|
|
文章數：196 |
 TANG Zhan 湯棧包廂適合尾牙嗎？》台中公益路高人氣餐廳推薦｜10家好吃又好拍 |
| 在地生活｜大台北 2026/04/21 03:13:53 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人，臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」，從精緻西餐到創意火鍋，從日式丼飯到義式早午餐，每走幾步，就會有完全不同的特色料理餐廳。 這次我特別花了一整個月，實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店，也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準，整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你，找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型，從高質感牛排館到巷弄系早午餐，每一間都有自己獨特的風格。為了讓整體比較更客觀，我依照以下四大面向進行評比，並搭配實際用餐體驗來打分。
整體而言，我希望這份評比不只是「哪家好吃」，而是幫你在不同情境下（約會、家庭聚餐、朋友小聚、商業午餐）都能快速找到合適的選擇。畢竟，美食不只是味覺的滿足，更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段，從火鍋、燒肉到中式料理與早午餐，每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單，橫跨平價、創意、高級各路風格。
一頭牛日式燒肉｜炭香濃郁的和牛饗宴，約會聚餐首選
走在公益路上，很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面，搭配昏黃燈光與暖色調的內裝，讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大，但桌距規劃得宜，每桌皆設有獨立排煙設備，烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂，絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網，讓人完全不用分心。整場用餐過程就像一場表演，從視覺、嗅覺到味覺都被滿足。 綜合評分
地址：408臺中市南屯區公益路二段162號電話：04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」，更像是一場五感盛宴。從進門那一刻到最後一道甜點，都能感受到他們對細節的用心。 TANG Zhan 湯棧｜文青系火鍋代表，麻香湯底與視覺美感並重
在公益路這條美食戰線上，TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址：408臺中市南屯區公益路二段248號電話：04-22580617 官網：https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度，又結合現代風格與細節服務，讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店，湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店｜明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂，那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主，陽光灑進室內，讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧，建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味，選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口，米芯保留微Q口感；而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香，口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹，無論是一個人帶電腦工作、或朋友聚餐，都能找到舒服角落。餐點上桌速度穩定，服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」，很適合想遠離忙碌日常的人。 綜合評分
地址：40861臺中市南屯區公益路二段18號電話：04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇，而是以「剛剛好」的份量與風味，陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店，NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物｜平價卻用心的湯頭系火鍋，家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上，加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐，但靠著實在的湯頭與親切的服務，默默吸引許多回頭客。每到用餐時間，總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底，湯頭清澈卻不單薄，越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向，桌距寬敞、座位舒適，帶小孩來也不覺擁擠。店員態度親切，補湯、收盤都很勤快，給人一種「被照顧著」的安心感。 綜合評分
地址：403臺中市西區公益路288號電話：0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤，而是用最簡單的湯頭與新鮮食材，傳遞出家常卻不平凡的溫度。 印月餐廳｜中式料理的藝術演繹，宴客與家庭聚會首選
說到臺中公益路的中式料理代表，印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名，把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設，每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏，都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法，讓人感受到「被款待」的尊榮感。 綜合評分
地址：408臺中市南屯區公益路二段818號電話：0422511155 小結語印月餐廳是一間「不只吃飯，更像品味生活」的地方。 KoDō 和牛燒肉｜極致職人精神，專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗，一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉，以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址：403臺中市西區公益路260號電話：0423220312 官網：https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳，而是一場體驗。 永心鳳茶｜在茶香裡用餐的優雅時光，臺味早午餐的新詮釋
走進 永心鳳茶公益店，彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤，無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」，都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和，對茶品介紹詳盡。上餐節奏剛好，不急不徐。 綜合評分
地址：40360臺中市西區公益路68號三樓(勤美誠品)電話：0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓｜老饕級江浙功夫菜，穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主，兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址：408臺中市南屯區公益路二段95號電話：0423202322 官網：https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司｜低調奢華的無菜單日料，職人手藝詮釋旬味極致
在熱鬧的公益路上，一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase（無菜單料理） 形式，每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘，節奏緩慢但沉穩。 綜合評分
地址：408臺中市南屯區公益路二段25號電話：0423206368 官網：https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂｜人氣爆棚的和牛燒肉聖地，肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳，茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐，價格約落在 $700–$1000 間，份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快，整場用餐流程流暢無壓力。 綜合評分
地址：403臺中市西區公益路268號電話：0423281167 官網：https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質＋輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後，臺中公益路不只是一條美食街，而是一段生活風景線。 有的餐廳講究細膩與儀式感，像 一頭牛日式燒肉 與 一笈壽司，讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心，像 加分昆布鍋物、永心鳳茶，讓人明白吃飯不只是為了飽足，而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店，則用穩定的品質與熱絡的氛圍，成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店，構成了公益路最動人的縮影 有華麗的，也有溫柔的；有傳統的，也有創新的。 每一家都在自己的風格裡發光，讓人吃到的不只是料理，而是一種生活的溫度與節奏。 對我而言，這不僅是一場美食旅程，更是一趟關於「臺中味道」的回憶之旅。 FAQ：關於臺中公益路美食常見問題Q1：公益路哪一區的餐廳最集中？ Q2：需要提前訂位嗎？ 最後的話若要用一句話形容這趟美食之旅，我會說： KoDō 和牛燒肉商務聚餐適合嗎？ 如果你也和我一樣喜歡用味蕾探索一座城市，那就把這篇公益路美食攻略收藏起來吧。KoDō 和牛燒肉值得排隊嗎？ 無論是約會、慶生、家庭聚餐，或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。加分100%浜中特選昆布鍋物情侶來合適嗎？ 下一餐，不妨從這10家開始。茶六燒肉堂份量足夠嗎？ 打開手機、約上朋友，讓公益路成為你生活裡最容易抵達的小確幸。NINI 尼尼臺中店單點比較好嗎？ 如果你有私心愛店，也歡迎留言分享，永心鳳茶尾牙聚餐表現如何？ 你的推薦，可能讓我下一趟美食旅程變得更精彩。NINI 尼尼臺中店人潮很多嗎？ RNA particles swarm an X chromosome from a mouse in a new visualization of X chromosome inactivation. Credit: Los Alamos National Laboratory Combining lab data with supercomputing power reveals role of RNA and chromosome structure in regulating gene expression. Using supercomputer-driven dynamic modeling based on experimental data, researchers can now probe the process that turns off one X chromosome in female mammal embryos. This new capability is helping biologists understand the role of RNA and the chromosome’s structure in the X inactivation process, leading to a deeper understanding of gene expression and opening new pathways to drug treatments for gene-based disorders and diseases. “This is the first time we’ve been able to model all the RNA spreading around the chromosome and shutting it down,” said Anna Lappala, a visiting scientist at Los Alamos National Laboratory and a polymer physicist at Massachusetts General Hospital and the Harvard Department of Molecular Biology. Lappala is the first author of the paper published on October 4, 2021, in the Proceedings of the National Academy of Sciences. “From experimental data alone, which is 2D and static, you don’t have the resolution to see a whole chromosome at this level of detail. With this modeling, we can see the processes regulating gene expression, and the modeling is grounded in 2D experimental data from our collaborators at Massachusetts General Hospital and Harvard.” The model—considered 4D because it shows motion, including time as the fourth dimension—runs on Los Alamos supercomputers. The model also incorporates experimental data from mice genomes obtained through a molecular method called 4DHiC. The combined molecular and computational methodology is a first. In the visualization, RNA particles swarm over the X chromosome. The tangled-spaghetti-like strands writhe, changing shape, then the particles engulf and penetrate the depths of the chromosome, turning it off. See the visualization here: “The method allows us to develop an interactive model of this epigenetic process,” said Jeannie T. Lee, professor of Genetics at Harvard Medical School and vice chair in molecular biology at Massachusetts General Hospital, whose lab contributed the experimental data underpinning the model. Epigenetics is the study of changes in gene expression and heritable traits that don’t involve mutations in the genome. “What’s been missing in the field is some way for a user who’s not computationally savvy to go interactively into a chromosome,” Lee said. She compared using the Los Alamos model to using Google Earth, where “you can zoom into any location on an X chromosome, pick your favorite gene, see the other genes around it, and see how they interact.” That capability could lend insight into how diseases spread, for instance, she said. Based on the work in this paper, Los Alamos is currently developing a Google Earth-style browser where any scientist can upload their genomic data and view it dynamically in 3D at various magnifications, said Karissa Sanbonmatsu, a structural biologist at Los Alamos National Laboratory, corresponding author of the paper, and a project leader in developing the computational method. In mammals, a female embryo is conceived with two X chromosomes, one inherited from each parent. X inactivation shuts off the chromosome, a crucial step for the embryo to survive, and variations in X inactivation can trigger a variety of developmental disorders. The new Los Alamos model will facilitate a deeper understanding of gene expression and related problems, which could lead to pharmacological treatments for various gene-based diseases and disorders, Lee said. “Our main goal was to see the chromosome change its shape and to see gene-expression levels over time,” said Sanbonmatsu. To understand how genes are turned on and off, Sanbonmatsu said, “It really helps to know the structure of the chromosome. The hypothesis is that a compacted, tightly structured chromosome tends to turn off genes, but there are not a lot of smoking guns about this. By modeling 3D structures in motion, we can get closer to the relationship between structural compaction and turning off genes.” Lee likened the chromosome’s structure to origami. A complicated shape akin to an origami crane offers lots of surface for gene expression and might be biologically preferred to remain active. The model shows a variety of substructures in the chromosome. When it is shut down, “it’s a piecemeal process in which some substructures are kept but some are dissolved,” Sanbonmatsu said. “We see beginning, intermediate, and end stages, through a gradual transition. That’s important for epigenetics because it’s the first time we have been able to analyze the detailed structural transition in an epigenetic change.” The modeling also shows genes on the surface of the chromosome that escape X chromosome inactivation, confirming early experimental work. In the model, they cluster and apparently interact or work together on the surface of the chromosome. In another insight from the modeling, “As the chromosome goes from an active X, when it’s still fairly large, to a compact inactive X, that’s smaller, we notice there’s a core of the chromosome that’s extremely dense, but the surface is much less dense. We see a lot more motion on the surface too,” Lappala said. “Then there’s an intermediate region that’s not too fast or slow, where the chromosome can rearrange.” An inactive X can activate later in a process called age-related activation of inactive X. “It’s associated with problems in blood cells in particular that are known to cause autoimmunity,” Lee said. “Some research is trying pharmacologically to activate the inactive X to treat neurological disorders in children by giving them something back that’s missing on their active X chromosome. For instance, a child could have a mutation that can cause disease. We think if we can reactivate the normal copy on the inactive X, then we would have an epigenetic treatment for that mutation.” Reference: “Four-dimensional chromosome reconstruction elucidates the spatiotemporal reorganization of the mammalian X chromosome” by Anna Lappala, Chen-Yu Wang, Andrea Kriz, Hunter Michalk, Kevin Tan, Jeannie T. Lee and Karissa Y. Sanbonmatsu, 13 October 2021, Proceedings of the National Academy of Sciences. DOI: 10.1073/pnas.2107092118 Funding: Laboratory Directed Research and Development program at Los Alamos National Laboratory. Researchers have identified the genetic pathway between the heart and brain responsible for fainting, revealing a two-way communication that could lead to new treatments for syncope-related disorders. Neurobiologists have discovered sensory neurons that regulate fainting, providing a foundation for targeted treatments for related disorders. Syncope, commonly known as fainting, affects nearly 40 percent of people at least once in their lifetime. These transient losses of consciousness can be precipitated by various triggers such as pain, fear, heat, or hyperventilation, and they are a substantial cause of emergency room visits. Despite their prevalence, the fundamental mechanisms underlying syncope have largely remained enigmatic. Breakthrough in Genetic Pathways Publishing a new report in Nature, University of California San Diego researchers, along with colleagues at The Scripps Research Institute and other institutions, have for the first time identified the genetic pathway between the heart and brain tied to fainting. One of their unique approaches was to think of the heart as a sensory organ rather than the longstanding viewpoint that the brain sends out signals and the heart simply follows directions. School of Biological Sciences Assistant Professor Vineet Augustine, the paper’s senior author, applies a variety of approaches to better understand these neural connections between the heart and brain. An image of a heart labeled by vagal sensory neurons. In a new study published in the journal Nature, UC San Diego researchers and their colleagues found that these neurons trigger fainting, laying a foundation for addressing fainting-related disorders. Credit: Augustine Lab, UC San Diego “What we are finding is that the heart also sends signals back to the brain, which can change brain function,” said Augustine. Information resulting from the study could be relevant to better understanding and treating various psychiatric and neurological disorders linked with brain-heart connections, the researchers note in their paper. “Our study is the first comprehensive demonstration of a genetically defined cardiac reflex, which faithfully recapitulates characteristics of human syncope at physiological, behavioral, and neural network levels.” Study on the Bezold-Jarisch Reflex Augustine, along with Biological Sciences Staff Research Associate Jonathan Lovelace and Graduate Student Jingrui Ma, the first authors of the paper, and their colleagues studied neural mechanisms related to Bezold-Jarisch reflex (BJR), a cardiac reflex first described in 1867. For decades researchers have hypothesized that the BJR, which features reduced heart rate, blood pressure, and breathing, may be associated with fainting. But information lacked in proving the idea since the neural pathways involved in the reflex were not well known. Researchers at UC San Diego and collaborating institutions have highlighted the immense crosstalk between the heart and the nervous system. The video displays heart activity dramatically slowing down with stimulation of vagal sensory neurons, which were found to trigger fainting. Credit: Augustine Lab, UC San Diego The researchers focused on the genetics behind a sensory cluster known as the nodose ganglia, which are part of the vagus nerves that carry signals between the brain and visceral organs, including the heart. Specifically, vagal sensory neurons, or VSNs, project signals to the brainstem and are thought to be associated with BJR and fainting. In their search for a novel neural pathway they discovered that VSNs expressing the neuropeptide Y receptor Y2 (known as NPY2R) are tightly linked to the well-known BJR responses. Optogenetic Studies and Findings Studying this pathway in mice, the researchers were surprised to find that when they proactively triggered NPY2R VSNs using optogenetics, a method of stimulating and controlling neurons, mice that had been freely moving about immediately fainted. During these episodes, they recorded from thousands of neurons in the brains of the mice, as well as heart activity and changes in facial features including pupil diameter and whisking. They also employed machine learning in several ways to analyze the data and pinpoint features of interest. Once NPY2R neurons were activated, they found, mice exhibited rapid pupil dilation and the classic “eye-roll” seen during human fainting, as well as suppressed heart rate, blood pressure, and breathing rate. They also measured reduced blood flow to the brain, an area of collaboration with Professor David Kleinfeld’s laboratory in the UC San Diego Departments of Neurobiology and Physics. “We were blown away when we saw how their eyes rolled back around the same time as brain activity rapidly dropped,” the researchers reported in a paper summary. “Then, after a few seconds, brain activity and movement returned. This was our eureka moment.” Further testing showed that when NPY2R VSNs were removed from mice, the BJR and fainting conditions vanished. Previous studies have shown that fainting is caused by a reduction in brain blood flow, which the new study also found to be true, but the new evidence indicated that brain activity itself could be playing an important role. The findings, therefore, implicate the activation of the newly genetically identified VSNs and their neural pathways not only with BJR, but more centrally in overall animal physiology, certain brain networks, and even behavior. Implications and Future Research Such findings were difficult to tease out previously because neuroscientists study the brain and cardiologists study the heart, but many do so in isolation of the other. “Neuroscientists traditionally think the body just follows the brain, but now it is becoming very clear that the body sends signals to the brain and then the brain changes function,” said Augustine. As a result of their findings, the researchers would like to continue tracking the precise conditions under which vagal sensory neurons are triggered into action. “We also hope to more closely examine cerebral blood flow and neural pathways in the brain during the moment of syncope, to better understand this common but mysterious condition,” they note. They also hope to use their research as a model to develop targeted treatments for fainting-associated conditions. The study was funded by UC San Diego, Scripps Research Institute, the Helen Dorris Foundation, the National Institutes of Health, the American Heart Association Early Faculty Independence Award, the Mallinckrodt Foundation, the Dorris Scholarship, the Dorris-Skaggs Fellowship, and the Shurl and Kay Curci Foundation Fellowship. A recent study indicates that the ability to recognize and remember sequential information may be unique to humans, as even close relatives like bonobos don’t exhibit the same capability. This ability, essential for language, planning, and sequential thinking, sets humans apart and could be a foundational aspect of our unique cultural evolution. A recent study suggests that the ability to remember and process sequences—critical for language, planning, and cultural development—may be unique to humans. Remembering the order of information is crucial when engaging in dialogues, organizing daily activities, or pursuing education. A recent study in the scientific journal PLOS ONE suggests that this ability might be unique to humans. Even the closest relatives of humans, such as bonobos, do not learn order in the same way. “The study contributes another piece of the puzzle to the question of how the mental abilities of humans and other animals differ, and why only humans speak languages, plan space travel, and have learned to exploit the earth so efficiently that we now pose a serious threat to countless other life forms,” says Johan Lind, associate professor in ethology and deputy director at the Center for Cultural Evolution, Stockholm University. Since September he has also been an associate professor of ethology at Linköping University. Cognitive Divide Between Humans and Apes Already earlier research at Stockholm University has suggested that only humans have the ability to recognize and remember so-called sequential information, and that this ability is a fundamental building block underlying unique human cultural abilities. But previously, this sequence memory-hypothesis has not been tested in humans’ closest relatives, the great apes. The new experiments now show that also bonobos, one of the great apes, struggle to learn the order of stimuli. In the recently published book The Human Evolutionary Transition: From Animal Intelligence to Culture (Princeton University Press), ethologists Magnus Enquist and Johan Lind at Stockholm University, and Stefano Ghirlanda, a researcher in psychology at Brooklyn College, New York, have launched a new theory for how humans became cultural beings. A central idea concerns the difference in how humans and other animals recognize and remember sequential information. “We have previously analyzed a large number of studies that suggest that only humans recognize and remember sequential information faithfully. But, even though we analyzed data from a number of mammals and birds, including monkeys, there has been a lack of information from our closest relatives, the other great apes,” says Johan Lind. In a series of experiments, the memory abilities of bonobos and humans were tested by having them press computer screens to, among other things, learn to distinguish between short sequences, including pressing right if a yellow square comes before a blue square, or by pressing to the left of the blue square appears before the yellow square. “The study shows that bonobos forget that they have seen a blue square already five to 10 seconds after it has disappeared from the screen and that they have great difficulty learning to distinguish the sequences blue-square-before-yellow-square from yellow-square- before-blue-square, even though they have been trained for thousands of trials,” says Vera Vinken, associated with Stockholm University, now a PhD student in Great Britain at the Biosciences Institute, Newcastle University. Humans Excel in Sequence Recognition In contrast, the study shows that humans learned to distinguish the short sequences nearly immediately. However, it still remains to be shown exactly how our closest relatives can remember and use sequential information. “We now know that our closest relatives do not share the same sequential mental abilities with humans. But even if the results indicate that their working memory works in principle in the same way as in rats and pigeons, no one has yet demonstrated this in practice,`” says Magnus Enquist, professor emeritus and one of the founders of the Center for Cultural Evolution. The new results provide further support for the sequence memory-hypothesis, that during human prehistory an ability to remember and process sequences evolved, a necessary mechanism for many uniquely human phenomena such as language, planning ability and sequential thinking. Reference: “A test of memory for stimulus sequences in great apes” by Johan Lind, Vera Vinken, Markus Jonsson, Stefano Ghirlanda and Magnus Enquist, 6 September 2023, PLOS ONE. DOI: 10.1371/journal.pone.0290546 RRG455KLJIEVEWWF |
||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
| 最新創作 |
|
||||
|
||||
|
||||
|
||||
|
||||
