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文章數：86 |
 NINI 尼尼台中店會太油嗎？》台中公益路吃爆指南｜10家餐廳逐間介紹 |
| 時事評論｜政治 2026/04/21 02:04:20 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
身為一個熱愛美食、喜歡在城市裡挖掘驚喜的人，臺中公益路一直是我最常出沒的地方之一。這條路可說是「臺中人的美食戰場」，從精緻西餐到創意火鍋，從日式丼飯到義式早午餐，每走幾步，就會有完全不同的特色料理餐廳。 這次我特別花了一整個月，實際造訪了公益路上十間口碑不錯的餐廳。有的是網友熱推的打卡名店，也有隱藏在巷弄裡的小驚喜。我以環境氛圍、口味表現、價格CP值與再訪意願為基準，整理出這篇實測評比。希望能幫正在猶豫去哪裡吃飯的你，找到那一間「吃完會想再來」的餐廳。 評比標準與整理方向
這次我走訪的10家餐廳橫跨不同料理類型，從高質感牛排館到巷弄系早午餐，每一間都有自己獨特的風格。為了讓整體比較更客觀，我依照以下四大面向進行評比，並搭配實際用餐體驗來打分。
整體而言，我希望這份評比不只是「哪家好吃」，而是幫你在不同情境下（約會、家庭聚餐、朋友小聚、商業午餐）都能快速找到合適的選擇。畢竟，美食不只是味覺的滿足，更是一段段與朋友共享的生活記憶。 10間臺中公益路餐廳評比懶人包公益路向來是臺中人聚餐的首選地段，從火鍋、燒肉到中式料理與早午餐，每走幾步就有驚喜。以下是我實際造訪過的10間代表性餐廳清單，橫跨平價、創意、高級各路風格。
一頭牛日式燒肉｜炭香濃郁的和牛饗宴，約會聚餐首選
走在公益路上，很難不被 一頭牛日式燒肉 的木質外觀吸引。低調卻不失質感的門面，搭配昏黃燈光與暖色調的內裝，讓人一進門就感受到濃濃的日式職人氛圍。店內空間不大，但桌距規劃得宜，每桌皆設有獨立排煙設備，烤肉時完全不怕滿身油煙味。 餐點特色
一頭牛的靈魂，絕對是他們招牌的「三國和牛拼盤」。 用餐體驗整體節奏掌握得非常好。店員會在你剛想烤下一片肉時貼心遞上夾子、幫忙換烤網，讓人完全不用分心。整場用餐過程就像一場表演，從視覺、嗅覺到味覺都被滿足。 綜合評分
地址：408臺中市南屯區公益路二段162號電話：04-23206800 小結語一頭牛日式燒肉不僅是「吃肉的地方」，更像是一場五感盛宴。從進門那一刻到最後一道甜點，都能感受到他們對細節的用心。 TANG Zhan 湯棧｜文青系火鍋代表，麻香湯底與視覺美感並重
在公益路這條美食戰線上，TANG Zhan 湯棧 是讓人一眼就會想走進去的那一種。 餐點特色
湯棧最有名的當然是它的「麻香鍋」。 用餐體驗整體氛圍比一般火鍋店更有質感。 綜合評分
地址：408臺中市南屯區公益路二段248號電話：04-22580617 官網：https://www.facebook.com/TangZhan.tw/ 小結語TANG Zhan 湯棧 把傳統火鍋做出新的樣貌保留臺式鍋物的溫度，又結合現代風格與細節服務，讓吃鍋這件事變得更有品味。 如果你想找一間兼具「好吃、好拍、好放鬆」的火鍋店，湯棧會是公益路上最有風格的選擇之一。 NINI 尼尼臺中店｜明亮寬敞的義式早午餐天堂
如果說前兩間是肉食愛好者的天堂，那 NINI 尼尼臺中店 絕對是想放鬆、聊聊天的好地方。餐廳外觀以白色系與大片玻璃窗為主，陽光灑進室內，讓人一踏入就有種度假般的輕盈感。假日早午餐時段特別熱鬧，建議提早訂位。 餐點特色
NINI 的菜單融合義式與臺灣人口味，選擇多樣且份量十足。主打的 松露燉飯 濃郁卻不膩口，米芯保留微Q口感；而 香蒜海鮮義大利麵 則以新鮮白蝦、花枝與淡菜搭配微辣蒜香，口感層次豐富。 用餐體驗店內氣氛輕鬆不拘謹，無論是一個人帶電腦工作、或朋友聚餐，都能找到舒服角落。餐點上桌速度穩定，服務人員態度親切、補水與收盤都非常主動。整體節奏讓人覺得「時間變慢了」，很適合想遠離忙碌日常的人。 綜合評分
地址：40861臺中市南屯區公益路二段18號電話：04-23288498 小結語NINI 尼尼臺中店是一間能讓人放下手機、慢慢吃飯的餐廳。餐點不追求浮誇，而是以「剛剛好」的份量與風味，陪伴每個平凡午後。如果你在找一間能邊吃邊聊天、拍照也漂亮的早午餐店，NINI 會是你在公益路上最不費力的幸福選擇。 加分100%浜中特選昆布鍋物｜平價卻用心的湯頭系火鍋，家庭聚餐好選擇
在公益路這條高質感餐廳林立的戰場上，加分100%浜中特選昆布鍋物 走的是截然不同的路線。它沒有浮誇的裝潢、也沒有高價位的套餐，但靠著實在的湯頭與親切的服務，默默吸引許多回頭客。每到用餐時間，總能看到家庭或情侶三兩成群地圍著鍋邊聊天。 餐點特色
主打 北海道浜中昆布湯底，湯頭清澈卻不單薄，越煮越能喝出海藻與柴魚的自然香氣。 用餐體驗整體氛圍偏家庭取向，桌距寬敞、座位舒適，帶小孩來也不覺擁擠。店員態度親切，補湯、收盤都很勤快，給人一種「被照顧著」的安心感。 綜合評分
地址：403臺中市西區公益路288號電話：0910855180 小結語加分100%浜中特選昆布鍋物是一間「不浮誇、但會讓人想再訪」的火鍋店。它不追求豪華擺盤，而是用最簡單的湯頭與新鮮食材，傳遞出家常卻不平凡的溫度。 印月餐廳｜中式料理的藝術演繹，宴客與家庭聚會首選
說到臺中公益路的中式料理代表，印月餐廳 絕對是榜上有名。這間開業多年的餐廳以「中菜西吃」的概念聞名，把傳統中式料理以現代手法重新詮釋。從建築外觀到餐具擺設，每個細節都散發著低調的典雅氣息。 餐點特色
印月最令人印象深刻的是他們將傳統中菜融入創意手法。 用餐體驗服務方面完全對得起餐廳的高級定位。從入座、點餐到上菜節奏，都拿捏得恰如其分。每道菜都會有服務人員細心介紹食材與吃法，讓人感受到「被款待」的尊榮感。 綜合評分
地址：408臺中市南屯區公益路二段818號電話：0422511155 小結語印月餐廳是一間「不只吃飯，更像品味生活」的地方。 KoDō 和牛燒肉｜極致職人精神，專為儀式感與頂級味覺而生
若要形容 KoDō 和牛燒肉 的用餐體驗，一句話足以總結——「像在欣賞一場關於肉的表演」。 餐點特色
這裡主打 日本A5和牛冷藏肉，以「精切厚燒」的方式呈現。 用餐體驗KoDō 的最大特色是「儀式感」。 綜合評分
地址：403臺中市西區公益路260號電話：0423220312 官網：https://www.facebook.com/kodo2018/ 小結語KoDō 和牛燒肉不是日常餐廳，而是一場體驗。 永心鳳茶｜在茶香裡用餐的優雅時光，臺味早午餐的新詮釋
走進 永心鳳茶公益店，彷彿進入一間有氣質的茶館。 餐點特色
永心鳳茶的餐點結合中式靈魂與西式擺盤，無論是「炸雞腿飯」還是「紅玉紅茶拿鐵」，都能讓人感受到熟悉卻不平凡的味道。 用餐體驗店內服務人員態度溫和，對茶品介紹詳盡。上餐節奏剛好，不急不徐。 綜合評分
地址：40360臺中市西區公益路68號三樓(勤美誠品)電話：0423221118 小結語永心鳳茶讓人重新定義「臺味」。 三希樓｜老饕級江浙功夫菜，穩重又帶人情味的中式饗宴
位於公益路上的 三希樓 是許多臺中老饕的口袋名單。 餐點特色
三希樓的菜色以 江浙與港式料理 為主，兼顧傳統與現代風味。 用餐體驗三希樓的服務給人一種老派但貼心的感覺。 綜合評分
地址：408臺中市南屯區公益路二段95號電話：0423202322 官網：https://www.sanxilou.com.tw/ 小結語三希樓是一間「吃得出功夫」的餐廳。 一笈壽司｜低調奢華的無菜單日料，職人手藝詮釋旬味極致
在熱鬧的公益路上，一笈壽司 低調得幾乎不顯眼。 餐點特色
一笈壽司採 Omakase（無菜單料理） 形式，每一餐都由主廚根據當日食材設計。 用餐體驗整場用餐約90分鐘，節奏緩慢但沉穩。 綜合評分
地址：408臺中市南屯區公益路二段25號電話：0423206368 官網：https://www.facebook.com/YIJI.sushi/ 小結語一笈壽司是一間真正讓人「放慢呼吸」的餐廳。 茶六燒肉堂｜人氣爆棚的和牛燒肉聖地，肉香與幸福感同時滿分
若要票選公益路上「最難訂位」的餐廳，茶六燒肉堂 絕對名列前茅。 餐點特色
茶六主打 和牛燒肉套餐，價格約落在 $700–$1000 間，份量與品質兼具。 用餐體驗茶六的服務效率相當高。店員親切、換網勤快、補水速度快，整場用餐流程流暢無壓力。 綜合評分
地址：403臺中市西區公益路268號電話：0423281167 官網：https://inline.app/booking/-L93VSXuz8o86ahWDRg0:inline-live-karuizawa/-LUYUEIOYwa7GCUpAFWA 小結語茶六燒肉堂用「穩定品質＋輕奢氛圍」抓住了臺中年輕族群的心。 吃完10家公益路餐廳後的心得與結語吃完這十家餐廳後，臺中公益路不只是一條美食街，而是一段生活風景線。 有的餐廳講究細膩與儀式感，像 一頭牛日式燒肉 與 一笈壽司，讓人感受到食材最純粹的美好 有的則以親切與溫度打動人心，像 加分昆布鍋物、永心鳳茶，讓人明白吃飯不只是為了飽足，而是一種被照顧的幸福。 而像茶六燒肉堂、TANG Zhan 湯棧 這類人氣名店，則用穩定的品質與熱絡的氛圍，成為許多臺中人心中「想吃肉就去那裡」的代名詞。 這十家店，構成了公益路最動人的縮影 有華麗的，也有溫柔的；有傳統的，也有創新的。 每一家都在自己的風格裡發光，讓人吃到的不只是料理，而是一種生活的溫度與節奏。 對我而言，這不僅是一場美食旅程，更是一趟關於「臺中味道」的回憶之旅。 FAQ：關於臺中公益路美食常見問題Q1：公益路哪一區的餐廳最集中？ Q2：需要提前訂位嗎？ 最後的話若要用一句話形容這趟美食之旅，我會說： 加分100%浜中特選昆布鍋物團體宴客合適嗎？ 如果你也和我一樣喜歡用味蕾探索一座城市，那就把這篇公益路美食攻略收藏起來吧。KoDō 和牛燒肉CP 值高嗎？ 無論是約會、慶生、家庭聚餐，或只是想犒賞一下辛苦的自己——這條路上永遠會有一間剛剛好的餐廳在等你。一頭牛日式燒肉必點有哪些？ 下一餐，不妨從這10家開始。一笈壽司再訪意願高嗎？ 打開手機、約上朋友，讓公益路成為你生活裡最容易抵達的小確幸。永心鳳茶有什麼推薦搭配？ 如果你有私心愛店，也歡迎留言分享，一頭牛日式燒肉適合多人分享嗎？ 你的推薦，可能讓我下一趟美食旅程變得更精彩。TANG Zhan 湯棧包廂適合尾牙嗎？ UK scientists have completed a synthetic chromosome for the first synthetic yeast genome, marking a major advancement in synthetic biology with broad implications for medicine, bioenergy, and biotechnology. A team of scientists from the United Kingdom, including leading experts from the University of Nottingham and Imperial College London, have successfully constructed a synthetic chromosome. This achievement is a significant milestone in a major international initiative aimed at creating the world’s first synthetic yeast genome. The work, which is published in Cell Genomics, represents the completion of one of the 16 chromosomes of the yeast genome by the UK team, which is part of the biggest project ever in synthetic biology; the international synthetic yeast genome collaboration. The collaboration, known as ‘Sc2.0’ has been a 15-year project involving teams from around the world (UK, US, China, Singapore, UK, France, and Australia), working together to make synthetic versions of all of yeast’s chromosomes. Alongside this paper, another 9 publications are also released today from other teams describing their synthetic chromosomes. The final completion of the genome project – the largest synthetic genome ever – is expected next year. Progress and Significance of the Project This effort is the first to build a synthetic genome of a eukaryote – a living organism with a nucleus, such as animals, plants, and fungi. Yeast was the organism of choice for the project as it has a relatively compact genome and the innate ability to stitch DNA together, allowing the researchers to build synthetic chromosomes within the yeast cells. Humans have a long history with yeast, having domesticated it for baking and brewing over thousands of years and, more recently, using it for chemical production and as a model organism for how our own cells work. This relationship means that we know more about the genetics of yeast than any other organism. These factors made yeast the obvious candidate. The UK-based team, led by Dr Ben Blount from the University of Nottingham and Professor Tom Ellis at Imperial College London, have now reported completion of their chromosome, synthetic chromosome XI. The project to build the chromosome has taken 10 years and the DNA sequence constructed consists of around 660,000 base pairs – which are the ‘letters’ making up the DNA code. The synthetic chromosome has replaced one of the natural chromosomes of a yeast cell and, after a painstaking debugging process, now allows the cell to grow with the same fitness level as a natural cell. The synthetic genome will not only help scientists to understand how genomes function, but it will have many applications. Rather than being a straight copy of the natural genome, the Sc2.0 synthetic genome has been designed with new features that give cells novel abilities not found in nature. One of these features allows researchers to force the cells to shuffle their gene content, creating millions of different versions of the cells with different characteristics. Individuals can then be picked with improved properties for a wide range of applications in medicine, bioenergy, and biotechnology. The process is effectively a form of super-charged evolution. Applications and Future Potential The team has also shown that its chromosome can be repurposed as a new system to study extrachromosomal circular DNAs (eccDNAs). These are free-floating DNA circles that have “looped out” of the genome and are being increasingly recognized as factors in aging and as a cause of malignant growth and chemotherapeutic drug resistance in many cancers, including glioblastoma brain tumors. Dr Ben Blount, one of the lead scientists on the project, is an Assistant Professor in the School of Life Sciences at the University of Nottingham. He said: “The synthetic chromosomes are massive technical achievements in their own right, but will also open up a huge range of new abilities for how we study and apply biology. This could range from creating new microbial strains for greener bioproduction, to helping us understand and combat disease. “The synthetic yeast genome project is a fantastic example of science on a large scale that has been achieved by a large group of researchers from around the world. It’s been a great experience to be part of such a monumental effort, where all involved were striving towards the same shared goal.” Professor Tom Ellis from the Centre for Synthetic Biology and Department of Bioengineering at Imperial College London, said: “By constructing a redesigned chromosome from telomere to telomere, and showing it can replace a natural chromosome just fine, our team’s work establishes the foundations for designing and making synthetic chromosomes and even genomes for complex organisms like plants and animals.” Reference: “Synthetic yeast chromosome XI design provides a testbed for the study of extrachromosomal circular DNA dynamics” by Benjamin A. Blount, Xinyu Lu, Maureen R.M. Driessen, Dejana Jovicevic, Mateo I. Sanchez, Klaudia Ciurkot, Yu Zhao, Stephanie Lauer, Robert M. McKiernan, Glen-Oliver F. Gowers, Fiachra Sweeney, Viola Fanfani, Evgenii Lobzaev, Kim Palacios-Flores, Roy S.K. Walker, Andy Hesketh, Jitong Cai, Stephen G. Oliver, Yizhi Cai, Giovanni Stracquadanio and Tom Ellis, 9 November 2023, Cell Genomics. DOI: 10.1016/j.xgen.2023.100418 As well as the leads of Nottingham and Imperial College London, the UK team also includes scientists from the universities of Edinburgh, Cambridge, and Manchester in the UK, as well as John Hopkins University and New York University Langone Health in the USA and Universidad Nacional Autónoma de México, Querétaro in Mexico. The work was funded by the BBSRC. Methylene Blue, a century-old medicine, is safe for coral reefs and has the potential to be a highly effective, broad-spectrum UV irradiation protector that absorbs UVA and UVB, and repairs ROS and UV irradiation induced DNA damages. Scientists find that Methylene Blue could be an effective alternative to sunscreens that cause coral damage. A new study published in Nature Scientific Reports has found that Methylene Blue, a century-old medicine, has the potential to be a highly effective, broad-spectrum UV irradiation protector that absorbs UVA and UVB, repairs ROS and UV irradiation induced DNA damages, and is safe for coral reefs. The study suggests that Methylene Blue could become an alternative sunscreen ingredient that supports the environment and protects human skin health. 80% of today’s sunscreens use Oxybenzone as a chemical UV blocker, despite multiple studies that have shown it expedites the destruction of coral reefs. Several states and countries have now banned the use of Oxybenzone and its derivatives to stop the devastating effects on the world’s marine ecosystem. In addition, consumers focus primarily on the Sun Protection Factor (SPF) to prevent sunburns and potentially dangerous long-term health issues. However, SPF only measures UVB exposure, leaving sunscreen users vulnerable to UVA-triggered oxidative stress and photo-aging. “Our work suggests that Methylene Blue is an effective UVB blocker with a number of highly desired characteristics as a promising ingredient to be included in sunscreens. It shows a broad spectrum absorption of both UVA and UVB rays, promotes DNA damage repair, combats reactive oxygen species (ROS) induced by UVA, and most importantly, poses no harm to coral reefs.” says the study’s senior author Dr. Kan Cao, Founder of Mblue Labs, Bluelene Skincare and a Professor at the University of Maryland Department of Cell Biology and Molecular Genetics. The research team, which included scientists from Mblue Labs and the University of Maryland, looked at the UV protection benefits of Methylene Blue from several angles in primary human keratinocytes and skin fibroblasts from young and old donors and compared those results with Oxybenzone. They concluded that Methylene Blue not only absorbs UVA & UVB as the traditional sunscreen actives do, it also helps repair the DNA damage caused by UV irradiation, thereby leading to better cell survival. They also exposed the same amounts of Methylene Blue or Oxybenzone in Xenia umbellate, a soft coral species, in isolated tanks and monitored coral reefs’ growth and responses to these chemicals. They reported drastic coral bleaching and death in Oxybenzone-treated Xenia corals in less than a week, while Methylene Blue does not have any negative effects on coral health even at a relatively high concentration (1 micromolar). They also compared Methylene Blue with other well-known skincare antioxidants such as Vitamin A (Retinol) and Vitamin C in their ability to reduce cellular oxidative stress. “We are extremely excited to see that skin fibroblasts, derived from both young and old individuals, have improved so much in terms of proliferation and cellular stress in a methylene blue-containing cell culture medium.” Dr. Cao shares. “Most surprisingly,” according to Dr. Cao,” we found that the combination of Methylene Blue and Vitamin C could deliver amazing anti-aging effects, particularly in skin cells from older donors, suggesting a strong synergistic reaction between these two beneficial antioxidants.” “Altogether, our study suggests that Methylene Blue has the potential to be a coral reef-friendly sunscreen active ingredient that can provide broad-spectrum protection against UVA and UVB.” The team concludes in the abstract. The researchers are so confident in their findings that Methylene Blue is an effective UV blocking agent that also delays skin aging and promotes DNA damage repair, that they have filed a patent application and have started developing sunscreen prototypes containing Methylene Blue. In addition, Mblue Lab (Bluelene) recently launched the first multifunctional skincare product that combines Methylene Blue and Vitamin C to deliver the optimal anti-aging effects (Bluelene’s Night Plus+). Reference: “Ultraviolet radiation protection potentials of Methylene Blue for human skin and coral reef health” by Zheng-Mei Xiong, Xiaojing Mao, Mason Trappio, Chanda Arya, Jasmin el Kordi and Kan Cao, 28 May 2021, Scientific Reports. DOI: 10.1038/s41598-021-89970-2 This research was supported by a National Science Foundation (NSF) Small Business Technology Transfer Grant (Grant: 1842745). This study was conducted jointly by researchers at Mblue Labs and the University of Maryland. About Mblue Labs + Bluelene: The research team at the University of Maryland, led by Dr. Kan Cao, has measured the effectiveness of Methylene Blue for increased skin hydration, expanded thickness of the dermis, and improved skin texture when compared to products that have mitochondrial-targeting antioxidants. Methylene Blue outperformed MitoQ, NAC and MTemp in this experiment, as a more effective way to enhance mitochondrial health and delay senescence of the cells. It’s with our steadfast research that in 2019 we introduced Bluelene, an anti-aging face and body skincare line infused with Methylene Blue to deliver lasting results. Norepinephrine-mediated slow vasomotion drives glymphatic clearance during sleep. Credit: Nadia Alzoubi and Natalie Hauglund, edited Deep sleep doesn’t just rejuvenate your body — it clears your mind by flushing out waste that builds up during waking hours. Scientists discovered that norepinephrine, released in rhythmic waves during deep sleep, drives the brain’s glymphatic system, which pumps cerebrospinal fluid to clear toxins. However, sleep aids like zolpidem may disrupt this process, reducing the brain’s cleaning efficiency. Understanding the Brain’s Waste Removal System Getting a good night’s sleep does more than leave you feeling refreshed — it actively clears your brain. New research, published on January 8 in the journal Cell, reveals that deep sleep helps flush out waste that builds up in the brain during waking hours. This natural cleansing process is vital for maintaining brain health. The study also highlights how sleep aids might interfere with this “brainwashing” mechanism, potentially impacting long-term cognitive function. The brain relies on a built-in waste removal system called the glymphatic system, which circulates fluid throughout the brain and spinal cord to eliminate waste. This system helps clear away toxic proteins that can accumulate and form sticky plaques, a hallmark of neurological disorders. However, the mechanisms driving this system had remained unclear — until now. The Role of Norepinephrine in Brain Cleaning Researchers in Denmark discovered that a molecule called norepinephrine plays a crucial role in this process, at least in mice. During deep sleep, the brainstem releases tiny waves of norepinephrine roughly every 50 seconds. These waves cause blood vessels in the brain to contract, creating slow, rhythmic pulsations that propel surrounding fluid to carry away waste effectively. “It’s like turning on the dishwasher before you go to bed and waking up with a clean brain,” says senior author Maiken Nedergaard of the University of Rochester and University of Copenhagen, Denmark. “We’re essentially asking what drives this process and trying to define restorative sleep based on glymphatic clearance.” How Norepinephrine Drives Fluid Flow To find clues, Nedergaard and her team looked into what happens in mice when the brain sleeps. Specifically, they focused on the relationship between norepinephrine and blood flow during deep slumber. They found that norepinephrine waves correlate to variations in brain blood volume, suggesting norepinephrine triggers a rhythmic pulsation in the blood vessels. Blood Vessels as Brain Fluid Pumps The team then compared the changes in blood volume to brain fluid flow. They found that the brain fluid flow fluctuates in correspondence to blood volume changes, suggesting that the vessels act as pumps to propel the surrounding brain fluid to flush out waste. “You can view norepinephrine as this conductor of an orchestra,” says lead author Natalie Hauglund of the University of Copenhagen and the University of Oxford, UK. “There’s a harmony in the constriction and dilation of the arteries, which then drives the cerebrospinal fluid through the brain to remove the waste products.” Does All Sleep Promote Brain Health? Hauglund then had another question — is all sleep created equal? To find out, the researchers gave mice zolpidem, a common drug to aid sleep. They found that the norepinephrine waves during deep sleep was 50% lower in zolpidem-treated mice than in naturally sleeping mice. Although the zolpidem-treated mice fell asleep faster, fluid transport into the brain dropped more than 30%. The findings suggest that the sleeping aid may disrupt the norepinephrine-driven waste clearance during sleep. “More and more people are using sleep medication, and it’s really important to know if that’s healthy sleep,” says Hauglund. “If people aren’t getting the full benefits of sleep, they should be aware of that so they can make informed decisions.” Human Implications of Sleep Research The team says that the findings likely apply to humans, who also have a glymphatic system, although this needs further testing. Researchers have observed similar norepinephrine waves, blood flow patterns, and brain fluid flux in humans. Their findings may offer insights into how poor sleep may contribute to neurological disorders like Alzheimer’s disease. “Now we know norepinephrine is driving the cleaning of the brain, we may figure out how to get people a long and restorative sleep,” says Nedergaard. For more on this study, see How Zolpidem May Damage Your Brain’s Cleanup System. Reference: “Norepinephrine-mediated slow vasomotion drives glymphatic clearance during sleep” by Natalie L. Hauglund, Mie Andersen, Klaudia Tokarska, Tessa Radovanovic, Celia Kjaerby, Frederikke L. Sørensen, Zuzanna Bojarowska, Verena Untiet, Sheyla B. Ballestero, Mie G. Kolmos, Pia Weikop, Hajime Hirase and Maiken Nedergaard, 8 January 2025, Cell. DOI: 10.1016/j.cell.2024.11.027 This work was supported by the Lundbeck Foundation, the Novo Nordisk Foundation, the National Institutes of Health, the US Army Research Office, the Human Frontier Science Program, the Dr. Miriam and Sheldon G. Adelson Medical Research Foundation, the Simons Foundation, the Cure Alzheimer Fund, the Danmarks Frie Forskningsfond, and JPND/Good Vibes. RRG455KLJIEVEWWF |
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