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司|低調奢華的無菜單日料,職人手藝詮釋旬味極致
在熱鬧的公益路上,一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase(無菜單料理) 形式,每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘,節奏緩慢但沉穩。 綜合評分
地址:408臺中市南屯區公益路二段25號電話:0423206368 官網:https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂|人氣爆棚的和牛燒肉聖地,肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳,茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐,價格約落在 $700–$1000 間,份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快,整場用餐流程流暢無壓力。 綜合評分
地址:403臺中市西區公益路268號電話:0423281167 官網:https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質+輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後,臺中公益路不只是一條美食街,而是一段生活風景線。 有的餐廳講究細膩與儀式感,像 一頭牛日式燒肉 與 一笈壽司,讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心,像 加分昆布鍋物、永心鳳茶,讓人明白吃飯不只是為了飽足,而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店,則用穩定的品質與熱絡的氛圍,成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店,構成了公益路最動人的縮影 有華麗的,也有溫柔的;有傳統的,也有創新的。 每一家都在自己的風格裡發光,讓人吃到的不只是料理,而是一種生活的溫度與節奏。 對我而言,這不僅是一場美食旅程,更是一趟關於「臺中味道」的回憶之旅。 FAQ:關於臺中公益路美食常見問題Q1:公益路哪一區的餐廳最集中? Q2:需要提前訂位嗎? 最後的話若要用一句話形容這趟美食之旅,我會說: TANG Zhan 湯棧尾牙聚餐表現如何? 如果你也和我一樣喜歡用味蕾探索一座城市,那就把這篇公益路美食攻略收藏起來吧。一頭牛日式燒肉需要訂位嗎? 無論是約會、慶生、家庭聚餐,或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。永心鳳茶調味偏重嗎? 下一餐,不妨從這10家開始。NINI 尼尼臺中店服務態度如何? 打開手機、約上朋友,讓公益路成為你生活裡最容易抵達的小確幸。茶六燒肉堂真的有那麼好吃嗎? 如果你有私心愛店,也歡迎留言分享,三希樓家庭過節聚會適合嗎? 你的推薦,可能讓我下一趟美食旅程變得更精彩。加分100%浜中特選昆布鍋物尾牙聚餐表現如何? Researchers have discovered a cellular uptake pathway that is crucial for larger molecules. These molecules can be used to create new drugs for treating cancer and other diseases as they bind in unique ways to their targets and are efficiently taken up by cells. Revolutionary Bitopic Inhibitors Pave the Way for Innovative Disease Treatment Strategies The development of drugs is a balancing act between making sure the drug is a good fit for its target and ensuring it can penetrate the cell membrane to reach that target. Typically, the search for drugs that can cross the cell membrane has focused on small, rigid molecules with nonpolar chemical structures. However, new therapeutic strategies break traditional drug design rules by employing larger, flexibly linked chemical entities. Recently, a team of researchers from the University of California, San Francisco (UCSF) published a study in Science, where they unveiled a new discovery of a cellular uptake pathway that is crucial for larger molecules. These large, complex molecules bind in unique ways to their targets, are efficiently taken up by cells, and have the potential to be used in creating new drugs for the treatment of cancer and other diseases. Through a combination of functional genomics and chemical methods, the scientists uncovered an endogenous pathway involving interferon-induced transmembrane (IFITM) proteins that promote the cellular uptake of diverse linked chemotypes. These proteins are found in plasma membranes and often provide cellular resistance to viruses. Challenges in Traditional Drug Targeting Most traditional pharmaceuticals are small molecules that follow simple molecular rules including limits on the molecular size and number of sticky chemical groups on the molecule’s surface. Many key drug targets, such as kinase enzymes often involved in cancer, are difficult to selectively target with traditional drugs. “There are over 500 human kinase enzymes that are very similar in the pocket where the drug binds, making it a challenge to selectively target a single member of this family and leading to undesirable medication side effects,” explains the study’s first author Kevin Lou. “Increasingly, it has been found that certain linked molecules outside this traditional framework can maintain drug-like properties and gain new mechanisms of action.” There are many important intracellular drug targets that researchers have been unable to target with small, compact, and rigid molecules. To address this challenge, scientists have taken to linking multiple ligands into a single chemical entity (a linked chemotype). These linked chemotypes can have enhanced potency, greater selectivity, and the capacity to induce the association of more than one target. “Given this discrepancy between the favorable biological activity of many large, bivalent molecules and traditional concepts of passive permeability, we inferred that linked chemotypes might hijack cellular processes to assist with passage through the cell membrane,” wrote Lou. We selected as an example a bitopic inhibitor of mTOR, RapaLink-1, whose molecular weight falls well beyond common guidelines.” Designing Multi-Pronged Inhibitors The team designed two new linked drugs that they hypothesized might take advantage of this cellular entry pathway. They generated DasatiLink-1 through a linker-joined combination of two known inhibitors of the leukemia protein BCL-ABL1, known as dasatinib and asciminib. Since each drug binds a distinct pocket on the target protein, the researchers reasoned that the linked version could affix itself to two points of contact like a two-pronged key inserting into two locks, enhancing its specificity and effectiveness. They also designed BisRoc-1 by linking two molecules of the chemotherapy drug rocaglamide together in a way that would allow it to bridge two copies of the drug’s protein target. Despite the fact that both of these drugs violate traditional drug design principles, the team showed that both drugs enter cells, bind tightly to their intended targets, and work just as well as the unlinked versions. The linked versions were uniquely dependent on IFITM protein expression in the target cells, supporting a general role for the IFITM pathway across many types of linked molecules. The researchers showed that DasatiLink-1 is specific for only the BCL-ABL1 kinase, unlike the more relaxed specificity of its two constituent drugs when unlinked. Advantages of Selectivity and Efficiency “Linked inhibitors that require a multi-pronged binding mechanism are much more selective,” Lou explains. “They offer substantial advantages as long as they can enter cells efficiently.” “We discovered that IFITM proteins enable bitopic inhibitors to enter cells and this will likely allow us to target previously untargetable proteins in disease,” said Luke Gilbert, Ph.D., co-corresponding author and the Goldberg-Benioff Endowed Professorship in Prostate Cancer Translational Biology at UCSF. “Hopefully, our study will generate new clues for how IFITM proteins function mechanistically that can be pursued by drug design scientists and virologists.” The scientists are working on chemically optimizing the properties of the linked BCR-ABL inhibitors to increase their potency and position them as next-generation therapeutics for BCR-ABL mutant cancers. “We are also excited to expand the scope of intracellular targets amenable to bitopic inhibition,” said Gilbert. Reference: “IFITM proteins assist cellular uptake of diverse linked chemotypes” by Kevin Lou, Douglas R. Wassarman, Tangpo Yang, YiTing Paung, Ziyang Zhang, Thomas A. O’Loughlin, Megan K. Moore, Regina K. Egan, Patricia Greninger, Cyril H. Benes, Markus A. Seeliger, Jack Taunton, Luke A. Gilbert and Kevan M. Shokat, 8 December 2022, Science. DOI: 10.1126/science.abl5829 The study was funded by the National Institutes of Health, the Damon Runyon Cancer Foundation, the Pew-Stewart Scholars program, the Goldberg-Benioff Endowed Professorship, the Howard Hughes Medical Institute, the Samuel Waxman Cancer Research Foundation, Wellcome Trust, the Ono Pharma Foundation, Pfizer, and Arc Institute. Bottom view of a larval blacklegged tick. The tick was microinjected with various fluorescent dyes to enhance its visual appearance. Credit: Utpal Pal / University of Maryland Ticks have the ability to detect a cytokine from infected mammalian blood, triggering a defense mechanism against bacteria that cause Lyme disease. A team of researchers from the University of Maryland has uncovered the first inter-species signaling pathway between a host and an arthropod parasite. The pathway involves molecules in the host’s blood triggering the immunity and development of the parasite. The study demonstrates that when ticks feed on blood from mice infected with the Lyme disease-causing bacteria Borrelia burgdorferi, a protein from the mouse immune system activates receptors on tick cells, signaling the tick’s organs to develop more quickly and producing an immune response before the bacteria can infect the tick. The study, which was recently published in the journal Science, identifies a potential target for anti-tick vaccines or therapeutics to prevent the spread of infections like Lyme disease. The findings also provide important new insights into the evolution of biomolecular interdependencies between species, and highlight, for the first time, both the integration of immunity and animal development and the adaptability of an ancient cell signaling system or pathway that all plant and animal cells use for sensing and responding to their environment. “This adaptive flexibility of a conserved cell signaling pathway was surprising,” said Utpal Pal, senior author of the study and a professor in the Virginia-Maryland College of Veterinary Medicine at College Park. “It is remarkable that this pathway that is present in everything from sponges to humans is so flexible it can adapt to accept a ligand [a binding molecule] from another distant species. This tool that everybody has is being used in a way that we didn’t imagine.” On the right side is a control blacklegged nymphal tick with a normal body. In the nymph on the left, the protein DOME-1, which triggers the JAK/STAT pathway receptor was knocked down, causing developmental defects such as an abnormal and swollen body, translucent abdomen with undigested bloodmeal, unequal or rudimentary legs, and malformed mouthparts. Credit: Utpal Pal / University of Maryland The finding suggests that other cell signaling pathways may have been adapted for novel uses in other organisms and points to a new area in immunology and molecular biology ripe for future exploration. A Novel Role for the JAK/STAT Pathway in Ticks Pal and his colleagues made their discovery while investigating tick immunity, which is a poorly understood area of tick biology. In their initial study, seeking to understand how tick immune systems recognize the Borrelia bacteria, the researchers fed ticks a blood meal from either a Borrelia-infected mouse or an uninfected mouse. Comparing the two groups, they found that the infected blood meal activated a protein in ticks that normally produces energy inside cells. The protein is associated with a simple signaling pathway called JAK/STAT, which is present in all multicellular organisms. As in all cellular signaling pathways, a specific molecule senses something in the environment and then binds to a receptor on the outside of a cell wall. This sets off a cascade of reactions inside the cell that turns a specific gene on or off and produces a response to whatever outside stimuli was sensed. Assuming that JAK/STAT was triggered by the Borrelia in the infected mouse blood, the researchers isolated the bacteria and injected it directly into ticks to see what molecules were binding with the JAK/STAT receptor. Surprisingly, the bacteria did not activate JAK/STAT. To find out what did, the researchers removed the Borrelia bacteria from the blood of infected mice and fed the “clean” blood to ticks. The JAK/STAT pathway kicked into action. The researchers discovered that a protein in tick digestive systems was serving as the JAK/STAT receptor, and that it had evolved to bind with the cytokine protein interferon, which is produced by the immune systems of mammals infected with a bacteria like Borrelia. University of Maryland’s Utpal Pal on the discovery of cross-species signaling pathway in which host blood triggers immunity and development in parasite. Credit: University of Maryland Implications for Tick-Borne Disease Prevention The researchers also found that the JAK/STAT receptor and pathway are important for normal tick development, even if the pathway is not activated by an infected blood meal. When Pal and his colleagues knocked down the expressed gene that produces the receptor for JAK/STAT, the ticks grew deformed legs, mouthparts, and digestive systems, and were unable to feed and complete the developmental cycle to grow further. These results suggest that in ticks, the JAK/STAT signaling pathway, and the protein receptor have evolved to integrate immunity with development. Bacteria will compete with ticks for nutrients in the blood of an infected host, so when a tick gets the signal that a blood meal is infected, growing rapidly is a way to use up those nutrients before the bacteria gets them. Laboratory experiments concur that ticks fed on Borrelia-infected mouse blood developed much quicker than those that fed on uninfected mouse blood. “Understanding that this pathway integrates immunity and development has important implications for potential strategies to prevent tick-borne disease transmission,” Pal said. “Because if you delete the pathway, ticks with malformed mouthparts cannot feed or transmit disease. But what is also really exciting to me is that we see this sort of early-warning system, where the tick’s immune system indirectly detects a pathogen using an immune response from its host rather than the pathogen itself, accelerating its own development.” Reference: “Dome1–JAK–STAT signaling between parasite and host integrates vector immunity and development” by Vipin S. Rana, Chrysoula Kitsou, Shraboni Dutta, Michael H. Ronzetti, Min Zhang, Quentin Bernard, Alexis A. Smith, Julen Tomás-Cortázar, Xiuli Yang, Ming-Jie Wu, Oleksandra Kepple, Weizhong Li, Jennifer E. Dwyer, Jaqueline Matias, Bolormaa Baljinnyam, Jonathan D. Oliver, Nallakkandi Rajeevan, Joao H F Pedra, Sukanya Narasimhan, Yan Wang, Ulrike Munderloh, Erol Fikrig, Anton Simeonov, Juan Anguita and Utpal Pal, 13 January 2023, Science. DOI: 10.1126/science.abl3837 The study was funded by the National Institute of Allergy and Infectious Diseases, the Steven and Alexandra Cohen Foundation, the Howard Hughes Medical Institute, the National Center for Advancing Translational Sciences (NCATS), and the National Institute of Dental and Craniofacial Research. New research unveils the decision-making pathways in bee brains, shedding light on their ability to quickly and accurately assess flowers for nectar, which could inspire more autonomous robot designs. The study, led by various academic experts, also emphasizes the efficiency of evolutionarily refined insect brains that could guide future AI development in industries. Credit: Théotime Colin A New Study Reveals How We Could Design Robots To Think Like Bees Honey bees excel in weighing effort against reward and risk, quickly determining which flowers can provide sustenance for their colony. A study recently published in the journal eLife illustrates how eons of evolution have fine-tuned honey bees to make swift judgments while minimizing danger. This research sheds light on the workings of insect minds, the evolution of human cognition, and offers insights for improved robot design. Modeling Decision-Making in Bees The paper presents a model of decision-making in bees and outlines the paths in their brains that enable fast decision-making. The study was led by Professor Andrew Barron from Macquarie University in Sydney, and Dr. HaDi MaBouDi, Neville Dearden, and Professor James Marshall from the University of Sheffield. “Decision-making is at the core of cognition,” says Professor Barron. “It’s the result of an evaluation of possible outcomes, and animal lives are full of decisions. A honey bee has a brain smaller than a sesame seed. And yet she can make decisions faster and more accurately than we can. A robot programmed to do a bee’s job would need the backup of a supercomputer. “Today’s autonomous robots largely work with the support of remote computing,” Professor Barron continues. “Drones are relatively brainless, they have to be in wireless communication with a data center. This technology path will never allow a drone to truly explore Mars solo – NASA’s amazing rovers on Mars have traveled about 75 kilometers (50 miles) in years of exploration.” Bee. Credit: Théotime Colin Bees need to work quickly and efficiently, finding nectar and returning it to the hive while avoiding predators. They need to make decisions. Which flower will have nectar? While they’re flying, they’re only prone to aerial attack. When they land to feed, they’re vulnerable to spiders and other predators, some of which use camouflage to look like flowers. “We trained 20 bees to recognize five different colored ‘flower disks’. Blue flowers always had sugar syrup,” says Dr. MaBouDi. “Green flowers always had quinine [tonic water] with a bitter taste for bees. Other colors sometimes had glucose.” “Then we introduced each bee to a ‘garden’ where the ‘flowers’ just had distilled water. We filmed each bee then watched more than 40 hours of video, tracking the path of the bees and timing how long it took them to make a decision. How Bees Weigh Uncertainty in Decision-Making “If the bees were confident that a flower would have food, then they quickly decided to land on it taking an average of 0.6 seconds),” says Dr. MaBouDi. “If they were confident that a flower would not have food, they made a decision just as quickly.” If they were unsure, then they took much more time – on average 1.4 seconds – and the time reflected the probability that a flower had food. The team then built a computer model from first principles aiming to replicate the bees’ decision-making process. They found the structure of their computer model looked very similar to the physical layout of a bee brain. “Our study has demonstrated complex autonomous decision-making with minimal neural circuitry,” says Professor Marshall. “Now we know how bees make such smart decisions, we are studying how they are so fast at gathering and sampling information. We think bees are using their flight movements to enhance their visual system to make them better at detecting the best flowers.” AI researchers can learn much from insects and other ‘simple’ animals. Millions of years of evolution have led to incredibly efficient brains with very low power requirements. The future of AI in the industry will be inspired by biology, says Professor Marshall, who co-founded Opteran, a company that reverse-engineers insect brain algorithms to enable machines to move autonomously, like nature. Reference: “How honey bees make fast and accurate decisions” by HaDi MaBouDi, James AR Marshall, Neville Dearden and Andrew B Barron, 27 June 2023, eLife. DOI: 10.7554/eLife.86176 RRG455KLJIEVEWWF 加分100%浜中特選昆布鍋物小孩適合去嗎? 》公益路人氣美食完整評比|10家一次破解三希樓上餐速度快嗎? 》台中公益路吃爆指南|10家餐廳逐間介紹一笈壽司整體體驗如何? 》公益路10家必訪餐廳|吃貨必備指南 |
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