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KoDō 和牛燒肉適合請客嗎?》台中公益路聚餐推薦|10大類型餐廳評比 |
| 心情隨筆|心情日記 2026/04/20 13:15:51 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人,臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」,從精緻西餐到創意火鍋,從日式丼飯到義式早午餐,每走幾步,就會有完全不同的特色料理餐廳。 這次我特別花了一整個月,實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店,也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準,整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你,找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型,從高質感牛排館到巷弄系早午餐,每一間都有自己獨特的風格。為了讓整體比較更客觀,我依照以下四大面向進行評比,並搭配實際用餐體驗來打分。
整體而言,我希望這份評比不只是「哪家好吃」,而是幫你在不同情境下(約會、家庭聚餐、朋友小聚、商業午餐)都能快速找到合適的選擇。畢竟,美食不只是味覺的滿足,更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段,從火鍋、燒肉到中式料理與早午餐,每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單,橫跨平價、創意、高級各路風格。
一頭牛日式燒肉|炭香濃郁的和牛饗宴,約會聚餐首選
走在公益路上,很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面,搭配昏黃燈光與暖色調的內裝,讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大,但桌距規劃得宜,每桌皆設有獨立排煙設備,烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂,絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網,讓人完全不用分心。整場用餐過程就像一場表演,從視覺、嗅覺到味覺都被滿足。 綜合評分
地址:408臺中市南屯區公益路二段162號電話:04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」,更像是一場五感盛宴。從進門那一刻到最後一道甜點,都能感受到他們對細節的用心。 TANG Zhan 湯棧|文青系火鍋代表,麻香湯底與視覺美感並重
在公益路這條美食戰線上,TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址:408臺中市南屯區公益路二段248號電話:04-22580617 官網:https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度,又結合現代風格與細節服務,讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店,湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店|明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂,那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主,陽光灑進室內,讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧,建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味,選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口,米芯保留微Q口感;而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香,口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹,無論是一個人帶電腦工作、或朋友聚餐,都能找到舒服角落。餐點上桌速度穩定,服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」,很適合想遠離忙碌日常的人。 綜合評分
地址:40861臺中市南屯區公益路二段18號電話:04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇,而是以「剛剛好」的份量與風味,陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店,NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物|平價卻用心的湯頭系火鍋,家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上,加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐,但靠著實在的湯頭與親切的服務,默默吸引許多回頭客。每到用餐時間,總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底,湯頭清澈卻不單薄,越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向,桌距寬敞、座位舒適,帶小孩來也不覺擁擠。店員態度親切,補湯、收盤都很勤快,給人一種「被照顧著」的安心感。 綜合評分
地址:403臺中市西區公益路288號電話:0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤,而是用最簡單的湯頭與新鮮食材,傳遞出家常卻不平凡的溫度。 印月餐廳|中式料理的藝術演繹,宴客與家庭聚會首選
說到臺中公益路的中式料理代表,印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名,把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設,每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏,都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法,讓人感受到「被款待」的尊榮感。 綜合評分
地址:408臺中市南屯區公益路二段818號電話:0422511155 小結語印月餐廳是一間「不只吃飯,更像品味生活」的地方。 KoDō 和牛燒肉|極致職人精神,專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗,一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉,以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址:403臺中市西區公益路260號電話:0423220312 官網:https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳,而是一場體驗。 永心鳳茶|在茶香裡用餐的優雅時光,臺味早午餐的新詮釋
走進 永心鳳茶公益店,彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤,無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」,都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和,對茶品介紹詳盡。上餐節奏剛好,不急不徐。 綜合評分
地址:40360臺中市西區公益路68號三樓(勤美誠品)電話:0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓|老饕級江浙功夫菜,穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主,兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址:408臺中市南屯區公益路二段95號電話:0423202322 官網:https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司|低調奢華的無菜單日料,職人手藝詮釋旬味極致
在熱鬧的公益路上,一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase(無菜單料理) 形式,每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘,節奏緩慢但沉穩。 綜合評分
地址:408臺中市南屯區公益路二段25號電話:0423206368 官網:https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂|人氣爆棚的和牛燒肉聖地,肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳,茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐,價格約落在 $700–$1000 間,份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快,整場用餐流程流暢無壓力。 綜合評分
地址:403臺中市西區公益路268號電話:0423281167 官網:https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質+輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後,臺中公益路不只是一條美食街,而是一段生活風景線。 有的餐廳講究細膩與儀式感,像 一頭牛日式燒肉 與 一笈壽司,讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心,像 加分昆布鍋物、永心鳳茶,讓人明白吃飯不只是為了飽足,而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店,則用穩定的品質與熱絡的氛圍,成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店,構成了公益路最動人的縮影 有華麗的,也有溫柔的;有傳統的,也有創新的。 每一家都在自己的風格裡發光,讓人吃到的不只是料理,而是一種生活的溫度與節奏。 對我而言,這不僅是一場美食旅程,更是一趟關於「臺中味道」的回憶之旅。 FAQ:關於臺中公益路美食常見問題Q1:公益路哪一區的餐廳最集中? Q2:需要提前訂位嗎? 最後的話若要用一句話形容這趟美食之旅,我會說: KoDō 和牛燒肉清淡口味適合嗎? 如果你也和我一樣喜歡用味蕾探索一座城市,那就把這篇公益路美食攻略收藏起來吧。三希樓用餐環境舒服嗎? 無論是約會、慶生、家庭聚餐,或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。三希樓有提供尾牙方案嗎? 下一餐,不妨從這10家開始。一頭牛日式燒肉慶生氣氛夠嗎? 打開手機、約上朋友,讓公益路成為你生活裡最容易抵達的小確幸。一頭牛日式燒肉整體值得推薦嗎? 如果你有私心愛店,也歡迎留言分享,一頭牛日式燒肉第一次來要點什麼? 你的推薦,可能讓我下一趟美食旅程變得更精彩。一頭牛日式燒肉年節期間價格會變嗎? Gene regulation is vital for development, but silencers, which repress genes, are less understood. Stark lab’s Silencer-seq identified 800 silencers in fruit flies, discovered key transcription motifs, and uncovered Saft, a transcription factor. This research sheds light on silencer function and chromatin regulation. Credit: SciTechDaily.com Using silencer-seq, the Stark lab identified 800+ silencers and the transcription factor Saft in fruit flies, enhancing our understanding of gene regulation. The precise regulation of gene activity is crucial for cell specialization and the development of healthy organisms. While significant progress has been made in understanding enhancers—DNA sequences that activate genes—silencers, which suppress gene activity, remain far less studied despite their equally vital role. Technical limitations and research biases have historically made it challenging to identify silencers across the genome. However, Alexander Stark’s lab at the IMP has overcome these obstacles with a new technique called “Silencer-seq.” Using this method, they uncovered hundreds of silencers in the fruit fly genome, a breakthrough now detailed in the journal Molecular Cell. Gene Regulation and Its Role in Cell Specialization The development of a healthy organism depends on the precise regulation of gene activity—determining when specific genes are switched on or off. This process, called gene regulation, is critical for cell specialization, guiding cells to develop into distinct types like muscle, nerve, or skin cells, which together form a functioning organism. For over 40 years, scientists have recognized the importance of two types of regulatory DNA sequences located away from genes: enhancers and silencers. Enhancers act as activators, “switching on” genes and increasing their expression when needed. In contrast, silencers act as repressors, “switching off” specific genes to maintain balance and ensure proper cellular function. Research on enhancers increased the understanding of enhancers in recent years, with the Stark lab making remarkable strides, from reading their DNA sequences and understanding their gene-regulatory functions to designing tissue-specific enhancers from scratch. However, despite their critical role, scientists still know only a few silencers and don’t understand how they look and function as much. Challenges in Identifying Silencers The challenge mostly lies in scouting for silencers across the entirety of the genome. “Over the past decade, researchers have focused on mapping sequences that look like enhancers, assuming silencers are just enhancers in reverse,” says Lorena Hofbauer, first author of the study and a recent graduate of the Vienna BioCenter PhD Program. “They’re thought to share similar features, such as being located in open chromatin, but shutting genes down instead of switching them on.” While several tools were developed to study elements with enhancer-like features, this focus has created a bias. As a result, tools specifically designed to detect silencers based on their gene-repressive function lag behind, leaving these genetic sequences overlooked and poorly understood. A Pioneering Approach: The Silencer-Seq Method Researchers from the Stark lab have now taken a pioneering, unbiased approach to find silencers in the genome of the fruit fly Drosophila melanogaster. Using ‘silencer-seq’, a new method developed for this task, the scientists discovered hundreds of silencers that differ from traditional enhancer profiles, revealing previously hidden regulatory elements in the genome. Their findings are now published in the journal Molecular Cell. To address this gap, the Stark lab created “Silencer-seq,” a novel method based on the STARR-seq technology they previously developed. Revealing Silencer Mechanisms With Silencer-Seq The approach begins by creating a comprehensive library of DNA fragments, each representing a small segment of the fruit fly genome. These fragments, which together represent the entire genome, are paired with a strong enhancer designed to drive transcription—unless the DNA fragment acts as a silencer, actively repressing the process. The resulting DNA constructs are introduced into fruit fly cells, where they interact with the cells’ transcription machinery. Each fragment’s activity is assessed by monitoring messenger RNA (mRNA) output: fragments that suppress transcription of the reporter gene lead to little or no mRNA production, identifying them as silencers. By sequencing the mRNA, researchers can then pinpoint which genomic fragments possess silencing activity. “We found over 800 silencers across the entire fruit fly genome with this tool,” says Hofbauer “This is the first time an unbiased large-scale catalog of these regulatory elements is assembled.” With access to such a collection of silencers, the team was able to identify key features shared by these regulatory elements. Key Discoveries: Transcription Factor Motifs and Saft They discovered three transcription factor motifs—specific DNA sequences that transcription factors bind to in order to regulate gene activity—individually driving silencer activity. One of these motifs, DLM3 (Drosophila Long Motif 3), was computationally discovered a decade ago but had never been characterized before. “Something very interesting that we found is that DLM3 is bound by a previously unknown transcription factor, Saft or Silencer Associated Factor,” explains Hofbauer. “We discovered that Saft helps keep genes turned off in the brain and ovaries, which is crucial for proper development and fertility in flies.” Silencers appear inaccessible in their native chromatin, where DNA is tightly packed with nucleosomes. However, the researchers show that Saft can locate and bind to its target motifs even in an apparently chromatin state—a unique feature that can explain why it had been so difficult to find silencers before, by approaches that focused on open chromatin. “The discovery of Saft’s role in gene regulation helps us figure out how silencers work and adds to our understanding of the machinery that controls cell identity and development,” says Hofbauer. Implications for Broader Research With a detailed map of silencers now available in Drosophila, researchers can begin to explore whether similar mechanisms operate in other organisms, including humans. “What we’ve learned here is that to really study these silencers, we need to go in without bias,” says Alexander Stark. “Now that we’ve identified this class of silencers, we can start finding more, dig into how they work, and get closer to fully mapping out the human genome.” Reference: “A genome-wide screen identifies silencers with distinct chromatin properties and mechanisms of repression” by Lorena Hofbauer, Lisa-Marie Pleyer, Franziska Reiter, Alexander Schleiffer, Anna Vlasova, Leonid Serebreni, Annie Huang and Alexander Stark, 20 November 2024, Molecular Cell. DOI: 10.1016/j.molcel.2024.10.041 A new study has found that a class of toxins found in snake and mammalian venom evolved from the same ancestral gene. A new study has found that a class of toxins found in snake and mammalian venom evolved from the same ancestral gene. A new study has found that venoms found in snakes and mammals share a common origin Researchers traced the origin of a class of toxins, called kallikrein serine proteases, to a salivary protein found in a common ancestor Results from the evolutionary tree also showed that non-toxic salivary kallikreins in mammals, including those found in mice and human saliva, also evolved from the same ancestral gene The study provides strong evidence for the hypothesis that venom evolved from a common group of genes with toxic potential that existed in the ancestor of snakes and mammals Snakes, some lizards, and even a few mammals can have a venomous bite. Although these lineages split more than 300 million years ago, their venoms have evolved from the same ancestral salivary protein, reported scientists today (December 22, 2021) in BMC Biology. Researchers from the Okinawa Institute of Science and Technology Graduate University (OIST) in Japan and the Australian National University focused on a class of toxins found in most snake venoms and all other reptile and mammalian venoms called kallikrein serine proteases and traced their origins to a gene found in a common ancestor. “Venoms are cocktails of toxic proteins that have evolved across the whole animal kingdom, typically as a method of killing or immobilizing prey,” explained Agneesh Barua, co-first author and PhD student at OIST. “The oral venom systems found in snakes are particularly complex, and the origin of their venoms is still unclear.” Salivary kallikreins, like those found in mice, humans, and venomous mammals like shrews and solenodons, are closely related to toxic serine protease kallikreins found in venomous snakes. Credit: OIST In a previous paper, Barua and his colleagues found that the mammal salivary gland and snake venom gland share a similar pattern of activity in a group of regulatory genes, suggesting that the foundation needed for venom to evolve exists in both snakes and mammals. “In that paper, we hypothesized that in the ancestor of snakes and mammals, there was a common group of genes that had a toxic potential,” said Barua. “Snakes and mammals then took different evolutionary paths, with snake lineages evolving diverse and increasingly toxic concoctions, while in mammals, venom did evolve, but to a much lesser degree. But what we wanted to know is whether the toxins within mammal and snake venom evolved from a common ancestral gene.” Kallikrein serine proteases are a kind of protein-degrading enzyme, which play a key role in regulating blood pressure. Mammal saliva contains small quantities of these proteins, although their function remains unclear to this day. But in venomous snakes and mammals, like shrews and solenodons, these proteins have evolved toxicity. When injected in high amounts, they drastically reduce blood pressure, potentially causing unconsciousness and even death. Early on, researchers noticed biochemical similarities between kallikrein serine proteases in snake venoms and those in mammal saliva, but scientists did not know until now whether they were, in fact, related. “There are so many different serine proteases that have a high degree of similarity, that until now, it was too difficult to isolate the right genes needed to determine the evolutionary history,” said Barua. With recent advances in genomic methods, the research group were able to identify and compare all the kallikrein genes in reptiles, amphibians, fishes and mammals to create an evolutionary tree. Excitingly, they found that snake venom kallikrein serine proteases and mammal salivary kallikreins did evolve from the same ancestral gene. “This is really strong evidence for our hypothesis that venom evolved from a common group of genes in an ancestor that had a toxic potential,” said Barua. “But the most surprising thing was that non-toxic salivary kallikreins, like those found in humans and mice, also evolved from the same ancestral gene.” In fact, the researchers found that the non-toxic kallikreins in mammal saliva were more closely related to the venomous toxins found in snakes than to other kallikreins found within mammals. Overall, this evidence suggests that salivary kallikrein proteins in mammals, including humans, also have the evolutionary potential to become toxic. But, Barua quickly added, there is a caveat. “Just because we have the building blocks to evolve venom doesn’t mean this will occur. Venom is really energetically expensive to make, so there had to be a strong ecological pressure for it, which humans, and most mammals don’t have.” But what this does tell us, he said, is that the line between venomous and non-venomous mammals is blurrier than previously thought. Reference: “Co-option of the same ancestral gene family gave rise to mammalian and reptilian toxins” by Agneesh Barua, Ivan Koludarov and Alexander S. Mikheyev, 23 December 2021, BMC Biology. DOI: 10.1186/s12915-021-01191-1 Concept of artificial chloroplasts and mitochondria within a liposome for self-sustaining energy generation through photosynthesis and cellular respiration. Credit: Biological Interface Group, Sogang University Assessing how energy-generating synthetic organelles could sustain artificial cells. Researchers have assessed the progress and challenges in creating artificial mitochondria and chloroplasts for energy production in synthetic cells. These artificial organelles could potentially enable the development of new organisms or biomaterials. The researchers identified proteins as the most crucial components for molecular rotary machinery, proton transport, and ATP production, which serves as the cell’s primary energy currency. Energy production in nature is the responsibility of chloroplasts and mitochondria and is crucial for fabricating sustainable, synthetic cells in the lab. Mitochondria are not only “the powerhouses of the cell,” as the middle school biology adage goes, but also one of the most complex intracellular components to replicate artificially. In Biophysics Reviews, by AIP Publishing, researchers from Sogang University in South Korea and the Harbin Institute of Technology in China identified the most promising advancements and greatest challenges of artificial mitochondria and chloroplasts. “This could be an important milestone in understanding the origin of life and the origin of cells.” Kwanwoo Shin “If scientists can create artificial mitochondria and chloroplasts, we could potentially develop synthetic cells that can generate energy and synthesize molecules autonomously. This would pave the way for the creation of entirely new organisms or biomaterials,” author Kwanwoo Shin said. In plants, chloroplasts use sunlight to convert water and carbon dioxide into glucose. Mitochondria, found in plants and animals alike, produce energy by breaking down glucose. Once a cell produces energy, it often uses a molecule called adenosine triphosphate (ATP) to store and transfer that energy. When the cell breaks down the ATP, it releases energy that powers the cell’s functions. “In other words, ATP acts as the main energy currency of the cell, and it is vital for the cell to perform most of the cellular functions,” said Shin. Advancements in Artificial Organelles The team describes the components required to construct synthetic mitochondria and chloroplasts and identifies proteins as the most important aspects for molecular rotary machinery, proton transport, and ATP production. Previous studies have replicated components that make up the energy-producing organelles. Some of the most promising work investigates the intermediate operations involved in the complex energy-generating process. By connecting the sequence of proteins and enzymes, researchers have improved energy efficiency. One of the most significant challenges remaining in trying to reconstruct the energy production organelles is enabling self-adaptation in changing environments to maintain a stable supply of ATP. Future studies must investigate how to improve upon this limiting feature before synthetic cells are self-sustainable. The authors believe it is important to create artificial cells with biologically realistic energy-generation methods that mimic natural processes. Replicating the entire cell could lead to future biomaterials and lend insight into the past. “This could be an important milestone in understanding the origin of life and the origin of cells,” Shin said. Reference: “Artificial organelles for sustainable chemical energy conversion and production in artificial cells: Artificial mitochondrion and chloroplasts” by Hyun Park, Weichen Wang, Seo Hyeon Min, Yongshuo Ren, Kwanwoo Shin and Xiaojun Han, 28 March 2023, Biophysics. DOI: 10.1063/5.0131071 RRG455KLJIEVEWWF |
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