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茶六燒肉堂春酒菜色豐富嗎? 》公益路餐廳完整攻略|10大人氣店家解析 |
| 時事評論|繪圖 2025/12/22 05:00:38 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人,臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」,從精緻西餐到創意火鍋,從日式丼飯到義式早午餐,每走幾步,就會有完全不同的特色料理餐廳。 這次我特別花了一整個月,實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店,也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準,整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你,找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型,從高質感牛排館到巷弄系早午餐,每一間都有自己獨特的風格。為了讓整體比較更客觀,我依照以下四大面向進行評比,並搭配實際用餐體驗來打分。
整體而言,我希望這份評比不只是「哪家好吃」,而是幫你在不同情境下(約會、家庭聚餐、朋友小聚、商業午餐)都能快速找到合適的選擇。畢竟,美食不只是味覺的滿足,更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段,從火鍋、燒肉到中式料理與早午餐,每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單,橫跨平價、創意、高級各路風格。
一頭牛日式燒肉|炭香濃郁的和牛饗宴,約會聚餐首選
走在公益路上,很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面,搭配昏黃燈光與暖色調的內裝,讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大,但桌距規劃得宜,每桌皆設有獨立排煙設備,烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂,絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網,讓人完全不用分心。整場用餐過程就像一場表演,從視覺、嗅覺到味覺都被滿足。 綜合評分
地址:408臺中市南屯區公益路二段162號電話:04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」,更像是一場五感盛宴。從進門那一刻到最後一道甜點,都能感受到他們對細節的用心。 TANG Zhan 湯棧|文青系火鍋代表,麻香湯底與視覺美感並重
在公益路這條美食戰線上,TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址:408臺中市南屯區公益路二段248號電話:04-22580617 官網:https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度,又結合現代風格與細節服務,讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店,湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店|明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂,那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主,陽光灑進室內,讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧,建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味,選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口,米芯保留微Q口感;而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香,口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹,無論是一個人帶電腦工作、或朋友聚餐,都能找到舒服角落。餐點上桌速度穩定,服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」,很適合想遠離忙碌日常的人。 綜合評分
地址:40861臺中市南屯區公益路二段18號電話:04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇,而是以「剛剛好」的份量與風味,陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店,NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物|平價卻用心的湯頭系火鍋,家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上,加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐,但靠著實在的湯頭與親切的服務,默默吸引許多回頭客。每到用餐時間,總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底,湯頭清澈卻不單薄,越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向,桌距寬敞、座位舒適,帶小孩來也不覺擁擠。店員態度親切,補湯、收盤都很勤快,給人一種「被照顧著」的安心感。 綜合評分
地址:403臺中市西區公益路288號電話:0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤,而是用最簡單的湯頭與新鮮食材,傳遞出家常卻不平凡的溫度。 印月餐廳|中式料理的藝術演繹,宴客與家庭聚會首選
說到臺中公益路的中式料理代表,印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名,把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設,每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏,都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法,讓人感受到「被款待」的尊榮感。 綜合評分
地址:408臺中市南屯區公益路二段818號電話:0422511155 小結語印月餐廳是一間「不只吃飯,更像品味生活」的地方。 KoDō 和牛燒肉|極致職人精神,專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗,一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉,以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址:403臺中市西區公益路260號電話:0423220312 官網:https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳,而是一場體驗。 永心鳳茶|在茶香裡用餐的優雅時光,臺味早午餐的新詮釋
走進 永心鳳茶公益店,彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤,無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」,都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和,對茶品介紹詳盡。上餐節奏剛好,不急不徐。 綜合評分
地址:40360臺中市西區公益路68號三樓(勤美誠品)電話:0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓|老饕級江浙功夫菜,穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主,兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址:408臺中市南屯區公益路二段95號電話:0423202322 官網:https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司|低調奢華的無菜單日料,職人手藝詮釋旬味極致
在熱鬧的公益路上,一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase(無菜單料理) 形式,每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘,節奏緩慢但沉穩。 綜合評分
地址:408臺中市南屯區公益路二段25號電話:0423206368 官網:https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂|人氣爆棚的和牛燒肉聖地,肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳,茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐,價格約落在 $700–$1000 間,份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快,整場用餐流程流暢無壓力。 綜合評分
地址:403臺中市西區公益路268號電話:0423281167 官網:https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質+輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後,臺中公益路不只是一條美食街,而是一段生活風景線。 有的餐廳講究細膩與儀式感,像 一頭牛日式燒肉 與 一笈壽司,讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心,像 加分昆布鍋物、永心鳳茶,讓人明白吃飯不只是為了飽足,而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店,則用穩定的品質與熱絡的氛圍,成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店,構成了公益路最動人的縮影 有華麗的,也有溫柔的;有傳統的,也有創新的。 每一家都在自己的風格裡發光,讓人吃到的不只是料理,而是一種生活的溫度與節奏。 對我而言,這不僅是一場美食旅程,更是一趟關於「臺中味道」的回憶之旅。 FAQ:關於臺中公益路美食常見問題Q1:公益路哪一區的餐廳最集中? Q2:需要提前訂位嗎? 最後的話若要用一句話形容這趟美食之旅,我會說: 一頭牛日式燒肉停車方便嗎? 如果你也和我一樣喜歡用味蕾探索一座城市,那就把這篇公益路美食攻略收藏起來吧。TANG Zhan 湯棧有提供尾牙方案嗎? 無論是約會、慶生、家庭聚餐,或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。TANG Zhan 湯棧份量足夠嗎? 下一餐,不妨從這10家開始。TANG Zhan 湯棧價格合理嗎? 打開手機、約上朋友,讓公益路成為你生活裡最容易抵達的小確幸。一笈壽司整體值得推薦嗎? 如果你有私心愛店,也歡迎留言分享,永心鳳茶用餐環境舒服嗎? 你的推薦,可能讓我下一趟美食旅程變得更精彩。三希樓年末聚餐推薦嗎? A team of KAUST researchers used nematode worms to explore how short-term genetic memories can be inherited across generations. Credit: © 2022 KAUST; Veronica Moraru Researchers created a piRNA tool for gene silencing in nematode worms. The method offers insights into epigenetic memory and holds potential for therapeutic use in humans. A gene-silencing tool could enable new opportunities for advancing basic biomedical research and drug development. The technique draws on the power of small noncoding RNA molecules that normally suppress gene activity. Known as Piwi-interacting RNAs, or piRNAs, these regulatory molecules normally play a critical role in bringing genomic parasites to heel. But geneticist Christian Frøkjær-Jensen and his colleagues at KAUST co-opted this piRNA pathway to deliberately quell the activity of target genes of interest. Working in nematode worms — a common laboratory model for genetics research — Frøkjær-Jensen’s team created synthetic 21-letter RNA sequences that interacted with the natural piRNA machinery to silence intended genes. Proof of Concept and Multiplexed Gene Silencing As a proof of principle, the researchers designed such “guide piRNAs” directed against two genes involved in determining worm sex, thereby skewing the ratio of male-to-female offspring. Using this piRNA-mediated interference mechanism —piRNAi for short — they also silenced many other genes, either alone or in a multiplexed manner. “We have reprogrammed a pathway that normally guards the organism’s genome,” Frøkjær-Jensen says. “Our technique is an important step in enabling precise and scalable biological engineering of a very simple living organism.” What’s more, since the same gene-silencing pathway is found in humans, Frøkjær-Jensen notes, “it is interesting to consider whether piRNAi could be used as a potential therapeutic in people.” Expanding Molecular Tools and Therapeutic Potential Already, other gene-specific silencing tools, including conventional RNA interference and CRISPR-based gene editing, are being used in patients to fix genetic diseases. But these methods do not always work well for all gene targets in worms. The new approach from Frøkjær-Jensen’s team expands the molecular toolkit for gene manipulations and allows for more detailed investigations in the model laboratory species. The researchers have developed a web portal for scientists anywhere to generate their own piRNAi designs. Frøkjær-Jensen’s own research focuses on understanding how short-term genetic memories can be inherited across generations. So, his group examined how long piRNAi-mediated gene silencing might last, from parent to offspring and beyond. Epigenetic Memory and Inheritance As it turned out, different genes could be turned off for different lengths of time, ranging from one to six generations. However, the researchers could also make the gene silencing permanent by depleting the entire piRNA pathway, showing how the same mechanism is needed first to initiate but then also to limit the inherited epigenetic state. “We find these short-term memory systems fascinating,” says Monika Priyadarshini, who developed piRNAi as a graduate student in Frøkjær-Jensen’s lab. “Our tool will help us and others understand how epigenetic memories are passed on, and whether higher organisms such as humans have similar systems.” Reference: “Reprogramming the piRNA pathway for multiplexed and transgenerational gene silencing in C. elegans” by Monika Priyadarshini, Julie Zhouli Ni, Amhed M. Vargas-Velazquez, Sam Guoping Gu and Christian Frøkjær-Jensen, 3 February 2022, Nature Methods. DOI: 10.1038/s41592-021-01369-z Hippocampal dentate gyrus in HSHA mice, showing significant astrocyte activation (GFAP: white) and oxidative stress (8OHdG: green) surrounding the cerebral capillaries (laminin-a4: magenta). Nuclei staining with DAPI (blue). Credit: John Charles Louis Mamo, Lam V et al., 2021, PLOS Biology, CC BY 4.0 Peripherally produced amyloid causes neurodegeneration. Amyloid protein made in the liver can cause neurodegeneration in the brain, according to a new study in the open-access journal PLOS Biology, by John Mamo of Curtin University in Bentley, Australia, and colleagues. Since the protein is thought to be a key contributor to development of Alzheimer’s disease (AD), the results suggest that the liver may play an important role in the onset or progression of the disease. Deposits of amyloid-beta (A-beta) in the brain are one of the pathological hallmarks of AD and are implicated in neurodegeneration in both human patients and animal models of the disease. But A-beta is also present in peripheral organs, and blood levels of A-beta correlate with cerebral amyloid burden and cognitive decline, raising the possibility that peripherally produced a-beta may contribute to the disease. Testing that hypothesis has been difficult, since the brain also produces A-beta, and distinguishing protein from the two sources is challenging. In the current study, the authors surmounted that challenge by developing a mouse that produces human a-beta only in liver cells. They showed that the protein was carried in the blood by triglyceride-rich lipoproteins, just as it is in humans, and passed from the periphery into the brain. They found that mice developed neurodegeneration and brain atrophy, which was accompanied by neurovascular inflammation and dysfunction of cerebral capillaries, both commonly observed with Alzheimer’s disease. Affected mice performed poorly on a learning test that depends on function of the hippocampus, the brain structure that is essential for the formation of new memories. The findings from this study indicate that peripherally derived A-beta has the ability to cause neurodegeneration and suggest that A-beta made in the liver is a potential contributor to human disease. If that contribution is significant, the findings may have major implications for understanding Alzheimer’s disease. To date, most models of the disease have focused on brain overproduction of A-beta, which mimics the rare genetic cases of human Alzheimer’s. But for the vast majority of AD cases, overproduction of A-beta in the brain is not thought to be central to the disease etiology. Instead, lifestyle factors may play a more important role, including a high-fat diet, which might accelerate liver production of A-beta. The effects of peripheral A-beta on brain capillaries may be critical in the disease process, Mamo adds. “While further studies are now needed, this finding shows the abundance of these toxic protein deposits in the blood could potentially be addressed through a person’s diet and some drugs that could specifically target lipoprotein amyloid, therefore reducing their risk or slowing the progression of Alzheimer’s disease.” Reference: “Synthesis of human amyloid restricted to liver results in an Alzheimer disease–like neurodegenerative phenotype” by Virginie Lam, Ryusuke Takechi, Mark J. Hackett, Roslyn Francis, Michael Bynevelt, Liesl M. Celliers, Michael Nesbit, Somayra Mamsa, Frank Arfuso, Sukanya Das, Frank Koentgen, Maree Hagan, Lincoln Codd, Kirsty Richardson, Brenton O’Mara, Rainer K. Scharli, Laurence Morandeau, Jonathan Gauntlett, Christopher Leatherday, Jan Boucek, John C. L. Mamo, 14 September 2021, PLOS Biology. DOI: 10.1371/journal.pbio.3001358 Funding: This work was funded by the National Health and Medical Research Council (GNT1135590 (RT), GNT1064567 (JM), GNT1156582 (VL)), and Western Australian Department of Health (RT). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript. Recent research on tardigrades uncovers a complex genetic basis for their extreme resilience, challenging previous assumptions about their ecological adaptations and pointing to independent evolutionary events in their anhydrobiosis capability. Tardigrades may be nature’s ultimate survivors. While these tiny, nearly translucent animals are easily overlooked, they represent a diverse group that has successfully colonized freshwater, marine, and terrestrial environments on every continent, including Antarctica. Commonly known as “water bears”, these unusual creatures may be among the most resilient organisms on the planet thanks to their unparalleled ability to survive extreme conditions, with various species being resistant to drought, high doses of radiation, low oxygen environments, and both high and low temperatures and pressures. While numerous genes have been suggested to contribute to this extremotolerance, a comprehensive understanding of the origins and history of these unique adaptations has remained elusive. In a new study published in Genome Biology and Evolution, scientists at Keio University Institute for Advanced Biosciences, the University of Oslo Natural History Museum, and the University of Bristol reveal a surprisingly intricate network of gene duplications and losses associated with tardigrade extremotolerance, highlighting the complex genetic landscape that drives modern tardigrade ecology. Understanding Tardigrade Gene Families As one form of extremotolerance, tardigrades can survive almost complete desiccation by entering a dormant state called anhydrobiosis (i.e., life without water), which allows them to reversibly halt their metabolism. Multiple tardigrade-specific gene families were previously found to be associated with anhydrobiosis. Three of these gene families are referred to as cytosolic, mitochondrial, and secretory abundant heat soluble proteins (CAHS, MAHS, and SAHS, respectively) based on the cellular location in which the proteins are expressed. Some tardigrades appear to possess a variant pathway that involves two families of abundant heat-soluble proteins first identified in the tardigrade Echiniscus testudo and usually referred to as EtAHS alpha and beta. A photograph of the tardigrade Ramazzottius varieornatus, in the center of a phylogeny of CAHS, the largest of the six desiccation-related protein families analyzed in this study. Credit: Kazuharu Arakawa, Keio Institute of Advanced Biosciences Tardigrades also possess stress resistance genes that can be found in animals more broadly, such as the meiotic recombination 11 (MRE11) gene, which has been implicated in desiccation tolerance in other animals. Unfortunately, since the identification of these gene families, limited information has been available from most tardigrade lineages, making it difficult to draw conclusions on their origins, history, and ecological implications. Investigating Tardigrade Evolution To better shed light on the evolution of tardigrade extremotolerance, the authors of the new study—James Fleming, Davide Pisani, and Kazuharu Arakawa—identified sequences from these six gene families across 13 tardigrade genera, including representatives from both of the major tardigrade lineages, the Eutardigrades, and Heterotardigrades. Their analysis revealed 74 CAHS, 8 MAHS, 29 SAHS, 22 EtAHS alpha, 18 EtAHS beta, and 21 MRE11 sequences, allowing them to build the first tardigrade phylogenies for these gene families. As resistance to desiccation is likely to have emerged as an adaptation to terrestrial environments, the authors assumed that they would find a link between gene duplications and losses in these gene families and habitat changes within tardigrades. “When we began the work, we expected to find that each clade would be clearly grouped around ancient duplications, with few independent losses. That would help us easily tie them to an understanding of modern habitats and ecology,” says the study’s lead author, James Fleming. “It’s an intuitive hypothesis,” he continues, “that the evolution of the duplications of these desiccation-related genes should, in theory, contain remnants of the ecological history of these organisms, although, in reality, this turned out to be overly simplistic.” Instead, the authors were surprised by the sheer number of independent duplications of heat-soluble genes, which painted a much more complex picture of anhydrobiosis-related gene evolution. Notably, however, there was no clear link between strongly anhydrobiotic species and the number of anhydrobiosis-related genes a species possessed. “What we found was far more exciting,” says Fleming, “a complex network of independent gains and losses that does not necessarily correlate to modern terrestrial species ecologies.” Independent Adaptations in Tardigrade Lineages Despite the lack of a relationship between gene duplications and tardigrade ecology, the study did provide crucial insight into the major transitions that led to the acquisition of anhydrobiosis. The distinct distributions of gene families across the two major groups of tardigrades—CAHS, MAHS, and SAHS in the Eutardigrades and EtAHS alpha and beta in the Heterotardigrades—suggest that two independent transitions from marine to limno-terrestrial environments occurred within tardigrades, once in the Eutardigrade ancestor and once within the Heterotardigrades. This research marks a significant step forward in our understanding of the evolution of anhydrobiosis in tardigrades. It also provides a foundation for future studies into tardigrade extremotolerance, which will require the continued development of genomic resources from more diverse tardigrade lineages. “We, unfortunately, have no representatives from several important families, such as the Isohypsibiidae, and this does limit how firmly we can stand by our conclusions,” notes Fleming. “With more freshwater and marine tardigrade samples, we will be better able to appreciate the adaptations of terrestrial members of the group.” Unfortunately, some tardigrades can be especially elusive, presenting a major obstacle to such studies. As an example, Tanarctus bubulubus, one of Fleming’s favorite tardigrades, is too small to see with the naked eye and is found only in sediment in the North Atlantic at depths of around 150 m. “Hopefully,” says Fleming, “large-scale sequencing initiatives through the Earth Biogenome Project will steadily bridge this gap in our understanding, and it’s an effort I’m excited to see continue.” Reference: “The Evolution of Temperature and Desiccation-Related Protein Families in Tardigrada Reveals a Complex Acquisition of Extremotolerance” by James F Fleming, Davide Pisani and Kazuharu Arakawa, 29 November 2023, Genome Biology and Evolution. DOI: 10.1093/gbe/evad217 RRG455KLJIEVEWWF NINI 尼尼台中店整體值得推薦嗎? 》公益路美食2026最新版|10家必吃大評比永心鳳茶家庭聚餐合適嗎? 》公益路10大美食推薦|從燒肉到火鍋全攻略加分100%浜中特選昆布鍋物包廂適合尾牙嗎? 》公益路餐廳怎麼挑?10家人氣店幫你選 |
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