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永心鳳茶清淡口味適合嗎?》台中公益路聚餐推薦|10大類型餐廳評比 |
| 休閒生活|旅人手札 2026/04/21 14:03:32 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人,臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」,從精緻西餐到創意火鍋,從日式丼飯到義式早午餐,每走幾步,就會有完全不同的特色料理餐廳。 這次我特別花了一整個月,實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店,也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準,整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你,找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型,從高質感牛排館到巷弄系早午餐,每一間都有自己獨特的風格。為了讓整體比較更客觀,我依照以下四大面向進行評比,並搭配實際用餐體驗來打分。
整體而言,我希望這份評比不只是「哪家好吃」,而是幫你在不同情境下(約會、家庭聚餐、朋友小聚、商業午餐)都能快速找到合適的選擇。畢竟,美食不只是味覺的滿足,更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段,從火鍋、燒肉到中式料理與早午餐,每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單,橫跨平價、創意、高級各路風格。
一頭牛日式燒肉|炭香濃郁的和牛饗宴,約會聚餐首選
走在公益路上,很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面,搭配昏黃燈光與暖色調的內裝,讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大,但桌距規劃得宜,每桌皆設有獨立排煙設備,烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂,絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網,讓人完全不用分心。整場用餐過程就像一場表演,從視覺、嗅覺到味覺都被滿足。 綜合評分
地址:408臺中市南屯區公益路二段162號電話:04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」,更像是一場五感盛宴。從進門那一刻到最後一道甜點,都能感受到他們對細節的用心。 TANG Zhan 湯棧|文青系火鍋代表,麻香湯底與視覺美感並重
在公益路這條美食戰線上,TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址:408臺中市南屯區公益路二段248號電話:04-22580617 官網:https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度,又結合現代風格與細節服務,讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店,湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店|明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂,那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主,陽光灑進室內,讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧,建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味,選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口,米芯保留微Q口感;而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香,口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹,無論是一個人帶電腦工作、或朋友聚餐,都能找到舒服角落。餐點上桌速度穩定,服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」,很適合想遠離忙碌日常的人。 綜合評分
地址:40861臺中市南屯區公益路二段18號電話:04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇,而是以「剛剛好」的份量與風味,陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店,NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物|平價卻用心的湯頭系火鍋,家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上,加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐,但靠著實在的湯頭與親切的服務,默默吸引許多回頭客。每到用餐時間,總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底,湯頭清澈卻不單薄,越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向,桌距寬敞、座位舒適,帶小孩來也不覺擁擠。店員態度親切,補湯、收盤都很勤快,給人一種「被照顧著」的安心感。 綜合評分
地址:403臺中市西區公益路288號電話:0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤,而是用最簡單的湯頭與新鮮食材,傳遞出家常卻不平凡的溫度。 印月餐廳|中式料理的藝術演繹,宴客與家庭聚會首選
說到臺中公益路的中式料理代表,印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名,把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設,每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏,都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法,讓人感受到「被款待」的尊榮感。 綜合評分
地址:408臺中市南屯區公益路二段818號電話:0422511155 小結語印月餐廳是一間「不只吃飯,更像品味生活」的地方。 KoDō 和牛燒肉|極致職人精神,專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗,一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉,以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址:403臺中市西區公益路260號電話:0423220312 官網:https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳,而是一場體驗。 永心鳳茶|在茶香裡用餐的優雅時光,臺味早午餐的新詮釋
走進 永心鳳茶公益店,彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤,無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」,都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和,對茶品介紹詳盡。上餐節奏剛好,不急不徐。 綜合評分
地址:40360臺中市西區公益路68號三樓(勤美誠品)電話:0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓|老饕級江浙功夫菜,穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主,兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址:408臺中市南屯區公益路二段95號電話:0423202322 官網:https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司|低調奢華的無菜單日料,職人手藝詮釋旬味極致
在熱鬧的公益路上,一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase(無菜單料理) 形式,每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘,節奏緩慢但沉穩。 綜合評分
地址:408臺中市南屯區公益路二段25號電話:0423206368 官網:https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂|人氣爆棚的和牛燒肉聖地,肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳,茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐,價格約落在 $700–$1000 間,份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快,整場用餐流程流暢無壓力。 綜合評分
地址:403臺中市西區公益路268號電話:0423281167 官網:https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質+輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後,臺中公益路不只是一條美食街,而是一段生活風景線。 有的餐廳講究細膩與儀式感,像 一頭牛日式燒肉 與 一笈壽司,讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心,像 加分昆布鍋物、永心鳳茶,讓人明白吃飯不只是為了飽足,而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店,則用穩定的品質與熱絡的氛圍,成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店,構成了公益路最動人的縮影 有華麗的,也有溫柔的;有傳統的,也有創新的。 每一家都在自己的風格裡發光,讓人吃到的不只是料理,而是一種生活的溫度與節奏。 對我而言,這不僅是一場美食旅程,更是一趟關於「臺中味道」的回憶之旅。 FAQ:關於臺中公益路美食常見問題Q1:公益路哪一區的餐廳最集中? Q2:需要提前訂位嗎? 最後的話若要用一句話形容這趟美食之旅,我會說: 加分100%浜中特選昆布鍋物適合辦尾牙嗎? 如果你也和我一樣喜歡用味蕾探索一座城市,那就把這篇公益路美食攻略收藏起來吧。加分100%浜中特選昆布鍋物長官聚餐合適嗎? 無論是約會、慶生、家庭聚餐,或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。TANG Zhan 湯棧海鮮表現如何? 下一餐,不妨從這10家開始。印月餐廳慶生氣氛夠嗎? 打開手機、約上朋友,讓公益路成為你生活裡最容易抵達的小確幸。TANG Zhan 湯棧尾牙拍照效果好嗎? 如果你有私心愛店,也歡迎留言分享,印月餐廳必點有哪些? 你的推薦,可能讓我下一趟美食旅程變得更精彩。永心鳳茶慶生氛圍夠嗎? A groundbreaking study by UC San Diego researchers has uncovered thousands of new bile acids, shedding light on their critical roles beyond digestion and highlighting their potential in treating various diseases. This discovery marks a significant step forward in our understanding of the gut microbiome’s communication with the body and human metabolism. Scientists at the Skaggs School of Pharmacy and Pharmaceutical Sciences, part of the University of California San Diego, have discovered thousands of new bile acids. These molecules are used by our gut microbiome to communicate with the rest of the body. “Bile acids are a key component of the language of the gut microbiome, and finding this many new types radically expands our vocabulary for understanding what our gut microbes do and how they do it,” said senior author Pieter Dorrestein, Ph.D., professor at Skaggs School of Pharmacy and Pharmaceutical Sciences and professor of pharmacology and pediatrics at UC San Diego School of Medicine. “It’s like going from ‘See Spot Run’ to Shakespeare.” Unraveling Microbial Languages The results, as described by study co-author and bile acids expert Lee Hagey, Ph.D., are akin to a molecular Rosetta stone, providing previously unknown insight into the biochemical language microbes use to influence distant organ systems. Bile acids originate in the liver, are stored in the gallbladder and ultimately released into the intestine, where they are deployed to aid in digestion following the consumption of a meal. The microbes in our gut metabolize the bile acids produced by the liver, changing them into a vast array of different molecules called secondary bile acids, which tend to be easier for the body to absorb. Until now, the rich diversity and range of functions of secondary bile acids have been underappreciated by scientists. This research was completed by members of the Dorrestein lab (from left to right: Helena Mannochio-Russo, Ipsita Mohanty, Lee Hagey, and Pieter Dorrestein). Credit: UC San Diego Health Sciences “When I started working in the lab, there were about a few hundred known bile acids,” said study co-author Ipsita Mohanty, Ph.D., a postdoctoral researcher in the Dorrestein lab. “Now we’ve discovered thousands more, and we’re also working toward realizing that these bile acids do so much more than just help with digestion.” In addition to aiding digestion, bile acids are also important signaling molecules that help regulate the immune system and serve important metabolic functions, such as controlling lipid and glucose metabolism. These molecules also help explain how microbes in the gut are able to influence distant organ systems. “Because of their interaction with our microbiome, the influence of bile acids spreads far beyond the digestive system, and so could the diseases we treat with them – the list of diseases related to bile acids is a mile long, and there are several FDA approvals for these kinds of acids as treatments,” said co-author Helena Mannochio-Russo, Ph.D., also a postdoctoral researcher in the Dorrestein lab. Helena Mannochio-Russo, shown in this image, and Ipsita Mohanty collaborated to conduct mass spectrometry experiments that led to the identification of thousands of new bile acids, a type of molecule used by microbes to communicate. Credit: UC San Diego Health Sciences Collaborative Efforts and Future Directions In order to discover these molecules, the researchers leveraged the unique resources of UC San Diego. Dorrestein is director of the Collaborative Microbial Metabolite Center (CMMC), a first-of-its-kind collaboration between UC San Diego and UC Riverside that seeks to gather and centralize information about the metabolites that microbes produce to help researchers learn more about their impact on human health and the environment. “In other areas of biology like genomics, sharing data is common, but there hasn’t been an infrastructure in place for microbial metabolomics researchers to share data until now,” said Dorrestein “Ultimately these breakthroughs are the result of a convergence of collaboration and computing power, and we expect many more breakthroughs to come out of the CMMC.” Earlier this year, the team debuted a new tool that can instantly match microbes to the metabolites they produce. The present study is the first of potentially many studies to utilize the tool for specific types of molecules. The researchers next hope to explore the specific functions of their newly discovered bile acids as well as use their approach on other types of biomolecules, such as lipids or other kinds of acids. “We’re rewriting the textbook of human metabolism,” said Dorrestein. “If you’d have spoken to me a few years ago, I would have said we were decades away from solving this puzzle, but now, it could happen within five years. It’s really a remarkable change in our capabilities, and we believe it’s going to revolutionize the way we approach disease.” Reference: “The underappreciated diversity of bile acid modifications” by Ipsita Mohanty, Helena Mannochio-Russo, Joshua V. Schweer, Yasin El Abiead, Wout Bittremieux, Shipei Xing, Robin Schmid, Simone Zuffa, Felipe Vasquez, Valentina B. Muti, Jasmine Zemlin, Omar E. Tovar-Herrera, Sarah Moraïs, Dhimant Desai, Shantu Amin, Imhoi Koo, Christoph W. Turck, Itzhak Mizrahi, Penny M. Kris-Etherton, Kristina S. Petersen, Jennifer A. Fleming, Tao Huan, Andrew D. Patterson, Dionicio Siegel, Lee R. Hagey, Mingxun Wang, Allegra T. Aron and Pieter C. Dorrestein, 11 March 2024, Cell. DOI: 10.1016/j.cell.2024.02.019 This study was funded, in part, by the National Institutes of Health (grants U24DK133658, R01GM107550, U19AG063744, U01DK119702, S10OD021750), the Biotechnology and Biological Sciences Research Council and National Science Foundation (grant BBSRC/NSF 2152526), the Gordon & Betty Moore Foundation, Natural Sciences and Engineering Research Council of Canada (grant RGPIN-2020-04895). Researchers at the University of Hawai‘i at Mānoa discovered that the virus FloV-SA2 encodes a ribosomal protein, eL40, essential for the production of ribosomes, which translate genetic information into proteins. This marks the first time a eukaryotic virus has been found to possess such a capability, shedding new light on the complexity of virus-host interactions. Credit: SciTechDaily.com A virus that infects ocean phytoplankton was found to encode a ribosomal protein, a first for eukaryotic viruses. Researchers believe this aids the virus in replication. Researchers at the University of Hawai‘i at Mānoa have identified a virus, FloV-SA2, that encodes one of the essential proteins needed for ribosome production. Ribosomes are critical cellular structures that convert genetic information into proteins—the fundamental building blocks of life. This marks the first discovery of a eukaryotic virus (a virus that infects organisms such as plants, animals, or fungi) capable of encoding such a protein. Viruses are essentially packets of genetic material encased in a protein shell. They reproduce by invading a host cell and commandeering its replication machinery to produce more viruses. While simpler viruses rely almost entirely on the host cell for this process, larger and more complex viruses often produce a variety of proteins to assist in their replication. In cells, ribosomes (shown as grey blobs) read RNA messages and convert the information into proteins. The researchers hypothesize that during infection, one component of the ribosome, eL40 (green oval) that is normally provided by the algal cell is replaced by a similar protein produced by the virus (red oval), and this changes the specificity of the ribosome so that it preferentially reads messages produced by the virus. Credit: Thomy/ UH Mānoa “We were excited to discover that this virus encodes a ribosomal protein called eL40,” said Julie Thomy, lead author of the study and postdoctoral researcher in the Daniel K. Inouye Center for Microbial Oceanography: Research and Education (C-MORE) and Department of Oceanography in the UH Mānoa School of Ocean and Earth Science and Technology (SOEST). “It makes sense that a virus could benefit from altering this critical piece of cell machinery, but there was just no evidence for it in any eukaryotic virus.” The virus was discovered as part of a larger effort by members of the Marine Viral Ecology Laboratories (MarVEL) in SOEST to isolate and characterize new viruses that live in the ocean. A former Oceanography graduate student, Christopher Schvarcz, sampled water from Station ALOHA 60 miles north of O’ahu, Hawai‘i, and subsequently isolated dozens of viruses. Among them was FloV-SA2, which infects a species of phytoplankton called Florenciella. Transmission electron micrograph of negatively stained FloV-SA2 virus particles. Scale bar: 100 nm. Credit: Thomy/ UH Mānoa “Chris was so productive at isolating viruses, he could not analyze them all before he left,” said Grieg Steward, Oceanography faculty member overseeing the project. “Detailed analysis of this virus had to wait until Dr. Thomy joined the lab, but it was worth the wait!” Preferential production of virus proteins? Previous discoveries have shown that, like FloV-SA2, other so-called ‘giant’ viruses code for proteins involved in a wide range of metabolic processes. Some, such as those involved in fermentation or sensing light, seem like surprising functions to find in a virus. These genes must help the virus replicate, but, as is the case with the ribosomal protein, it is not always clear how. The researchers are now focused on figuring out the details of how and when this protein is used by the virus. CTD water sampler prepared for deployment. Credit: Kelsey Maloney “Our working hypothesis is that by inserting one of its own proteins into the ribosome, the virus alters this key piece of machinery to favor the production of virus proteins, over the usual cell proteins,” said Thomy. “Viruses are integral to the functioning of ocean ecosystems, influencing biological productivity, shifting community interactions, and driving evolutionary change,” said Steward. “This discovery reveals new details about the complex ways viruses in the ocean interact with phytoplankton, which are the foundation of ocean ecosystems, but it also opens new avenues in our understanding of the fundamentals of viral biology.” The scientists expect that FloV-SA2 will be a valuable model system for investigating new mechanisms by which viruses manipulate cell metabolism and redirect host resources and energy. Reference: “Eukaryotic viruses encode the ribosomal protein eL40” by Julie Thomy, Christopher R. Schvarcz, Kelsey A. McBeain, Kyle F. Edwards and Grieg F. Steward, 23 October 2024, npj Viruses. DOI: 10.1038/s44298-024-00060-2 Recent animal studies have highlighted the crucial role of brain cells called astrocytes in sleep regulation. New research shows that activating these cells can keep mice awake for extended periods without making them sleepier later. This finding might lead to interventions that reduce the negative impacts of prolonged wakefulness, potentially benefiting shift workers, first responders, and military personnel. The Role of Astrocytes in Sleep Regulation New animal research suggests that little-studied brain cells known as astrocytes are major players in controlling sleep needs and may someday help humans go without sleep for longer without negative consequences such as mental fatigue and impaired physical health. Published in the Journal of Neuroscience, the study found that activating these cells kept mice awake for hours when they would normally be resting, without making them any sleepier. “Extended wakefulness normally increases sleep time and intensity, but what we saw in this study was that despite hours of added wakefulness these mice did not differ from well-rested controls in terms of how long and how intensely they slept,” said senior author Marcos Frank, a neuroscientist and professor at the Washington State University Elson S. Floyd College of Medicine. “This opens up the possibility that we might someday have interventions that could target astrocytes to mitigate the negative consequences of prolonged wakefulness.” Potential Applications for Shift Workers and Military Frank envisioned that might include medications that could be used to improve the productivity, safety, and health of shift workers and others who work long or odd hours, such as first responders and military personnel. Sleep loss and mistimed sleep have been shown to impact a variety of key processes, including attention, cognition, learning, memory, metabolism, and immune function. Astrocytes: More Than Just Brain “Glue” Astrocytes are types of non-neuronal cells that interact with neurons, brain cells that transmit easily measured electrical signals from the brain to other parts of the body. Previously thought of as merely the “glue” that holds the brain together, astrocytes have recently been shown to play an active role in various behaviors and processes through a much more subtle and difficult-to-measure process known as calcium signaling. This includes a previous WSU study that showed that suppressing astrocyte calcium signaling throughout the brain resulted in mice building up less sleep need after sleep deprivation. In this study, the researchers looked specifically at astrocytes in the basal forebrain, a brain region known to play a critical role in determining time spent asleep and awake as well as sleep needs. Using chemogenetics—a method to control and study signaling pathways within brain cells—they activated these astrocytes and found that this resulted in mice staying awake for 6 hours or more during their normal sleep period. What’s more, the researchers did not see subsequent changes in sleep time or sleep intensity in response to the added wakefulness, as would be expected. “Our findings suggest that our need for sleep isn’t just a function of prior wake time but is also driven by these long-ignored non-neuronal cells,” said first author Ashley Ingiosi, an assistant professor of neuroscience at Ohio State University who conducted the study while working as a postdoctoral research associate in Frank’s lab at WSU. “We can now start to pinpoint how astrocytes interact with neurons to trigger this response and how they drive the expression and regulation of sleep in different parts of the brain.” Future Research Directions Next, the researchers plan to conduct behavioral tests in mice to determine how activating basal forebrain astrocytes to induce wakefulness might impact other processes besides sleep needs, such as attention, cognition, learning, memory, metabolism, and immune function. To get at least some indication of the potential impact on attention and cognition, they looked at EEG markers of those two processes in this study and found them to be similar to those seen in well-rested controls. Reference: “Activation of Basal Forebrain Astrocytes Induces Wakefulness without Compensatory Changes in Sleep Drive” by Ashley M. Ingiosi, Christopher R. Hayworth and Marcos G. Frank, 8 August 2023, Journal of Neuroscience. DOI: 10.1523/JNEUROSCI.0163-23.2023 The study was funded by the National Institute of Neurological Disorders and Stroke, the National Institute of Mental Health, and the National Institute of Neurological Disorders and Stroke. RRG455KLJIEVEWWF |
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