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文章數：238 |
 一頭牛日式燒肉適合跨年聚餐嗎？》台中公益路人氣餐廳10選｜吃過都說讚 |
| 興趣嗜好｜偶像追星 2026/04/20 13:50:28 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人，臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」，從精緻西餐到創意火鍋，從日式丼飯到義式早午餐，每走幾步，就會有完全不同的特色料理餐廳。 這次我特別花了一整個月，實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店，也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準，整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你，找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型，從高質感牛排館到巷弄系早午餐，每一間都有自己獨特的風格。為了讓整體比較更客觀，我依照以下四大面向進行評比，並搭配實際用餐體驗來打分。
整體而言，我希望這份評比不只是「哪家好吃」，而是幫你在不同情境下（約會、家庭聚餐、朋友小聚、商業午餐）都能快速找到合適的選擇。畢竟，美食不只是味覺的滿足，更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段，從火鍋、燒肉到中式料理與早午餐，每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單，橫跨平價、創意、高級各路風格。
一頭牛日式燒肉｜炭香濃郁的和牛饗宴，約會聚餐首選
走在公益路上，很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面，搭配昏黃燈光與暖色調的內裝，讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大，但桌距規劃得宜，每桌皆設有獨立排煙設備，烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂，絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網，讓人完全不用分心。整場用餐過程就像一場表演，從視覺、嗅覺到味覺都被滿足。 綜合評分
地址：408臺中市南屯區公益路二段162號電話：04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」，更像是一場五感盛宴。從進門那一刻到最後一道甜點，都能感受到他們對細節的用心。 TANG Zhan 湯棧｜文青系火鍋代表，麻香湯底與視覺美感並重
在公益路這條美食戰線上，TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址：408臺中市南屯區公益路二段248號電話：04-22580617 官網：https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度，又結合現代風格與細節服務，讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店，湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店｜明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂，那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主，陽光灑進室內，讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧，建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味，選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口，米芯保留微Q口感；而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香，口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹，無論是一個人帶電腦工作、或朋友聚餐，都能找到舒服角落。餐點上桌速度穩定，服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」，很適合想遠離忙碌日常的人。 綜合評分
地址：40861臺中市南屯區公益路二段18號電話：04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇，而是以「剛剛好」的份量與風味，陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店，NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物｜平價卻用心的湯頭系火鍋，家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上，加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐，但靠著實在的湯頭與親切的服務，默默吸引許多回頭客。每到用餐時間，總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底，湯頭清澈卻不單薄，越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向，桌距寬敞、座位舒適，帶小孩來也不覺擁擠。店員態度親切，補湯、收盤都很勤快，給人一種「被照顧著」的安心感。 綜合評分
地址：403臺中市西區公益路288號電話：0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤，而是用最簡單的湯頭與新鮮食材，傳遞出家常卻不平凡的溫度。 印月餐廳｜中式料理的藝術演繹，宴客與家庭聚會首選
說到臺中公益路的中式料理代表，印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名，把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設，每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏，都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法，讓人感受到「被款待」的尊榮感。 綜合評分
地址：408臺中市南屯區公益路二段818號電話：0422511155 小結語印月餐廳是一間「不只吃飯，更像品味生活」的地方。 KoDō 和牛燒肉｜極致職人精神，專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗，一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉，以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址：403臺中市西區公益路260號電話：0423220312 官網：https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳，而是一場體驗。 永心鳳茶｜在茶香裡用餐的優雅時光，臺味早午餐的新詮釋
走進 永心鳳茶公益店，彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤，無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」，都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和，對茶品介紹詳盡。上餐節奏剛好，不急不徐。 綜合評分
地址：40360臺中市西區公益路68號三樓(勤美誠品)電話：0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓｜老饕級江浙功夫菜，穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主，兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址：408臺中市南屯區公益路二段95號電話：0423202322 官網：https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司｜低調奢華的無菜單日料，職人手藝詮釋旬味極致
在熱鬧的公益路上，一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase（無菜單料理） 形式，每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘，節奏緩慢但沉穩。 綜合評分
地址：408臺中市南屯區公益路二段25號電話：0423206368 官網：https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂｜人氣爆棚的和牛燒肉聖地，肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳，茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐，價格約落在 $700–$1000 間，份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快，整場用餐流程流暢無壓力。 綜合評分
地址：403臺中市西區公益路268號電話：0423281167 官網：https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質＋輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後，臺中公益路不只是一條美食街，而是一段生活風景線。 有的餐廳講究細膩與儀式感，像 一頭牛日式燒肉 與 一笈壽司，讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心，像 加分昆布鍋物、永心鳳茶，讓人明白吃飯不只是為了飽足，而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店，則用穩定的品質與熱絡的氛圍，成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店，構成了公益路最動人的縮影 有華麗的，也有溫柔的；有傳統的，也有創新的。 每一家都在自己的風格裡發光，讓人吃到的不只是料理，而是一種生活的溫度與節奏。 對我而言，這不僅是一場美食旅程，更是一趟關於「臺中味道」的回憶之旅。 FAQ：關於臺中公益路美食常見問題Q1：公益路哪一區的餐廳最集中？ Q2：需要提前訂位嗎？ 最後的話若要用一句話形容這趟美食之旅，我會說： 永心鳳茶好吃嗎？ 如果你也和我一樣喜歡用味蕾探索一座城市，那就把這篇公益路美食攻略收藏起來吧。TANG Zhan 湯棧必點有哪些？ 無論是約會、慶生、家庭聚餐，或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。一笈壽司適合請客嗎？ 下一餐，不妨從這10家開始。印月餐廳再訪意願高嗎？ 打開手機、約上朋友，讓公益路成為你生活裡最容易抵達的小確幸。印月餐廳用餐時間會不會太短？ 如果你有私心愛店，也歡迎留言分享，一頭牛日式燒肉婚前派對適合嗎？ 你的推薦，可能讓我下一趟美食旅程變得更精彩。三希樓適合約會嗎？ Poor gut health may worsen COVID-19 prognosis. This could occur by allowing the virus access to the digestive tract and internal organs, which are vulnerable due to the presence of ACE2 protein. COVID-19 severity may be tied to gut microbiome imbalances, especially in people with chronic illness or fiber-poor diets. Severe cases of COVID-19 often include GI symptoms Chronic diseases associated with severe COVID-19 are also associated with altered gut microbiota A growing body of evidence suggests poor gut health adversely affects prognosis If studies do empirically demonstrate a connection between the gut microbiota and COVID-19 severity, then interventions like probiotics or fecal transplants may help patients People infected with COVID-19 experience a wide range of symptoms and severities, the most commonly reported including high fevers and respiratory problems. However, autopsy and other studies have also revealed that the infection can affect the liver, kidney, heart, spleen — and even the gastrointestinal tract. A sizeable fraction of patients hospitalized with breathing problems also have diarrhea, nausea and vomiting, suggesting that when the virus does get involved in the GI tract it increases the severity of the disease. Leaky Gut May Worsen Viral Spread In a review published this week in the journal mBio, microbiologist Heenam Stanley Kim, Ph.D, from Korea University’s Laboratory for Human-Microbial Interactions, in Seoul, examined emerging evidence suggesting that poor gut health adversely affects COVID-19 prognosis. Based on his analysis, Kim proposed that gut dysfunction — and its associated leaky gut — may exacerbate the severity of infection by enabling the virus to access the surface of the digestive tract and internal organs. These organs are vulnerable to infection because they have widespread ACE2 — a protein target of SARS-CoV-2 — on the surface. “There seems to be a clear connection between the altered gut microbiome and severe COVID-19,” Kim said. The virus that causes COVID-19, called SARS-CoV-2, shown here in an electron microscope image. Credit: NIAID-RML Studies have demonstrated that people with underlying medical conditions including high blood pressure, diabetes and obesity face a higher risk of severe COVID-19. Risk also increases with age, with older adults most vulnerable to the most serious complications and likelihood of hospitalization. But both of these factors — advanced age and chronic conditions — have a well-known association with an altered gut microbiota. This imbalance can affect gut barrier integrity, Kim noted, which can allow pathogens and pathobionts easier access to cells in the intestinal lining. So far, the link between gut health and COVID-19 prognosis hasn’t been empirically demonstrated, Kim noted. Some researchers have argued, he said, that unhealthy gut microbiomes may be an underlying reason for why some people have such severe infections. What studies have been done hint at a complicated relationship. A study on symptomatic COVID-19 patients in Singapore, for example, found that about half had a detectable level of the coronavirus in fecal tests — but only about half of those experienced GI symptoms. That study suggests that even if SARS-CoV-2 reaches the GI tract, it may not cause problems. Kim also noted that a person’s gut health at the time of infection may be critical for symptom development. Microbiome Shifts Detected in COVID-19 Patients Many recent studies have found reduced bacterial diversity in gut samples collected from COVID-19 patients, compared to samples from healthy people. The disease has also been linked to a depletion of beneficial bacterial species — and the enrichment of pathogenic ones. A similar imbalance has been associated with influenza A infection, though the 2 viruses differ in how they change the overall microbial composition. The depleted bacterial species associated with COVID-19 infection include some families that are responsible for producing butyrate, a short-chain fatty acid, which plays a pivotal role in gut health by reinforcing gut-barrier function. Kim said he started analyzing the studies after realizing that wealthy countries with a good medical infrastructure — including the United States and nations in Western Europe — were among the hardest hit by the virus. The “western diet” that’s common in these countries is low in fiber, and “a fiber-deficient diet is one of the main causes of altered gut microbiomes,” he said, “and such gut microbiome dysbiosis leads to chronic diseases.” Fiber and Microbiome Restoration as Future Therapies The pathogenesis of COVID-19 is still not fully understood. If future studies do show that gut health affects COVID-19 prognosis, Kim argued, then clinicians and researchers should exploit that connection for better strategies aimed at preventing and managing the disease. Eating more fiber, he said, may lower a person’s risk of serious disease. And fecal microbiota transplantation might be a treatment worth considering for patients with the worst cases of COVID-19. The problem with gut health goes beyond COVID-19, though, he said. Once the pandemic passes, the world will still have to reckon with chronic diseases and other problems associated with poor gut health. “The whole world is suffering from this COVID-19 pandemic,” Kim said, “but what people do not realize is that the pandemic of damaged gut microbiomes is far more serious now.” Reference: “Do an Altered Gut Microbiota and an Associated Leaky Gut Affect COVID-19 Severity?” by Heenam Stanley Kim, 12 January 2021, mBio. DOI: 10.1128/mBio.03022-20 The American Society for Microbiology is one of the largest professional societies dedicated to the life sciences and is composed of 30,000 scientists and health practitioners. ASM’s mission is to promote and advance the microbial sciences. ASM advances the microbial sciences through conferences, publications, certifications and educational opportunities. It enhances laboratory capacity around the globe through training and resources. It provides a network for scientists in academia, industry and clinical settings. Additionally, ASM promotes a deeper understanding of the microbial sciences to diverse audiences. Organization of mitotic chromosomes (magenta) and spindle microtubules (green) at an early phase of cell division. Shortly after what’s shown in the image, the microtubules will invade the nuclear space. However, chromatin compaction regulated by histone acetylation will prevent the perforation of the chromosomes by microtubules. Credit: ©Gerlich/IMBA How the genome is packed into chromosomes that can be faithfully moved during cell division. Scientists discovered a molecular mechanism that confers special physical properties to chromosomes in dividing human cells to enable their faithful transport to the progeny. The research team showed how a chemical modification establishes a sharp surface boundary on chromosomes, thus allowing them to resist perforation by microtubules of the spindle apparatus. The researchers are from the Gerlich Group at IMBA – Institute of Molecular Biotechnology of the Austrian Academy of Sciences, and their findings are published today (August 3, 2022) in the journal Nature. Exactly one genome copy must be transported to each of the two daughter cells during cell division. Faithful genome segregation requires the packaging of extremely long chromosomal DNA molecules into discrete bodies. This allows them to be efficiently moved by the mitotic spindle, a filament system composed of thousands of microtubules. The new findings by the Gerlich Research Group at IMBA – Institute of Molecular Biotechnology of the Austrian Academy of Sciences – shed light on how mitotic chromosomes resist the constant pushing and pulling forces generated by the microtubules. “Amidst this complex system, the distinct physical properties are conferred to the chromosomes by changing the levels of histone acetylation, a chemical modification within the chromatin fiber,” says IMBA Group Leader Daniel Gerlich. Prior research had demonstrated that, in dividing cells, the chromatin fibers are folded into loops by a large protein complex called condensin. However, the role of condensin alone could not explain why chromosomes appear as dense bodies with a sharp surface rather than a loose structure resembling a bottlebrush. Some studies had suggested a role of histone acetylation in regulating the level of compaction during cell division, but the interplay of histone acetylation with condensin and its functional relevance remained unclear. “With our work, we are now able to conceptually disentangle the two mechanisms,” states Gerlich. Disentangling the Effects of Condensin and Histone Acetylation The scientists varied the levels of condensin and histone acetylation to study their precise effects. Removing condensin disrupted the elongated shape of chromosomes in dividing cells and lowered their resistance to pulling forces but did not affect their level of compaction. Combining condensin depletion with a treatment that increases the levels of histone acetylation caused massive chromatin decompaction in dividing cells, and perforation of chromosomes by microtubules. The team hypothesized that chromatin is organized as a swollen gel throughout most of the cell cycle (when it is relatively highly acetylated) and that this gel compacts to an insoluble form during cell division when the acetylation levels globally decrease. They then developed an assay to probe the solubility of chromatin by fragmenting mitotic chromosomes into small pieces. The fragments of mitotic chromosomes formed droplets of liquid chromatin, but when the acetylation level was increased, the chromatin fragments dissolved in the cytoplasm. These observations support a model where a global reduction of chromatin acetylation during mitosis establishes an immiscible chromatin gel with a sharp phase boundary, providing a physical basis for resistance against microtubule perforation. With further experiments involving pure chromatin that was reconstituted in vitro, and by probing chromatin access by various soluble macromolecules, the researchers discovered that immiscible chromatin forms a structure dense in negative charge that excludes negatively charged macromolecules and microtubules. Cooperation Between Condensin and Chromatin Phase Separation “Our study shows how DNA looping by the condensin complex cooperates with a chromatin phase separation process to build mitotic chromosomes that resist both pulling and pushing forces exerted by the spindle. The deacetylation of histones during cell division hence confers unique physical properties to chromosomes that are required for their faithful segregation,” concludes Daniel Gerlich. Reference: “A mitotic chromatin phase transition prevents perforation by microtubules” by Maximilian W. G. Schneider, Bryan A. Gibson, Shotaro Otsuka, Maximilian F. D. Spicer, Mina Petrovic, Claudia Blaukopf, Christoph C. H. Langer, Paul Batty, Thejaswi Nagaraju, Lynda K. Doolittle, Michael K. Rosen and Daniel W. Gerlich, 3 August 2022, Nature. DOI: 10.1038/s41586-022-05027-y Funding: Austrian Science Fund, Vienna Science and Technology Fund, Vienna Science and Technology Fund, Howard Hughes Medical Institute, NIH/National Institutes of Health, Welch Foundation, Boehringer Ingelheim Fonds A Belgian research team used deep learning models to study how genetic switches control brain cell types across species. Their findings reveal both conserved and divergent regulatory codes between birds and mammals, providing insights into brain evolution and potential applications for disease research. Scientists employ deep learning to analyze and compare gene regulation across various cell types in human and chicken brains. A recent study published in Science by a Belgian research team investigates how genetic switches that regulate gene activity define brain cell types across different species. Using deep learning models trained on brain data from humans, mice, and chickens, the researchers discovered that while some cell types have remained highly conserved between birds and mammals over millions of years of evolution, others have diverged significantly. These findings offer new insights into brain evolution and provide valuable tools for studying how gene regulation influences cell type development across species and in various disease states. Our brain, and by extension our entire body, is made up of many different types of cells. While they share the same DNA, all these cell types have their own shape and function. What makes each cell type different is a complex puzzle that researchers have been trying to put together for decades from short DNA sequences that act like switches, controlling which genes are turned on or off. The fine-tuned regulation of these switches ensures that each type of brain cell uses just the right genetic instructions from the genome to perform its unique role. Scientists refer to the unique patterns of these genetic switches as a regulatory code. AI to crack the code Prof. Stein Aerts and his team at VIB.AI and the VIB-KU Leuven Center for Brain & Disease Research study the fundamental principles of this regulatory code, and how it may impact diseases such as cancer or brain disorders. They develop deep learning methods to help make sense of the huge amount of information on gene regulation they gather from thousands and thousands of individual cells. “Deep-learning models working with the DNA sequence code have helped us enormously to identify regulatory mechanisms across different cell types,” explains Aerts. “Now, we wanted to explore whether this regulatory code could also inform us on how these cell types are conserved across species.” One example of where such a question is highly relevant is in the brain. Despite shared developmental trajectories, the brains of mammals and birds display a strikingly different neuroanatomy. Aerts and his team have now applied deep learning models to assess whether the existing differences and similarities are reflected in shared or divergent regulatory codes. Tool to study evolution Nikolai Hecker and Niklas Kempynck, respectively postdoc and PhD student in the Aerts lab, developed and implemented machine learning models to characterize and compare different types of cells across human, mouse, and chicken brains, covering approximately 320 million years of evolution. But before they could truly compare, they first had to better understand the cell type composition of the chicken brain, so they created a comprehensive transcriptomic atlas. “Our study demonstrates how we can use deep learning to characterize and compare different cell types based on their regulatory codes,” explains Hecker. “We can use these codes to compare genomes of different species, identify which regulatory codes have been evolutionarily preserved, and gain insights into how cell types have evolved.” The team found that while some regulatory cell type codes are highly conserved between birds and mammals, others have evolved differently. Notably, the regulatory codes for certain bird neurons resemble those of deep-layer neurons in the mammalian neocortex. “Looking directly at the regulatory code presents a significant advantage,” adds Kempynck, “It can tell us which regulatory principles are shared across species, even if the DNA sequence itself has changed.” Tool to study disease This regulatory information is useful beyond understanding evolution. In previous work, Aerts and his team already verified that regulatory codes for melanoma (skin cancer) cell states are conserved between mammals and zebrafish. They also identified variants in the genomes of melanoma patients. The models presented in the current study on brain cell types provide useful tools to study the impact of genomic variants and their association with mental or cognitive traits and disorders. Aerts: “Ultimately, models that learn the genomic regulatory code hold the potential to screen genomes and investigate the presence or absence of specific cell types or cell states in any species. This would be a powerful tool to study and better understand disease.” To the zoo Aerts and his team are already applying their models on both fronts, he says: “In collaboration with Zoo Science and Wildlife Rescue Center, we are now expanding our evolutionary modeling to many more animal brains: different types of fish to dear, hedgehogs and capibaras. At the same time, we’re also exploring how these AI models can help to unravel genetic variation linked to Parkinson’s disease.” Reference: “Enhancer-driven cell type comparison reveals similarities between the mammalian and bird pallium” by Nikolai Hecker, Niklas Kempynck, David Mauduit, Darina Abaffyová, Roel Vandepoel, Sam Dieltiens, Lars Borm, Ioannis Sarropoulos, Carmen Bravo González-Blas, Julie De Man, Kristofer Davie, Elke Leysen, Jeroen Vandensteen, Rani Moors, Gert Hulselmans, Lynette Lim, Joris De Wit, Valerie Christiaens, Suresh Poovathingal and Stein Aerts, 14 February 2025, Science. DOI: 10.1126/science.adp3957 RRG455KLJIEVEWWF |
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