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文章數：87 |
 永心鳳茶適合辦尾牙嗎？》公益路必吃Top10｜美食路線一次規劃好 |
| 休閒生活｜旅人手札 2026/04/20 12:44:02 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人，臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」，從精緻西餐到創意火鍋，從日式丼飯到義式早午餐，每走幾步，就會有完全不同的特色料理餐廳。 這次我特別花了一整個月，實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店，也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準，整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你，找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型，從高質感牛排館到巷弄系早午餐，每一間都有自己獨特的風格。為了讓整體比較更客觀，我依照以下四大面向進行評比，並搭配實際用餐體驗來打分。
整體而言，我希望這份評比不只是「哪家好吃」，而是幫你在不同情境下（約會、家庭聚餐、朋友小聚、商業午餐）都能快速找到合適的選擇。畢竟，美食不只是味覺的滿足，更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段，從火鍋、燒肉到中式料理與早午餐，每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單，橫跨平價、創意、高級各路風格。
一頭牛日式燒肉｜炭香濃郁的和牛饗宴，約會聚餐首選
走在公益路上，很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面，搭配昏黃燈光與暖色調的內裝，讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大，但桌距規劃得宜，每桌皆設有獨立排煙設備，烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂，絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網，讓人完全不用分心。整場用餐過程就像一場表演，從視覺、嗅覺到味覺都被滿足。 綜合評分
地址：408臺中市南屯區公益路二段162號電話：04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」，更像是一場五感盛宴。從進門那一刻到最後一道甜點，都能感受到他們對細節的用心。 TANG Zhan 湯棧｜文青系火鍋代表，麻香湯底與視覺美感並重
在公益路這條美食戰線上，TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址：408臺中市南屯區公益路二段248號電話：04-22580617 官網：https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度，又結合現代風格與細節服務，讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店，湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店｜明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂，那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主，陽光灑進室內，讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧，建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味，選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口，米芯保留微Q口感；而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香，口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹，無論是一個人帶電腦工作、或朋友聚餐，都能找到舒服角落。餐點上桌速度穩定，服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」，很適合想遠離忙碌日常的人。 綜合評分
地址：40861臺中市南屯區公益路二段18號電話：04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇，而是以「剛剛好」的份量與風味，陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店，NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物｜平價卻用心的湯頭系火鍋，家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上，加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐，但靠著實在的湯頭與親切的服務，默默吸引許多回頭客。每到用餐時間，總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底，湯頭清澈卻不單薄，越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向，桌距寬敞、座位舒適，帶小孩來也不覺擁擠。店員態度親切，補湯、收盤都很勤快，給人一種「被照顧著」的安心感。 綜合評分
地址：403臺中市西區公益路288號電話：0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤，而是用最簡單的湯頭與新鮮食材，傳遞出家常卻不平凡的溫度。 印月餐廳｜中式料理的藝術演繹，宴客與家庭聚會首選
說到臺中公益路的中式料理代表，印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名，把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設，每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏，都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法，讓人感受到「被款待」的尊榮感。 綜合評分
地址：408臺中市南屯區公益路二段818號電話：0422511155 小結語印月餐廳是一間「不只吃飯，更像品味生活」的地方。 KoDō 和牛燒肉｜極致職人精神，專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗，一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉，以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址：403臺中市西區公益路260號電話：0423220312 官網：https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳，而是一場體驗。 永心鳳茶｜在茶香裡用餐的優雅時光，臺味早午餐的新詮釋
走進 永心鳳茶公益店，彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤，無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」，都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和，對茶品介紹詳盡。上餐節奏剛好，不急不徐。 綜合評分
地址：40360臺中市西區公益路68號三樓(勤美誠品)電話：0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓｜老饕級江浙功夫菜，穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主，兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址：408臺中市南屯區公益路二段95號電話：0423202322 官網：https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司｜低調奢華的無菜單日料，職人手藝詮釋旬味極致
在熱鬧的公益路上，一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase（無菜單料理） 形式，每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘，節奏緩慢但沉穩。 綜合評分
地址：408臺中市南屯區公益路二段25號電話：0423206368 官網：https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂｜人氣爆棚的和牛燒肉聖地，肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳，茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐，價格約落在 $700–$1000 間，份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快，整場用餐流程流暢無壓力。 綜合評分
地址：403臺中市西區公益路268號電話：0423281167 官網：https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質＋輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後，臺中公益路不只是一條美食街，而是一段生活風景線。 有的餐廳講究細膩與儀式感，像 一頭牛日式燒肉 與 一笈壽司，讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心，像 加分昆布鍋物、永心鳳茶，讓人明白吃飯不只是為了飽足，而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店，則用穩定的品質與熱絡的氛圍，成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店，構成了公益路最動人的縮影 有華麗的，也有溫柔的；有傳統的，也有創新的。 每一家都在自己的風格裡發光，讓人吃到的不只是料理，而是一種生活的溫度與節奏。 對我而言，這不僅是一場美食旅程，更是一趟關於「臺中味道」的回憶之旅。 FAQ：關於臺中公益路美食常見問題Q1：公益路哪一區的餐廳最集中？ Q2：需要提前訂位嗎？ 最後的話若要用一句話形容這趟美食之旅，我會說： 一頭牛日式燒肉家庭過節聚會適合嗎？ 如果你也和我一樣喜歡用味蕾探索一座城市，那就把這篇公益路美食攻略收藏起來吧。一頭牛日式燒肉食材新鮮嗎？ 無論是約會、慶生、家庭聚餐，或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。加分100%浜中特選昆布鍋物清淡口味適合嗎？ 下一餐，不妨從這10家開始。加分100%浜中特選昆布鍋物套餐劃算嗎？ 打開手機、約上朋友，讓公益路成為你生活裡最容易抵達的小確幸。三希樓慶生氛圍夠嗎？ 如果你有私心愛店，也歡迎留言分享，NINI 尼尼臺中店海鮮表現如何？ 你的推薦，可能讓我下一趟美食旅程變得更精彩。TANG Zhan 湯棧慶生氛圍夠嗎？ Photographic examples of look-alike pairs (LALs) used in this study. Credit: François Brunelle A study on unrelated lookalikes found shared genetic variants that explain facial similarity, offering potential applications in forensics and genetic diagnosis. A collection of photos of genetically unrelated lookalikes, along with DNA analysis, has revealed that strong facial similarity is linked with shared genetic variants. The work will be published today (August 23rd) in the journal Cell Reports. “Our study provides a rare insight into human likeness by showing that people with extreme lookalike faces share common genotypes, whereas they are discordant at the epigenome and microbiome levels,” says senior author Manel Estellerof the Josep Carreras Leukaemia Research Institute in Barcelona, Spain. “Genomics clusters them together, and the rest sets them apart.” The number of people identified online as doubles or virtual twins who are genetically unrelated has increased with the expansion of the World Wide Web and the possibility of exchanging pictures of humans across the planet. In the new study, Esteller and his research team set out to characterize, on a molecular level, random human beings that objectively share facial features. To do this, they recruited human doubles from the photographic work of François Brunelle. He is a Canadian artist who has been obtaining worldwide pictures of lookalikes since 1999. They obtained headshot pictures of 32 lookalike couples. The scientists determined an objective measure of likeness for the pairs using three different facial recognition algorithms. DNA Analysis Reveals Shared Genotypes Additionally, the participants completed a comprehensive biometric and lifestyle questionnaire and provided saliva DNA for multiomics analysis. “This unique set of samples has allowed us to study how genomics, epigenomics, and microbiomics can contribute to human resemblance,” Esteller says. Overall, the results revealed that these individuals share similar genotypes, but differ in their DNA methylation and microbiome landscapes. Half of the lookalike pairs were clustered together by all three algorithms. Genetic analysis revealed that 9 of these 16 pairs clustered together, based on 19,277 common single-nucleotide polymorphisms. Physical and Behavioral Correlations Furthermore, physical traits such as weight and height, as well as behavioral traits such as smoking and education, were correlated in lookalike pairs. Taken together, the findings suggest that shared genetic variation not only relates to similar physical appearance, but may also influence common habits and behavior. “We provided a unique insight into the molecular characteristics that potentially influence the construction of the human face,” Esteller says. “We suggest that these same determinants correlate with both physical and behavioral attributes that constitute human beings.” There were a few study limitations. These include the small sample size, the use of 2D black-and-white images, and the predominance of European participants. Despite these caveats, the findings may provide a molecular basis for future applications in various fields such as biomedicine, evolution, and forensics. “These results will have future implications in forensic medicine—reconstructing the criminal’s face from DNA—and in genetic diagnosis—the photo of the patient’s face will already give you clues as to which genome he or she has,” Esteller says. “Through collaborative efforts, the ultimate challenge would be to predict the human face structure based on the individual’s multiomics landscape.” Reference: “Look-alike humans identified by facial recognition algorithms show genetic similarities” by Ricky S. Joshi, Maria Rigau, Carlos A. García-Prieto, Manuel Castro de Moura, David Piñeyro, Sebastian Moran, Veronica Davalos, Pablo Carrión, Manuel Ferrando-Bernal, Iñigo Olalde, Carles Lalueza-Fox, Arcadi Navarro, Carles Fernández-Tena, Decky Aspandi, Federico M. Sukno, Xavier Binefa, Alfonso Valencia and Manel Esteller, 23 August 2022, Cell Reports. DOI: 10.1016/j.celrep.2022.111257 This work was funded by the governments of Catalonia and Spain, as well as the Cellex Foundation. Declarations of interest can be found in the paper. Pseudomonas bacteria deploy their nano-spearguns when damaged by a sharp tip (15,000 x magnification). Credit: University of Basel, Biozentrum/SNI Nano Imaging Lab Researchers found that Pseudomonas aeruginosa bacteria rapidly assemble their T6SS nanoweapons in response to outer membrane damage, enabling precise retaliation against competitors.Some bacteria deploy tiny spearguns to retaliate against rival attacks. Researchers at the University of Basel simulated these attacks by poking bacteria with an ultra-sharp tip. Through this approach, they discovered that bacteria assemble their nanoweapons in response to cell envelope damage and rapidly strike back with high precision. In the microbial world, peaceful coexistence coexists with fierce competition for nutrients and space. Certain bacteria outcompete rivals and defend against attackers by injecting them with a lethal cocktail using tiny, nano-sized spearguns known as type VI secretion systems (T6SS). Bacteria respond to cell envelope damage The research group led by Professor Marek Basler at the Biozentrum, University of Basel, has been studying the T6SS of different bacterial species for many years. “We knew that Pseudomonas aeruginosa uses its T6SS to fire back when attacked”, explains Basler. “But we did not know what exactly triggers the assembly of the nano-speargun: the contact with neighbors, toxic molecules, or simply cell damage?” In close collaboration with Roderick Lim, Argovia Professor for Nanobiology at the Biozentrum and the Swiss Nanoscience Institute (SNI), the researchers have now demonstrated: Pseudomonas aeruginosa responds to ruptures in the outer membrane – initiated by mechanical force, such as poking with a sharp tip. The study has been published in Science Advances. Puncturing bacterial envelope with a tiny “needle” Roderick Lim’s lab has long-standing expertise in atomic force microscopy (AFM) technology. “Using AFM, we have been able to mimic a bacterial T6SS attack,” says Mitchell Brüderlin, PhD student at the SNI PhD School and first author of the study. “With the needle-like, ultra-sharp AFM tip, we can touch the bacterial surface and, with gradually increasing the pressure, puncture the outer and the inner membrane in a controlled manner.” In combination with fluorescence microscopy, the researchers revealed that the bacteria respond to outer membrane damage. “Within ten seconds the bacteria assemble their T6SS, often repeatedly, at the site of damage and fire back with pinpoint accuracy,” adds Basler. “Our work clearly shows that breaking the outer membrane is necessary and sufficient to trigger T6SS assembly.” New insights into bacterial defense mechanisms The biggest challenge for the researchers was the size and the shape of the bacteria. “So far, we have only used the AFM to study eukaryotic cells, including human cells,” explains Lim. “But Pseudomonas bacteria are more than ten times smaller than human cells, so it was demanding to poke them at a specific site.” In the microbial ecosystem, survival is all about strategy, and Pseudomonas aeruginosa has certainly mastered the art of defense. “The targeted and swift retaliation against local attacks minimizes misfiring and optimizes the cost-benefit ratio”, says Basler. This clever tactic gives Pseudomonas a survival advantage, enabling it to incapacitate attackers and thrive in diverse and often challenging environments. Reference: “Pseudomonas aeruginosa assembles H1-T6SS in response to physical and chemical damage of the outer membrane” by Mitchell Brüderlin, Maxim Kolesnikov, Florian Röthlin, Roderick Y. H. Lim and Marek Basler, 5 March 2025, Science Advances. DOI: 10.1126/sciadv.adr1713 The enteric nervous system (ENS), often called the second brain, plays a crucial role in digestion, immunity, and communication with the brain. Researchers have discovered that ENS development continues after birth and includes neurons derived from mesoderm, challenging long-held scientific beliefs and opening avenues for potential new treatments for aging and gastrointestinal diseases. Discoveries May Pave the Way for Improved Therapies for Gastrointestinal Issues Following your gut. Losing your appetite. A gutsy move. Though we often consider the gut as merely a digestive tool, these common expressions reflect the central role the gut plays in a much wider range of essential functions. The entire digestive tract is lined by the enteric nervous system (ENS), a vast network of millions of neurons and glial cells—the two primary cell types also found in the central nervous system. While often called the second brain, the ENS not only generates the same neurotransmitters but actually predates the evolution of the central nervous system in the brain. The functions of the ENS are crucial to life and extend far beyond digestion, as it regulates immunity, gut secretions, and enables complex, bi-directional communication between the gut and the brain. This is why a happy gut co-exists with a happy brain, and why digestive issues can lead to changes in mood and behavior. Since the mid-20th century, scientists have believed that the ENS is derived from the neural crest before birth and remains unchanged after. Now, in a paper published in the journal eLife, researchers at Beth Israel Deaconess Medical Center (BIDMC) present a completely new paradigm describing a developmental pathway by which ENS development continues after birth in mice and human tissue samples. This discovery overturns decades of scientific dogma on the fundamental biology of neuroscience and of ENS, by showing evidence for the first time of a non-ectodermal and a mesodermal origin for large numbers of enteric neurons born after birth. The findings show the relevance of these neurons to the maturation and aging of the ENS in health and disease. The Aging Process and ENS Neuron Evolution “These results indicate for the first time that the mesoderm is an important source of neurons in the second largest nervous system of the body,” said Subhash Kulkarni, Ph.D., a staff scientist at BIDMC and an assistant professor in the Division of Medical Sciences at Harvard Medical School. “How we mature and how we age is central to our understanding of health and disease in our rapidly aging population. The increasing proportion of neurons of mesodermal lineage is a natural consequence of maturation and aging; further, this lineage can be expected to have distinct vulnerabilities to disease.” Using transgenic mice models, high-resolution microscopy, and genetic analyses, Kulkarni and colleagues analyzed the ENS neuronal populations in adult mice and human tissues. Using mice models, the team found that while the early post-natal ENS cells were from the expected neural crest lineage, that pattern changed rapidly as the animal matured. Kulkarni and colleagues documented the arrival and continual expansion of a novel population of enteric neurons that were derived from the mesoderm—the same lineage that gives rise to the muscle and heart cells. This newly discovered population of mesoderm-derived neurons expanded with age, such that they comprised a third of all enteric neurons in adolescent mice, half of all enteric neurons in adult mice, and then eventually outnumbered the original neural crest-derived population of enteric neurons in aging mice. By assessing the molecular signature of these neurons, the team identified new cellular markers that were used to identify this population of mesoderm-derived neurons in human gut tissue. These markers also provided pharmacological targets, which the researchers used to not only manipulate the proportions of the mesodermal neurons in adolescent mice but also reduce their dominant proportions in the aging mouse gut to cure age-associated slowing of gut movement. A Paradigm Shift in Neuroscience and Clinical Medicine “We can now work to understand how these findings can be translated into human systems to provide a disease-modifying cure to aging patients whose chief complaint often includes diseases of the GI tract,” added Kulkarni. “By reversing one of the biggest dogmas of neuroscience, we are now in uncharted territory and, at the same time, have a huge opportunity to understand this hidden basic, translational, and clinical biology of neurons. The newly discovered lineage of neurons presents us with potential new drug targets that could help large populations of patients.” Reference: “Age-associated changes in lineage composition of the enteric nervous system regulate gut health and disease” by Subhash Kulkarni, Monalee Saha, Jared Slosberg, Alpana Singh, Sushma Nagaraj, Laren Becker, Chengxiu Zhang, Alicia Bukowski, Zhuolun Wang, Guosheng Liu, Jenna Leser, Mithra Kumar, Shriya Bakhshi, Matthew Anderson, Mark Lewandoski, Elizabeth Vincent, Loyal A. Goff and Pankaj Jay Pasricha, 7 August 2023, eLife. DOI: 10.7554/eLife.88051.1 Co-authors included Monalee Saha, Jared Slosberg, Alpana Singh, Sushma Nagaraj, Chengxiu Zhang, Alicia Bukowski, Zhuolun Wang, Guosheng Liu, Jenna Leser, Mithra Kumar, Shriya Bakhshi, Elizabeth Vincent, and Loyal A. Goff of Johns Hopkins University School of Medicine; Laren Becker and of Stanford University School of Medicine; Matthew Anderson and Mark Lewandoski of Center for Cancer Research, National Cancer Institute; and Pankaj Jay Pasricha of the Mayo Clinic. The microscopy was performed on the Ross Imaging Core at the Hopkins Conte Digestive Disease Center at the Johns Hopkins University (P30DK089502) using the Olympus FV 3000rs (procured with the NIH-NIDDK S10 OD025244 grant). The 10X Genomics Chromium processing for scRNAseq was performed at the GRCF Core and the sequencing was performed at the CIDR core at the Johns Hopkins University. This work was supported through a grant from the Ludwig Foundation, a grant from the NIA (R01AG066768), a pilot award from the Hopkins Digestive Diseases Basic & Translational Research Core Center grant (P30DK089502), a pilot award from the Diacomp initiative through Augusta University; a Johns Hopkins Catalyst Award; the Maryland Genetics, Epidemiology, and Medicine training program sponsored by the Burroughs Welcome Fund; the Hopkins Conte Digestive Disease Center at the Johns Hopkins University (P30DK089502); NIDDK (R01DK080920); the Maryland Stem Cell Research Foundation (MSCRF130005), and a grant from the AMOS family. RRG455KLJIEVEWWF |
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