|
|
文章數：132 |
 三希樓網路評價符合期待嗎？》公益路餐廳推薦Top10｜吃貨實測大公開，這些店真的值得再訪嗎？ |
| 休閒生活｜音樂 2026/04/21 04:18:42 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人，臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」，從精緻西餐到創意火鍋，從日式丼飯到義式早午餐，每走幾步，就會有完全不同的特色料理餐廳。 這次我特別花了一整個月，實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店，也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準，整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你，找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型，從高質感牛排館到巷弄系早午餐，每一間都有自己獨特的風格。為了讓整體比較更客觀，我依照以下四大面向進行評比，並搭配實際用餐體驗來打分。
整體而言，我希望這份評比不只是「哪家好吃」，而是幫你在不同情境下（約會、家庭聚餐、朋友小聚、商業午餐）都能快速找到合適的選擇。畢竟，美食不只是味覺的滿足，更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段，從火鍋、燒肉到中式料理與早午餐，每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單，橫跨平價、創意、高級各路風格。
一頭牛日式燒肉｜炭香濃郁的和牛饗宴，約會聚餐首選
走在公益路上，很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面，搭配昏黃燈光與暖色調的內裝，讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大，但桌距規劃得宜，每桌皆設有獨立排煙設備，烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂，絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網，讓人完全不用分心。整場用餐過程就像一場表演，從視覺、嗅覺到味覺都被滿足。 綜合評分
地址：408臺中市南屯區公益路二段162號電話：04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」，更像是一場五感盛宴。從進門那一刻到最後一道甜點，都能感受到他們對細節的用心。 TANG Zhan 湯棧｜文青系火鍋代表，麻香湯底與視覺美感並重
在公益路這條美食戰線上，TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址：408臺中市南屯區公益路二段248號電話：04-22580617 官網：https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度，又結合現代風格與細節服務，讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店，湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店｜明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂，那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主，陽光灑進室內，讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧，建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味，選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口，米芯保留微Q口感；而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香，口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹，無論是一個人帶電腦工作、或朋友聚餐，都能找到舒服角落。餐點上桌速度穩定，服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」，很適合想遠離忙碌日常的人。 綜合評分
地址：40861臺中市南屯區公益路二段18號電話：04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇，而是以「剛剛好」的份量與風味，陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店，NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物｜平價卻用心的湯頭系火鍋，家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上，加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐，但靠著實在的湯頭與親切的服務，默默吸引許多回頭客。每到用餐時間，總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底，湯頭清澈卻不單薄，越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向，桌距寬敞、座位舒適，帶小孩來也不覺擁擠。店員態度親切，補湯、收盤都很勤快，給人一種「被照顧著」的安心感。 綜合評分
地址：403臺中市西區公益路288號電話：0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤，而是用最簡單的湯頭與新鮮食材，傳遞出家常卻不平凡的溫度。 印月餐廳｜中式料理的藝術演繹，宴客與家庭聚會首選
說到臺中公益路的中式料理代表，印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名，把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設，每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏，都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法，讓人感受到「被款待」的尊榮感。 綜合評分
地址：408臺中市南屯區公益路二段818號電話：0422511155 小結語印月餐廳是一間「不只吃飯，更像品味生活」的地方。 KoDō 和牛燒肉｜極致職人精神，專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗，一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉，以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址：403臺中市西區公益路260號電話：0423220312 官網：https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳，而是一場體驗。 永心鳳茶｜在茶香裡用餐的優雅時光，臺味早午餐的新詮釋
走進 永心鳳茶公益店，彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤，無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」，都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和，對茶品介紹詳盡。上餐節奏剛好，不急不徐。 綜合評分
地址：40360臺中市西區公益路68號三樓(勤美誠品)電話：0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓｜老饕級江浙功夫菜，穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主，兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址：408臺中市南屯區公益路二段95號電話：0423202322 官網：https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司｜低調奢華的無菜單日料，職人手藝詮釋旬味極致
在熱鬧的公益路上，一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase（無菜單料理） 形式，每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘，節奏緩慢但沉穩。 綜合評分
地址：408臺中市南屯區公益路二段25號電話：0423206368 官網：https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂｜人氣爆棚的和牛燒肉聖地，肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳，茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐，價格約落在 $700–$1000 間，份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快，整場用餐流程流暢無壓力。 綜合評分
地址：403臺中市西區公益路268號電話：0423281167 官網：https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質＋輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後，臺中公益路不只是一條美食街，而是一段生活風景線。 有的餐廳講究細膩與儀式感，像 一頭牛日式燒肉 與 一笈壽司，讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心，像 加分昆布鍋物、永心鳳茶，讓人明白吃飯不只是為了飽足，而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店，則用穩定的品質與熱絡的氛圍，成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店，構成了公益路最動人的縮影 有華麗的，也有溫柔的；有傳統的，也有創新的。 每一家都在自己的風格裡發光，讓人吃到的不只是料理，而是一種生活的溫度與節奏。 對我而言，這不僅是一場美食旅程，更是一趟關於「臺中味道」的回憶之旅。 FAQ：關於臺中公益路美食常見問題Q1：公益路哪一區的餐廳最集中？ Q2：需要提前訂位嗎？ 最後的話若要用一句話形容這趟美食之旅，我會說： TANG Zhan 湯棧春酒活動適合在這裡辦嗎？ 如果你也和我一樣喜歡用味蕾探索一座城市，那就把這篇公益路美食攻略收藏起來吧。永心鳳茶大型聚餐空間夠不夠？ 無論是約會、慶生、家庭聚餐，或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。永心鳳茶春酒菜色豐富嗎？ 下一餐，不妨從這10家開始。一頭牛日式燒肉食材新鮮嗎？ 打開手機、約上朋友，讓公益路成為你生活裡最容易抵達的小確幸。NINI 尼尼臺中店婚前派對適合嗎？ 如果你有私心愛店，也歡迎留言分享，三希樓服務態度如何？ 你的推薦，可能讓我下一趟美食旅程變得更精彩。茶六燒肉堂有什麼推薦搭配？ Circadian rhythm refers to the internal 24-hour cycle that regulates our physiological and behavioral processes, such as sleep-wake cycles, hormone production, and metabolism. It is influenced by external factors such as light, food intake, and temperature. A recent study conducted by a multidisciplinary team of researchers sheds light on the underlying mechanisms of circadian rhythms, providing new hope for managing jet lag, insomnia, and other sleep disorders. By utilizing advanced cryo-electron microscopy techniques, the scientists have uncovered the structure of the photosensor that governs the circadian rhythm, along with its target in fruit flies (Drosophila melanogaster), a major model organism used in circadian rhythm research. The research was recently published in the journal Nature. The research focused on fruit fly cryptochromes, key components of the circadian clocks of plants and animals, including humans. In flies and other insects, cryptochromes, activated by blue light, serve as the primary light sensors for setting circadian rhythms. The target of the cryptochrome photosensor, known as “Timeless” (TIM), is a large, complex protein that could not previously be imaged and thus its interactions with the cryptochrome are not well understood. Circadian rhythms work via what are basically genetic feedback loops. The researchers found that the TIM protein, along with its partner, the Period (PER) protein, act together to inhibit the genes that are responsible for their own production. With suitable delays between the events of gene expression and repression, an oscillation in protein levels is established. This oscillation represents the “the ticking of the clock and seems to be fairly unique to the circadian rhythm,” said senior author Brian Crane, the George W. and Grace L. Todd Professor and chair of chemistry and chemical biology in the College of Arts and Sciences. The Role of Blue Light in Circadian Regulation Blue light, Crane said, changes the chemistry and structure of cryptochrome’s flavin cofactor, which allows the protein to bind the TIM protein and inhibit TIM’s ability to repress gene expression and thereby reset the oscillation. Much of the hard work of the study went into figuring out how to produce the complex of cryptochrome-TIM so it could be studied because TIM is such a large, unwieldy protein, Crane said. To achieve their results, first author Changfan Lin, M.S. ’17, Ph.D. ’21, modified the cryptochrome protein to improve the stability of the cryptochrome-TIM complex and used innovative techniques to purify the samples, making them suitable for high-resolution imaging. “These new methods allowed us to obtain detailed images of the protein structures and gain valuable insights into their function, said Lin, a Friedrich’s Ataxia Research Alliance Postdoctoral Fellow at the California Institute of Technology. “This research not only deepens our understanding of circadian rhythm regulation but also opens up new possibilities for developing therapies targeting related processes.” Co-author Shi Feng, a doctoral student in the field of biophysics, did much of the cryo-electron microscopy work. Cristina C. DeOliveira, a doctoral student in the field of biochemistry and molecular and cell biology, was also a co-author. One unexpected result from the study sheds light on how DNA damage is repaired in a cell. Cryptochromes are closely related to a family of enzymes involved in repairing damage to DNA, called photolyases. Crane said the research “explains why these families of proteins are closely related to each other, even though they’re doing quite different things – they’re making use of the same molecular recognition in different contexts.” Genetic Variations and Environmental Adaptations The study also offers an explanation for the genetic variation of flies that allows them to adapt to higher latitudes, where days are shorter in the winter and it’s cooler. These flies have more of a certain genetic variant that involves a change in the TIM protein, and it wasn’t clear why the variation could help them. The researchers found that because of how the cryptochrome binds TIM, the variation reduces the affinity of TIM for the cryptochrome. The interaction between the proteins is then modulated and the ability of light to reset the oscillation is changed, thus altering the circadian clock and extending the period of the fly’s dormancy, which helps it survive the winter. “Some of the interactions that we see here in the fruit fly can be mapped onto human proteins,” Crane said. “This study may help us understand key interactions between components that regulate sleep behavior in people, such as how the critical delays in the basic timing mechanism get built into the system.” Another exciting finding, said Lin, was the discovery of an important structural area in TIM, called the “groove,” which helps explain how TIM enters the cell nucleus. Previous studies had identified some factors involved in this process, but the exact mechanism remained unclear. “Our research provided a clearer understanding of this phenomenon,” Lin said. Reference: “Cryptochrome–Timeless structure reveals circadian clock timing mechanisms” by Changfan Lin, Shi Feng, Cristina C. DeOliveira and Brian R. Crane, 26 April 2023, Nature. DOI: 10.1038/s41586-023-06009-4 Marine bacteria in the cold waters of the Canadian Arctic can biodegrade oil and diesel fuel. Marine bacteria in the frigid waters of the Canadian Arctic are capable of biodegrading oil and diesel fuel, according to a new study published in Applied and Environmental Microbiology, a journal of the American Society for Microbiology. Genomic sequencing revealed unexpected potential for hydrocarbon bioremediation in lineages of bacteria including Paraperlucidibaca, Cycloclasticus, and Zhongshania, said coauthor Casey Hubert, Ph.D., Associate Professor of Geomicrobiology, University of Calgary. These “may represent key players in the response to Arctic marine oil spills.” “The study also confirmed that providing nutrients can enhance hydrocarbon biodegradation under these low temperature conditions,” said Dr. Hubert. The impetus for this work: “These permanently cold waters are seeing increasing industrial activity related to maritime shipping and offshore oil and gas sector activities,” said Dr. Hubert. Sean Murphy, Dr. Hubert’s student, who grew up in the region, instigated the project. Mr. Murphy, Aquatic Scientist, ERM Canada, had noted both the benefit offshore oil had brought to the people of Newfoundland and Labrador, but had been deeply troubled by the Deepwater Horizon oil spill, and had focused his master’s research on the Labrador Sea to “help inform future oil spill mitigation strategies… at cold temperatures in the region.” The Labrador coast—where the study took place—is important for Indigenous peoples who rely on the ocean for food, and unlike at lower latitudes, there’s been a dearth of research on bioremediation this far north, Dr. Hubert noted. “As climate change extends ice-free periods and increasing industrial activity takes place in the Arctic, it is important to understand the ways in which the Arctic marine microbiome will respond if there is an oil or fuel spill,” said Dr. Hubert. That’s especially important, as “this region remains vast and remote such that oil spill emergency response would be complicated and slow.” In the study, the investigators simulated oil spill remediation inside of bottles, by combining mud from the top few centimeters of seabed with artificial seawater, and with either diesel or crude oil, along with different nutrient amendments at different concentrations. The experiments were performed at 4°C, to approximate the temperature in the Labrador Sea, and took place over several weeks. “Our simulations demonstrated that naturally occurring oil-degrading bacteria in the ocean represent nature’s first responders to an oil spill,” said Dr. Hubert. Reference: “Biodegradation of diesel and crude oil by Labrador Sea cold adapted microbial communities” by Sean M. C. Murphy, María A. Bautista, Margaret A. Cramm and Casey R. J. Hubert, 11 August 2021, Applied and Environmental Microbiology. DOI: 10.1128/AEM.00800-21 The American Society for Microbiology is one of the largest professional societies dedicated to the life sciences and is composed of 30,000 scientists and health practitioners. ASM’s mission is to promote and advance the microbial sciences. Fruit fly germline stem cells, the cells that make sperm or eggs. Credit Jonathan Nelson/ Whitehead Institute The genetic makeup of many species, including humans, contains crucial components known as ribosomal DNA (rDNA) sequences. Due to their highly repetitive pattern, these DNA sequences tend to reduce in size over time, leading to cell death if they shrink excessively. If this happens in germ cells — cells that give rise to eggs and sperm — it can result in infertility and the potential extinction of the individual’s lineage. Scientists have long theorized that some mechanism works to preserve our rDNA over successive generations, thereby maintaining the fertility of humans and other species. However, the specifics of this process remained unclear until recently. New findings from Yukiko Yamashita, a member of the Whitehead Institute, and postdoc Jonathan Nelson, unveiled an unexpected defender of rDNA: a retrotransposon. Prior to this discovery, retrotransposons were predominantly considered genetic parasites because they seemed to exist only to replicate themselves. Their research, recently published in the journal PNAS, explains how this so-called parasite actually plays an essential role in maintaining rDNA and preserving fertility through the generations. The Puzzle of Why rDNA Does Not Disappear rDNA generates the RNA subunits of ribosomes, the cellular machines that make proteins, the cells’ essential workers, by translating genes. Our cells require many ribosomes to make all of the proteins they need to function, so rDNA is full of repeated copies of the sequence for making ribosome parts. The problem with this kind of repetitive DNA is that it’s easy for the cell to accidentally remove some of the identical repeats when replicating the genome during cell division. Over time, as cells go through multiple divisions, the number of repeats would be expected to get smaller and smaller. This problem would be particularly noticeable in the cells of aging individuals and in germ cells, the only cells that get passed from one generation to the next. If nothing were helping rDNA to recover its missing repeats, then each new generation would start out with fewer repeats than the last, until a generation did not have enough repeats left to make viable germ cells—and so that population would die out. Yamashita, who is also a professor of biology at the Massachusetts Institute of Technology and an Investigator with the Howard Hughes Medical Institute, studies germ cell immortality in male fruit flies (Drosophila melanogaster). That is, she studies how germ cells can keep making healthy sperm and eggs throughout many generations of individuals. Every other type of cell dies with the body it is born in, and so the genomes of these cells can accrue some damage over time—such as losing repeats in their rDNA—without much consequence. However, errors in the germ cell genome can accumulate over the generations, so germ cells must be especially careful to maintain their rDNA in order to preserve their immortality. When germ cells lose too many rDNA repeats, they are able to replace them with new repeats, but no one has known how they were able to do this. Nelson and Yamashita set out to find the answer. “Ribosomal DNA is repetitive and so is bound to be lost, and the logical consequence is that we should all lose the rDNA in our germ cells and the future generations would be totally gone,” Yamashita says. “So how come that hasn’t happened yet? This is the kind of question that’s so big you don’t even see it at first—you take it for granted that something is maintaining rDNA—but once we saw that the question was there, we needed to find the answer.” Retrotransposons: Not So Selfish After All What the researchers discovered is that rDNA is restored with the help of a retrotransposon, R2. Retrotransposons are genetic sequences whose primary function is to replicate themselves, even at the expense of the rest of the genome. They have been called genetic parasites, but their behavior is most similar to that of a virus, which manipulates cells into making copies of itself. The way a retrotransposon makes more copies of itself is by reversing the usual process of gene expression. When the DNA coding for a retrotransposon is read into RNA, that RNA can be read back into DNA. The retrotransposon then slices open the cell’s genome and inserts its new DNA, adding another copy of itself to the genome. This process not only balloons the size of a species’ genome over generations—nearly half of the human genome consists of transposable elements—but it can also cause damage to an individual cell. When a retrotransposon slices open the genome, especially if it then inserts itself into the middle of a necessary DNA sequence, that can render important genes unusable. However, Nelson and Yamashita found that the retrotransposon R2, which typically copies and inserts itself into fruit fly rDNA, can also help cells. In a dividing cell, there are two copies of each chromosome—one to go in each of the new daughter cells. R2 slices open both copies of the chromosome containing rDNA. When the cell tries to repair these breaks, the repetitive nature of the rDNA can essentially make it lose its place, so it stitches a stretch of rDNA repeats from one copy of the chromosome into the other copy of the chromosome instead. This means that one of the daughter cells will end up with more repeats in its rDNA than the original cell had, while the other daughter cell will have fewer repeats. The germ cells can then protect their immortality by making sure that the cell with more repeats in its rDNA is the one used to keep the germline going. Another paper from Yamashita’s lab, published in 2022, identified how germ cells make this selection. Germ cells divide asymmetrically, so one of the new daughter cells remains a germline stem cell, continuing to make more germ cells, and the other daughter cell differentiates or begins down the path of making sperm. Yamashita lab postdoc George Watase and Yamashita discovered a gene, which they named Indra, that creates a protein that attaches to the copy of the chromosome containing more rDNA repeats. This protein marks the daughter cell containing that chromosome to remain a stem cell, while the other daughter cell goes on to make sperm. Germ cells can combine these mechanisms, taking rDNA repeats from one chromosome to give to another and then earmarking the cell with more repeats, to constantly replenish the germline’s level of rDNA. This ensures that the number of rDNA repeats never gets too low across the population of germ cells, preserving the lineage of the cells and the individuals who carry them. Nelson and Yamashita’s work shows that R2 is not merely a selfish parasite, but instead plays a pivotal role in this process of germline rDNA rejuvenation. However, as a retrotransposon, R2 is also capable of causing damage. Nelson found that germ cells keep R2 inactive except in cases where the number of repeats in rDNA is too low. In this way, the cells may maximize the benefits of R2 and minimize its dangers, by only accepting the risk of damage when needed. This may allow the cell and retrotransposon to have a mutually beneficial relationship. Yamashita and Nelson speculate that other transposable elements may likewise provide unknown benefits to the cell. “A lot of transposable elements are thought of as existing because their ability to replicate in the genome is better than the ability of the host to defend itself from that replication,” Nelson says. “These elements make up large regions of the genome that we think of as non-functional, but what if the reason why there are so many of them is because they contribute some function that we just don’t understand yet?” References: “The retrotransposon R2 maintains Drosophila ribosomal DNA repeats” by Jonathan O. Nelson, Alyssa Slicko and Yukiko M. Yamashita, 30 May 2023, Proceedings of the National Academy of Sciences. DOI: 10.1073/pnas.2221613120 “Nonrandom sister chromatid segregation mediates rDNA copy number maintenance in Drosophila” by George J. Watase, Jonathan O. Nelson and Yukiko M. Yamashita, 27 July 2022, Science Advances. DOI: 10.1126/sciadv.abo4443 RRG455KLJIEVEWWF |
||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
| 最新創作 |
|
||||
|
||||
|
||||
|
||||
|
||||
