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文章數：79 |
 NINI 尼尼台中店長官聚餐合適嗎？》公益路美食推薦｜吃貨實測十間真心話 |
| 興趣嗜好｜偶像追星 2026/04/20 11:35:45 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人，臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」，從精緻西餐到創意火鍋，從日式丼飯到義式早午餐，每走幾步，就會有完全不同的特色料理餐廳。 這次我特別花了一整個月，實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店，也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準，整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你，找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型，從高質感牛排館到巷弄系早午餐，每一間都有自己獨特的風格。為了讓整體比較更客觀，我依照以下四大面向進行評比，並搭配實際用餐體驗來打分。
整體而言，我希望這份評比不只是「哪家好吃」，而是幫你在不同情境下（約會、家庭聚餐、朋友小聚、商業午餐）都能快速找到合適的選擇。畢竟，美食不只是味覺的滿足，更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段，從火鍋、燒肉到中式料理與早午餐，每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單，橫跨平價、創意、高級各路風格。
一頭牛日式燒肉｜炭香濃郁的和牛饗宴，約會聚餐首選
走在公益路上，很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面，搭配昏黃燈光與暖色調的內裝，讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大，但桌距規劃得宜，每桌皆設有獨立排煙設備，烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂，絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網，讓人完全不用分心。整場用餐過程就像一場表演，從視覺、嗅覺到味覺都被滿足。 綜合評分
地址：408臺中市南屯區公益路二段162號電話：04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」，更像是一場五感盛宴。從進門那一刻到最後一道甜點，都能感受到他們對細節的用心。 TANG Zhan 湯棧｜文青系火鍋代表，麻香湯底與視覺美感並重
在公益路這條美食戰線上，TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址：408臺中市南屯區公益路二段248號電話：04-22580617 官網：https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度，又結合現代風格與細節服務，讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店，湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店｜明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂，那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主，陽光灑進室內，讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧，建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味，選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口，米芯保留微Q口感；而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香，口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹，無論是一個人帶電腦工作、或朋友聚餐，都能找到舒服角落。餐點上桌速度穩定，服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」，很適合想遠離忙碌日常的人。 綜合評分
地址：40861臺中市南屯區公益路二段18號電話：04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇，而是以「剛剛好」的份量與風味，陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店，NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物｜平價卻用心的湯頭系火鍋，家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上，加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐，但靠著實在的湯頭與親切的服務，默默吸引許多回頭客。每到用餐時間，總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底，湯頭清澈卻不單薄，越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向，桌距寬敞、座位舒適，帶小孩來也不覺擁擠。店員態度親切，補湯、收盤都很勤快，給人一種「被照顧著」的安心感。 綜合評分
地址：403臺中市西區公益路288號電話：0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤，而是用最簡單的湯頭與新鮮食材，傳遞出家常卻不平凡的溫度。 印月餐廳｜中式料理的藝術演繹，宴客與家庭聚會首選
說到臺中公益路的中式料理代表，印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名，把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設，每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏，都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法，讓人感受到「被款待」的尊榮感。 綜合評分
地址：408臺中市南屯區公益路二段818號電話：0422511155 小結語印月餐廳是一間「不只吃飯，更像品味生活」的地方。 KoDō 和牛燒肉｜極致職人精神，專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗，一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉，以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址：403臺中市西區公益路260號電話：0423220312 官網：https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳，而是一場體驗。 永心鳳茶｜在茶香裡用餐的優雅時光，臺味早午餐的新詮釋
走進 永心鳳茶公益店，彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤，無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」，都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和，對茶品介紹詳盡。上餐節奏剛好，不急不徐。 綜合評分
地址：40360臺中市西區公益路68號三樓(勤美誠品)電話：0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓｜老饕級江浙功夫菜，穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主，兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址：408臺中市南屯區公益路二段95號電話：0423202322 官網：https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司｜低調奢華的無菜單日料，職人手藝詮釋旬味極致
在熱鬧的公益路上，一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase（無菜單料理） 形式，每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘，節奏緩慢但沉穩。 綜合評分
地址：408臺中市南屯區公益路二段25號電話：0423206368 官網：https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂｜人氣爆棚的和牛燒肉聖地，肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳，茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐，價格約落在 $700–$1000 間，份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快，整場用餐流程流暢無壓力。 綜合評分
地址：403臺中市西區公益路268號電話：0423281167 官網：https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質＋輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後，臺中公益路不只是一條美食街，而是一段生活風景線。 有的餐廳講究細膩與儀式感，像 一頭牛日式燒肉 與 一笈壽司，讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心，像 加分昆布鍋物、永心鳳茶，讓人明白吃飯不只是為了飽足，而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店，則用穩定的品質與熱絡的氛圍，成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店，構成了公益路最動人的縮影 有華麗的，也有溫柔的；有傳統的，也有創新的。 每一家都在自己的風格裡發光，讓人吃到的不只是料理，而是一種生活的溫度與節奏。 對我而言，這不僅是一場美食旅程，更是一趟關於「臺中味道」的回憶之旅。 FAQ：關於臺中公益路美食常見問題Q1：公益路哪一區的餐廳最集中？ Q2：需要提前訂位嗎？ 最後的話若要用一句話形容這趟美食之旅，我會說： TANG Zhan 湯棧春酒菜色豐富嗎？ 如果你也和我一樣喜歡用味蕾探索一座城市，那就把這篇公益路美食攻略收藏起來吧。一頭牛日式燒肉有什麼隱藏版必點嗎？ 無論是約會、慶生、家庭聚餐，或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。茶六燒肉堂尾牙預算好掌控嗎？ 下一餐，不妨從這10家開始。永心鳳茶真的有那麼好吃嗎？ 打開手機、約上朋友，讓公益路成為你生活裡最容易抵達的小確幸。一頭牛日式燒肉再訪意願高嗎？ 如果你有私心愛店，也歡迎留言分享，一笈壽司小孩適合去嗎？ 你的推薦，可能讓我下一趟美食旅程變得更精彩。NINI 尼尼臺中店值得推薦嗎？ Ninjurin-1 proteins assemble (green/yellow) into filaments and rupture the cell membrane (gray) until the cell disintegrates completely. Intracellular components are shown in blue. Credit: Biozentrum, University of Basel Scientists from the University of Basel have uncovered the mechanism by which cells rupture during programmed cell death. In our bodies, millions of cells meet their end on a daily basis. Contrary to popular belief, cells don’t just explode when they die. Instead, a particular protein acts as a trigger for the rupture of the cell membrane. Scientists from the University of Basel have recently been able to elucidate the exact mechanism at the atomic level. Their findings are published in the journal Nature. The self-elimination of cells is a vital process for all living organisms. When cells become damaged or infected with viruses or bacteria, they initiate an internal “self-destruct” sequence. This essential mechanism wards off the potential growth of tumors and prevents the spread of harmful pathogens throughout the body. Until recently, it was assumed that cells simply burst and die at the end of their life. Now, researchers at the Biozentrum of the University of Basel, the University of Lausanne, and the Department of Biosystems Science and Engineering (D-BSSE) at ETH Zurich have provided new insights into the final step of cell death. In the scientific journal Nature, they describe how a protein called ninjurin-1 assembles into filaments that work like a zipper and open the cell membrane, thus leading to the disintegration of the cell. The new insights are an important milestone in the understanding of cell death. Protein Acts as a Breaking Point in the Cell Membrane Various signals, such as bacterial components, trigger the cell death machinery. At the final stage of this process, the cell’s protective membrane is compromised by tiny pores which allow ions to stream into the cell. “The common understanding was that the cell then swells until it finally bursts due to increasing osmotic pressure,” explains Professor Sebastian Hiller who heads a research group at the Biozentrum, University of Basel. “We are now resolving how the cells really rupture. Instead of bursting like a balloon, the protein ninjurin-1 provides a breaking point in the cell membrane, causing rupture at specific sites.” At the end of their lives, cells do not simply burst. Instead, a specific protein serves as a breaking point for the cell membrane rupture. SNI PhD student Morris Degen (Biozentrum, University of Basel) explains how this mechanism works. Credit: Swiss Nanoscience Institute Using advanced techniques such as highly sensitive microscopes and NMR spectroscopy, the scientists have been able to elucidate the mechanism by which ninjurin-1 induces membrane rupture at the level of individual atoms. Ninjurin-1 is a small protein embedded in the cell membrane. “Upon receiving the suicide command, two ninjurin-1 proteins initially cluster together and drive a wedge into the membrane,” explains Morris Degen, first author of the study and Ph.D. student at the Ph.D. School of the Swiss Nanoscience Institute. “Large lesions and holes are formed by many further proteins attaching to the initial wedge. In this way, the cell membrane is cleaved open piece by piece until the cell disintegrates completely.” The cell debris is then removed by the body’s own cleaning service. “It is now evident that the cells do not burst without ninjurin-1. They do swell to a certain extent due to the influx of ions, but membrane rupture is contingent on the function of this protein,” adds Hiller. “The textbook’s chapter on cell death will be expanded with these beautiful structural insights.” Therapy To Prevent or Promote Cell Death A deeper understanding of cell death will facilitate the search for novel drug targets. Therapeutic interventions to treat cancer are conceivable since some tumor cells evade programmed cell death. Also, in the case of premature cell death observed in neurodegenerative diseases such as Parkinson’s disease or in life-threatening conditions such as septic shock, drugs that interfere in this process are a potential treatment option. Reference: “Structural basis of NINJ1-mediated plasma membrane rupture in cell death” by Morris Degen, José Carlos Santos, Kristyna Pluhackova, Gonzalo Cebrero, Saray Ramos, Gytis Jankevicius, Ella Hartenian, Undina Guillerm, Stefania A. Mari, Bastian Kohl, Daniel J. Müller, Paul Schanda, Timm Maier, Camilo Perez, Christian Sieben, Petr Broz and Sebastian Hiller, 17 May 2023, Nature. DOI: 10.1038/s41586-023-05991-z A study confirms that there is no genetic evidence for a link between someone’s face and that individual’s behavior. An interdisciplinary team led by KU Leuven and Stanford has identified 76 overlapping genetic locations that shape both our face and our brain. What the researchers didn’t find is evidence that this genetic overlap also predicts someone’s behavioral-cognitive traits or risk of conditions such as Alzheimer’s disease. This means that the findings help to debunk several persistent pseudoscientific claims about what our face reveals about us. There were already indications of a genetic link between the shape of our face and that of our brain, says Professor Peter Claes from the Laboratory for Imaging Genetics at KU Leuven, who is the joint senior author of the study with Professor Joanna Wysocka from the Stanford University School of Medicine. “But our knowledge on this link was based on model organism research and clinical knowledge of extremely rare conditions,” Claes continues. “We set out to map the genetic link between individuals’ face and brain shape much more broadly, and for commonly occurring genetic variation in the larger, non-clinical population.” Brain scans and DNA from the UK Biobank To study genetic underpinnings of brain shape, the team applied a methodology that Peter Claes and his colleagues had already used in the past to identify genes that determine the shape of our face. Claes: “In these previous studies, we analyzed 3D images of faces and linked several data points on these faces to genetic information to find correlations.” This way, the researchers were able to identify various genes that shape our face. For the current study, the team relied on these previously acquired insights as well as the data available in the UK Biobank, a database from which they used the MRI brain scans and genetic information of 20,000 individuals. Claes: “To be able to analyse the MRI scans, we had to measure the brains shown on the scans. Our specific focus was on variations in the folded external surface of the brain – the typical ‘walnut shape’. We then went on to link the data from the image analyses to the available genetic information. This way, we identified 472 genomic locations that have an impact on the shape of our brain. 351 of these locations have never been reported before. To our surprise, we found that as many as 76 genomic locations predictive of the brain shape had previously already been found to be linked to the face shape. This makes the genetic link between face and brain shape a convincing one.” The team also found evidence that genetic signals that influence both brain and face shape are enriched in the regions of the genome that regulate gene activity during embryogenesis, either in facial progenitor cells or in the developing brain. This makes sense, Wysocka explains, as the development of the brain and the face are coordinated. “But we did not expect that this developmental cross-talk would be so genetically complex and would have such a broad impact on human variation.” No genetic link with behavior or neuropsychiatric disorders At least as important is what the researchers did not find, says Dr. Sahin Naqvi from the Stanford University School of Medicine, who is the first author of this study. “We found a clear genetic link between someone’s face and their brain shape, but this overlap is almost completely unrelated to that individual’s behavioral-cognitive traits.” Concretely: even with advanced technologies, it is impossible to predict someone’s behavior based on their facial features. Peter Claes continues: “Our results confirm that there is no genetic evidence for a link between someone’s face and that individual’s behavior. Therefore, we explicitly dissociate ourselves from pseudoscientific claims to the contrary. For instance, some people claim that they can detect aggressive tendencies in faces by means of artificial intelligence. Not only are such projects completely unethical, they also lack a scientific foundation.” In their study, the authors also briefly address conditions such as Alzheimer’s, schizophrenia, and bipolar disorder. Claes: “As a starting point, we used the results that were previously published by other teams about the genetic basis of such neuropsychiatric disorders. The possible link with the genes that determine the shape of our face had never been examined before. If you compare existing findings with our new ones, you see a relatively large overlap between the genetic variants that contribute to specific neuropsychiatric disorders and those that play a role in the shape of our brain, but not for those that contribute to our face.” In other words: our risk of developing a neuropsychiatric disorder is not written on our face either. Reference: “Shared heritability of human face and brain shape” by Sahin Naqvi, Yoeri Sleyp, Hanne Hoskens, Karlijne Indencleef, Jeffrey P. Spence, Rose Bruffaerts, Ahmed Radwan, Ryan J. Eller, Stephen Richmond, Mark D. Shriver, John R. Shaffer, Seth M. Weinberg, Susan Walsh, James Thompson, Jonathan K. Pritchard, Stefan Sunaert, Hilde Peeters, Joanna Wysocka and Peter Claes, 5 April 2021, Nature Genetics. DOI: 10.1038/s41588-021-00827-w These distant primate cousins of humans are among the few mammal species in which male-female partners stick together year after year. Credit: David Haring, Duke Lemur Center Lemurs Show There’s No Single Formula for Lasting Love Humans aren’t the only mammals that form long-term bonds with a single, special mate — some bats, wolves, beavers, foxes, and other animals do, too. But new research suggests the brain circuitry that makes love last in some species may not be the same in others. The study, appearing February 12 in the journal Scientific Reports, compares monogamous and promiscuous species within a closely related group of lemurs, distant primate cousins of humans from the island Madagascar. Red-bellied lemurs and mongoose lemurs are among the few species in the lemur family tree in which male-female partners stick together year after year, working together to raise their young and defend their territory. Once bonded, pairs spend much of their waking hours grooming each other or huddled side by side, often with their tails wrapped around each other’s bodies. Males and females of these species spend a third of their lifetime with the same mate. The same cannot be said of their closest relatives, who change partners often. The Rarity of Mammalian Monogamy To biologists, monogamy is somewhat a mystery. That’s in part because in many animal groups it’s rare. While around 90% of bird species practice some form of fidelity to one partner, only 3% to 5% of mammals do. The vast majority of the roughly 6,500 known species of mammals have open relationships, so to speak. “It’s an uncommon arrangement,” said lead author Nicholas Grebe, a postdoctoral associate in professor Christine Drea’s lab at Duke University. Which raises a question: what makes some species biologically inclined to pair up for the long haul while others play the field? Hormones Behind Lasting Bonds Studies over the last 30 years in rodents point to two hormones released during mating, oxytocin, and vasopressin, suggesting that the key to lasting love may lie in differences in how they act on the brain. Some of the first clues came from influential research on prairie voles, small mouse-like mammals that, unlike most rodents, mate for life. When researchers compared the brains of monogamous prairie voles with their promiscuous counterparts, montane voles, and meadow voles, they found that prairie voles had more “docking sites” for these hormones, particularly in parts of the brain’s reward system. Since these “cuddle chemicals” were found to enhance male-female bonds in voles, researchers have long wondered if they might work the same way in humans. That’s why the Duke-led team turned to lemurs. Despite being our most distant primate relatives, lemurs are a closer genetic match to humans than voles are. The researchers used an imaging technique called autoradiography to map binding sites for oxytocin and vasopressin in the brains of 12 lemurs that had died of natural causes at the Duke Lemur Center. The animals represented seven species: monogamous red-bellied and mongoose lemurs along with five promiscuous species in the same genus. “They’re really the only comparable natural experiment to look for biological signatures of monogamy in primates,” Grebe said. Comparing the brain imaging results in lemurs with previous results in voles and monkeys revealed some noticeable differences in the density and distribution of hormone receptors. In other words, oxytocin and vasopressin appear to act on different parts of the brain in lemurs — which means they may also have different effects, depending on their target cell’s location. But within lemurs, the researchers were surprised to find few consistent differences between monogamous species and promiscuous ones. “We don’t see evidence of a pair-bond circuit” akin to that found in rodent brains, Grebe said. Testing the Limits of Oxytocin As a next step, the team is looking at how lemur couples behave toward each other if the actions of oxytocin are blocked, by feeding them an antagonist that temporarily prevents oxytocin from binding to its receptors in the brain. So what can lemurs teach us about love? The authors say their findings caution against drawing simple conclusions based on rodent experiments about how human social behaviors came to be. Oxytocin may be the “potion of devotion” for voles, but it may be the combined actions and interactions of multiple brain chemicals, along with ecological factors, that create long-lasting bonds in lemurs and other primates, including humans, Grebe said. “There are probably a number of different ways through which monogamy is instantiated within the brain, and it depends on what animals we’re looking at,” Grebe said. “There’s more going on than we originally thought.” Reference: “Neural Correlates of Mating System Diversity: Oxytocin and Vasopressin Receptor Distributions in Monogamous and Non-Monogamous Eulemur” by Nicholas M. Grebe, Annika Sharma, Sara M. Freeman, Michelle C. Palumbo, Heather B. Patisaul, Karen L. Bales and Christine M. Drea, 12 February 2021, Scientific Reports. DOI: 10.1038/s41598-021-83342-6 Other authors were: Annika Sharma at Duke, Sara Freeman at Utah State University, Michelle Palumbo at the California National Primate Research Center, Heather Patisaul at North Carolina State University, and Karen Bales at the University of California, Davis. This work was supported by grants from the National Science Foundation (SBE-1808803), the National Institute of Mental Health (NIMH R21MH115680), the Josiah Charles Trent Memorial Foundation Endowment Fund, the Charles Lafitte Foundation for Research, and Duke University. RRG455KLJIEVEWWF |
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